10.2 (8.4 Fistula bits) Palliative wound and ostomy care Flashcards

1
Q

What are three types of skin failure?

A

Acute, chronic and end stage

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2
Q

Name 3 causes for:

(a) acute skin failure
(b) chronic skin failure

A

Acute skin failure:
- Peripheral arterial disease*
- Severe sepsis/septic shock*
- Respiratory failure*
- Mechanical ventilation
- Liver failure

Chronic skin failure:
- Peripheral arterial disease*
- Diabetes*
- Nephropathy*
- Neurodegen disorders

V - PAD
I - sepsis
N - cancer
D - liver or kidney failure
I - radiation, chemo
C
A - eczema
T - trauma
E - diabetes

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3
Q

What are 2 examples of end stage skin failures?

Are they avoidable or unavoidable injuries?

A

Kennedy terminal ulcers (KTUs)
Trombley-Brennan terminal tissue injury (TB-TTI)

Generally unavoidable injuries

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4
Q

2 ways to distinguish KTUs from TB-TTI

A

KTU:
- Over bony prominences
- Develop < 2 months prior to death

TB-TTI:
- Found in locations with little to no pressure (e.g thighs)
- Median time to death is 36 hours

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5
Q

Name 3 characteristics of KTUs (in terms of appearance)

A

Sacroccoygeal location*
Sudden onset*
Butterfly or pear shape*

Irregular borders
Purple, blue, black or red color

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6
Q

What are the 11 parts of systematic wound assessment?

How often should this be done?

A
  1. Location
  2. Etiology or type of skin damage
  3. Dimension
  4. Base description (e.g. granulation, nerotic, eschar, slough, epithelial, mixed)
  5. Undermining/tunneling (stage III or IV pressure injuries only)
  6. Stage
  7. Drainage
  8. Odor
  9. Periwound skin
  10. Pain
  11. Infection

Done weekly or with change in condition

LED
BUS
DOPPI

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7
Q

What are 5 common symptoms associate with wound?

A

Pain, odor, bleeding, exudates, pruritus

FS: DOPPI
Drain (bleeding, exudates)
Odor
Periwound skin - pruritus
Pain
Infection

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8
Q

What can exacerbate wound symptoms? List 2

A

Infection
Lymphedema

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9
Q

Name wound pain interventions:

  • 4 meds
  • 4 care methods
A

4 meds:
- Systemic antibiotics
- Systemic medications (opioids, adjuvants)
- Topical opioids
- Topical anesthetics (2% lidocaine jelly 3-5 min before wound care)

4 interventions:
- Anti-microbial dressings
- Non-adherent dressing (foams, silicone)*
- Soaking dressing before removal with NS or tap water*
- Gentle irrigation/patting/blotting wound bed*
- Decrease number of changes with moisture retentive dressings*

  • Use of burn nets or garments to secure dressing and normalize body image
  • If adhesives needed, use skin protectants and apply skin tape
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10
Q

Key components to wound bed preparation

A

Tissue that is nonviable must be debrided

Infection or inflammation must be managed

Moisture imabalance or exudate control must be addressed

Edge margins of wound must be examined for non-adherence

TIME

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11
Q

Common wound debridement methods

A

Autolytic debridement: hydrogel (Tegaderm) dressings

Biological debridement: larval therapy

Chemical debridement: iodine, medical grade honey

Surgical and sharp (doctor) debridement: scalpel, scissors, forceps by trained tissue viability nurse or surgeon

Enzymatic debridement: collagenase agents

Mechanical debridement: ultrasound and water irrigation devices

FS: ABCDE + M

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12
Q

Name 4 agents to help to control wound odour and drainage

A
  • Flagyl (spray, powder, tablets, gel)*
  • Honey*
  • Charcoal*
  • Silver*
  • Foams
  • Calcium alginate
  • Dakin solution (diluted bleach)*
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13
Q

How to manage wound related bleeding issues as below:

  • Prevention (3)
  • Minor bleeding (3)
  • Major bleeding (2)
  • Hemorrhage as a terminal event (3)
A

Prevention:
- Non-adherent dressing
- Wet dressings before removal
- Gentle cleansing or irrigation

Minor bleeding:
- Pressure
- Silver nitrate sticks
- Epinephrine soaks
- Calcium alginate

Major bleeding:
- Vascular intervention
- Radiation

Terminal bleed:
- Dark towel
- Benzo PRN
- Cover with blankets as blood loss can cause hypothermia

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14
Q

What are 2 peri-wound skin barriers to reduce pruritus and moisture-associated skin damage?

