What is glycemic index Flashcards
which is also a common standard) and have their blood sugar curves measured.
The glycemic index figures you see published are derived the same way as I explained above. Young, healthy subjects consume a test dose of glucose (or sometimes 50 grams of carb from white bread,
Glycemic index figures are calculated
Glycemic index figures are calculated using measured doses of certain foods compared to pure glucose, We know that 50 grams of potato starch gives 80% of the blood sugar rise compared to glucose. But what happens if we eat some butter along with the potato? Will the glucose curve be larger or smaller than it is with the potato alone? No one knows. Could you imagine the complexities involved in just trying to figure that one out? How about if we eat the potato with butter and drink a cup of coffee?
how do we calculate the glycemic index for the whole meal
It’s impossible.
And glycemic index figures are calculated using data derived from young, healthy subjects. What if I’m old and have a touch of the Metabolic Syndrome? Will these figures be the same for me? I don’t think so. But who knows? It’s never been tested.
GI and fructose, where does the energy go
50 grams of pure fructose gives a blood sugar curve that is about 22-24% of that produced by glucose, which means that fructose has a glycemic index of only 22-24, which is very low by anyone’s estimation. Why is fructose low? Because it doesn’t convert to blood sugar, it converts to fat in the liver instead. And causes a lot of problems. But has a low, low glycemic index. So foods containing fructose then have a low-glycemic index.
The glycemic load
is the glycemic index multiplied times the grams of carb in the food in question.
LCHF vs LFHC diet
The researchers calculated that the subjects on the LCHF diet consumed about three times as much saturated fat as those on the LFHC diet. Upon examining the blood after 12 weeks on the two diets, it was found that the subjects on the LCHF diet had a 57% decrease in the amount of saturated fat over the course of the study compared to those on the LFHC diets, who decreased the saturated fat content of their blood by only 24%. A three fold increase in dietary saturated fat when taken as part of a low-carb diet reduced the amount of saturated fat in the blood by a factor of 2.
palmitoleic acid (PA)
PA is a marker for endogenous (inside the self) lipogenesis (new fat making). In other words more PA in a subject means that the liver of that subject is making more fat from carbohydrate.
palmitoleic acid (PA)
PA is a marker for endogenous (inside the self) lipogenesis (new fat making). In other words more PA in a subject means that the liver of that subject is making more fat from carbohydrate.
LCHF diet
on the LCHF diet decreased the amount of PA in their triglycerides (the transport and storage structure of fat) by 32% whereas those on the LFHC diet didn’t reduce their levels of PA at all.
