Week 9- effects of resistance training Flashcards

1
Q

What is muscular strength?

A

maximal force that a muscle group can generate
-1 repetition maximum

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2
Q

What is muscular endurance?

A

ability to make repeated contractions against a submaximal load

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3
Q

What is high-resistance training?

A

-6-10 reps till failure
-results in strength increase

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4
Q

What is low-resistance training?

A

-30-40 reps till failure
-results in increased endurance

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5
Q

What is sarcopenia?

A

loss in muscle mass

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6
Q

What occurs to the muscles as we age?

A

-loss of muscle mass
-atrophy type 2 fibres
-loss of motor neurons
-reduced number of both type 1 and 2 fibres

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7
Q

What does resistance training promote?

A

Muscle hypertrophy

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8
Q

What is responsible for early gains in strength?

A

Neural adaptations

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9
Q

What are the strength gains during the first 8 weeks of training due to?

A

CNS adaptations

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10
Q

What shows the CNS adaptations in the early stages of training?

A

-strength increases of training whilst no change in fibre size
-training in one limb results in strength in untrained limb
-

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11
Q

What are the adaptations that occur in the muscle fibres that occurs later on in the training?

A

-increase muscle fibre specific tension
-increase muscle mass

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12
Q

What are the neural adaptations from resistance training?

A

-increased neural drive
-increase in number of motor units recruited
-increase in firing rate of motor units
-increase motor unit synchronisation
-improved neural transmission across neuromuscular junction
-Changes in rate of agonist and antagonist co-activation

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13
Q

What is the mechanism seemed to be linked with training-induced increase in specific tension in type 1 fibres?

A

increase in calcium sensitivity

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14
Q

what does an increase in calcium sensitivity cause?

A

greater number of cross-bridge bound to actin

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15
Q

What is the result of a greater number of cross-bridge binding to actin?

A

-more force per motor unit
-strength gains independent of muscle growth

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16
Q

What is hyperplasia?

A

increase in number of muscle fibres

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16
Q

What causes muscle hypertrophy?

A

increased muscle proteins (actin and myosin)

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16
Q

What is the definition of muscle hypertrophy?

A

increased cross-sectional area of muscle fibres

17
Q

What do type IIx muscle fibres change to with resistance training?

A

Type IIa

18
Q

How does muscle growth occur?

A

protein synthesis exceeds the rate of protein breakdown

19
Q

How many weeks must protein synthesis exceed the rate of protein breakdown to achieve significant fibre growth?

A

3 or more weeks

20
Q

What are the key factors that contribute to resistance-training induced increases in muscle protein synthesis?

A

-mRNA increases resulting in protein synthesis at the ribosome
-ribosomes increase in number and increase muscle synthesis capacity
-activation of protein kinase ‘mechanistic target of rapamycin’ (mTOR) is the key factor in accelerating protein synthesis following a bout of resistance training

21
Q

what are the two signalling molecules that stimulate mTOR activation?

A

-phosphatidic acid (PA)
-Ros homolog enriched in brain (Rheb)

22
Q

What activates sarcolemmal mechanoreceptors that activate extracellular signal-regulated kinase (ErK)?

A

muscle contractions

23
Q

What does active ErK inhibit?

A

TSC2

24
Q

What is TSC2?

A

a inhibitor of Rheb

25
Q

How does resistance training activate mTOR?

A

By synthesising PA and removing the TSC2 inhibition of Rheb

26
Q

What hormones are linked to mTOR activation and have the potential to increase muscle protein synthesis?

A

insulin-like growth factor-1 (IGF-1)

27
Q

What effects does resistance training have on IGF-1 and growth hormone?

A

small increases in circulating levels

28
Q

What are satellite cells?

A

Stem cells located between the sarcolemma and basal lamina

29
Q

What effects does resistance training have on satellite cells?

A

activates the satellite cells to divide and fuse with adjacent muscle fibres to increase myonuclei

30
Q

What effect does resistance training on myonuclei?

A

increase in myonuclei results on a constant ratio between number of myonuclei and size of muscle fibre

31
Q

What is the impact of the addition of new myonuclei to fibres?

A

support increased protein synthesis in large muscle fibres

32
Q

What % of muscle mass difference between individuals is due to genetic variation?

A

80%

33
Q

How many genes are major contributors to muscle mass?

A

47

34
Q

How are hypertrophy-linked genes activated?

A

resistance training linked t mTOR pathway

35
Q

What does the stopping of resistance training cause?

A

muscle atrophy and loss of strength

36
Q

Compared to endurance training, is the rate of detraining faster or slower for resistance training?

A

slower

37
Q

What is ‘muscle memory’?

A

ability for rapid recovery after the stopping of resistance training

38
Q

What is muscle memory due to?

A

resistance-training induced increase in myonuclei in trained fibres that are not lost due to retraining

39
Q

How much do muscle fibres reduce in size 20-30 days of muscle inactivity?

A

20-30%

40
Q

What is the conservation of muscle mass dependent upon?

A

Balance between protein synthesis and rate of protein breakdown

41
Q

What does increased radical production promote?

A

muscle atrophy during prolonged inactivity by depressing protein protein synthesis and increasing degradation

42
Q
A