A
  • Liquid polymer acrylates
  • Dimethicone
  • Zinc-oxide skin barrier* (triad)
  • Petroleum skin barrier* (Vaseline)
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15
Q

Name 2 pressure-injury risk assessment tools

A

Braden Scale

Norton Scale (Norton hears a who…)

Hospice Pressure Ulcer Risk Assessment Scale

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16
Q

Name 4 risk factors for pressure-injury

A
  • Advanced age*
  • Protein-calorie malnutrition*
  • Immobility*
  • Shear*
  • Friction
  • Moisture*
  • Incontinence
  • Altered sensory perception
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17
Q

Name 4 ways to reduce/manage pressure-injury

A
  • Maximizing nutrition and hydration*
  • Pressure redistribution*
  • Skin care (TIME)*
  • Managing incontinence*
  • Controlling wound related symptoms
  • Providing psychosocial support

FS: think of cause -> intervention

Age - psychosocial support
Malnutrition - hydration + nourish
Immobility - pressure redistribution
Shear/friction - skin care
H20/Moisture - manage incontinence

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18
Q

Name 3 ways to facilitate pressure redistribution? What about foam rings or cutouts?

A
  • Regular repositioning*
  • Heel suspension devices and protective dressing*
  • Support surfaces on beds and chairs (e.g. roho cushion)*
  • Minimize or reduce medical device

Don’t use foam rings or cutouts

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19
Q

Describe the 4 stages of NPUAP pressure injury staging system

A

Stage 1 pressure injury: non-blanchable erythema of intact skin
Stage 2: partial thickness skin loss with exposed dermis
Stage 3: full thickness skin loss (adipose tissue is visible)
Stage 4: full thickness skin and tissue loss (fascia, muscle, tendon, ligament, cartilage or bone visible)

FS:

Epidermis - Dermis - Hypodermis/SC tissue - Muscle

Skin = epidermis, dermis, hypodermis

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20
Q

What are 3 ways to prevent incontinence related dermatitis

A
  • Non-invasive containment products (absorptive products, fecal pouch, condom catheter, etc.)*
  • Invasive containment productrs (urine catheter, fecal management system)*
  • Incontinence care (pH balanced, non-rinse cleanser; skin moisturizer; skin barrier)*
  • Treatment of candidiases
21
Q

2 types of skin tears

A

Partial thickness (separation of epidermis from dermis)

Full thickness (separation of both epidermis and dermis from underlying structures - fat)

22
Q

Name 4 ways to prevent skin tears

A
  1. Asssess for risk upon admission and whenever condition change
  2. Implement systematic prevention protocol
  3. Have individuals at risk wear long sleeves, long pants, knee high socks*
  4. Shin guard*
  5. Safe patient handling techniques*
  6. Involve patients and families in preventative strategies
  7. Condult dietician to ensure adequate nutrition and hydration*
  8. Keep skin moisturized at least 2 times per day*

FS:
Nourish (dietician)
Moisturize (twice a day)
Cover skin (long sleeves, skin guard)
Careful patient handling

23
Q

How to manage new skin tears (5 steps)

A
  1. Cleansing with non-toxic solution (NS)
  2. Control bleeding and remove clots
  3. Approximating skin flap when possible
  4. Apply skin protectant on peri wound area
  5. Apply hydrogel (tegaderm), alginate, foam or non adherent dressing
24
Q

Chronic critical limb ischemia:
- What is the etiology?
- How does it manifest? (3)
- What is the possibility of healing?

A

Arterial occlusive disease

Manifest as rest pain, ulceration or gangrene

Healing is not realistic goal when invasive vascular interventions are not available

25
Q

What are 2 types of gangrene?

A

Dry and wet

26
Q

Wet gangrene:
- What does it signal?
- What organism can be involved?
- What is treatment?

A
  • Bacterial infection
  • Clostridium perfringens
  • Apply Dakin solution once or twice daily (bactericidal and decrease exudates)

FS: Dakin = diluted bleach

27
Q

What is malignant fungating wound?

Name 3 cancers affilitated

A

Non-healing wounds - from aggressive proliferation of malignant cells - with infiltration of skin, blood and lymph

Head and neck*
breast*
lung
GI
skin*

28
Q

More than 50% of those with malignant fungating wound will die how fast?

A

Within 6 months

29
Q

Beside wound symptoms (DOPPI), what are other negative effects of malignant fungating wounds?

A
  • Psych (Depression or anxiety)
  • Social (marginalization, isolation)
  • Spiritual (cause patients to focus on impending death, loss of hope)
30
Q

What are marjolin ulcers?

Commonly seen with which cancers?

A

Malignant degenerations over years or decades (from areas of chronic skin inflammation or injury)

Commonly squamous cell ca - BCC and melanoma have been reported

FS:

Marjolin ulcers are a type of skin cancer that can develop from chronic wounds or scars. Specifically, they arise from long-standing non-healing wounds, scars, or burns, and can occur anywhere on the body.

31
Q

Name 3 skin conditions (beside cancer) that can give rise to marjolin ulcers

A

Burns*
Frostbites*
Traumatic wounds*
Venous ulcers
Pressure injuries
Osteomyelitis

FS: VINDICATE
V - venous ulcer
I - osteomyelitis
N - SCC > BCC / melanoma
T - burns, frostbites, trauma

32
Q

How to treat marjolin ulcer?

A
  • Surgical excision +/- skin graft
  • Treat symptoms
33
Q

Calcinosis cutis VERSUS Calciphylaxis:

  • What are they?
  • What is the cause?
  • Where are they found?
A

Calcinosis cutis:
- abnormal deposition of calcium in skin and fat tissue (((extravascular)))
- idiopathic or malignant hypercalcemia - met ca, CKD, hyperparathryroidism
- commonly in lower extremeties

Calcyphylaxis:
- calcification of small vessels of skin and fat tissues resulting in localized ischemia and tissue damage (((intravascular)))
- usually due to ESRD
- can be anywhere on body and may affect multiple areas

34
Q

What are general principles for treating calcinosis cutis and calciphylaxis?

A
  • Treat underlying cause
  • Correct electrolytes
  • Plan for wound care
35
Q

What are 2 types of radiation dermatitis?

How do they manifest?

A

Acute rad dermatitis - manifest as erythema or dry/moist desquamation

Delayed rad dermatitis (months or years) - manifest as chronic dermatitis and/or skin fibrosis

36
Q

Describe general skin care during radiation therapy (3 ways)

A

Mild soaps and deodorants*
Loose fitted clothing over irradiated site
Avoid sun exposure*
Avoid scratching*
Adequate nutrition

Moisturize

37
Q

What 2 prophylactic treatments have shown promise in reducing radiation dermatitis

A

Topicals:
- Steroids
- Silver sulfadiazine (silver + sulfa = antimicrobial)

38
Q

Name if the following stomas are for stool or urine:

Ileostomy
Colostomy
Urostomy (ileal conduit)
Ureterstomy
Indiana pouch
Neobladder

table 10.2.12

A

Stool:
Ileostomy
Colostomy

39
Q

Besides DOPPI*, name 3 unique skin complications of stoma (and their treatments)

A

(1) Mucocutaneous separation: complete or partial detachment of stoma from surrounding skin (not correctable)

(2) Peristomal varices: dilatation of mucocutaneous vessels surrounding stoma (treat bleeding with pressure, silver nitrate, epinephrine gauze, suture ligation)

(3) Peristomal moisture-associated dermatitis: inflammation and erosion of skin related to moisture that begins at the stoma/skin junctin and extends outward (choosing pouching system that fits closely around stoma and prevent leakage)

*Drainage, odor, pain, peri wound skin, infection

40
Q

Name 4 etiologies of fistula

A

V - vascular deficiency
I - infection
N - advanced cancer
D - malnutrition
I - steroid, radiation
C
A
T - post op, dehiscence of interstitial anastomosis
E - endocrine abnormalities

41
Q

2 most common organ systems affiliated with fistula

A

GU and GI

42
Q

How to classify fistulas by output volume?

A

Low output - < 200ml/day

Mod output - 200-467 ml/day

High output - >467 ml/day

43
Q

Name 6 complications of fistulas

A

Pain*
Bleeding*
Infection*

Tissue destruction
Delirium*
Malnutrition*
Fluid and lyte imbalance*
Acid base imbalance

DOPPI

44
Q

Name 6 conditions which are barriers to closure of fistulas

A

A - Abcess
B - IBD
C - Cancer
D - Distal obstruction
E - Epithelialization of tract
F - Foreign body

45
Q

What investigations can be ordered to assess fistulas
- 2 general
- 2 GI
- 2 GU

A

CT scan

Fistulogram (injection of water soluable contrast directly into fistula followed by radiograph)

Barium swallow - Upper GI series (pt swallows barium and then have fluoroscopy of upper GI tract)

Barium enema (barium enema and then have fluoroscopy of colon)

IV pyelogram (xray of urinary tract with IV contrast)

Cystoscopy

46
Q

Name 4 main components of fistula management

A

Skin cleansing and protection*
Uneven skin surface management
Dressing or pouch selection
Containment of output *
Odor management*
Nutrition management*

FS:
DOPPI management including intake (nutrition)

47
Q

Name 3 meds that can slow GI fistula output

A

Anticholinergic (loperamide, atropine)
Opioids
Octreotide

48
Q

what are two types of bladder fistulae? What are three risk factors for this?

A

vesicovaginal
vesicoenteric

Risk factors:
advanced malignancy
prior pelvic floor sx
XRT
poor nutrition
poor wound healing
infection

FS: VINDICATE causes of fistula

49
Q

List 3 complications of vesicoenteric fistula and vesicovaginal fistula

A

rash
skin breakdown
ulcer
chronic infection
sepsis