Weaning to Finish 5 & 6 Flashcards

1
Q

What potential causes of D+ in older pigs?

A

Colitis – dietary factors
Ileitis - Lawsonia intracellularis
Salmonella spp.
Swine dysentery Brachyspira hyodysenteriae
Brachyspira pilosicoli
Yersinia enterolitica
Porcine Epidemic Diarrhoea / Transmissible Gastroenteritis
Notifiable diseases
Non specific – intestinal parasites, management, cold / damp

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2
Q

What is Lawsonia intracellularis?

A

Ileitis aka PIA= porcine inesinal adenomaosis

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3
Q

Presentations of Lawsonia?

A
  1. Peracute – sudden death from intestinal haemorrhage. ‘Haemorrhagic enteropathy’
  2. Acute – magenta colour scour
  3. Chronic – scour
  4. Subclinical – poor growth
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4
Q

Describe Acute ileitis?

A
  • Extensive haemorrhage in
    intestine
  • Can be difficult to distinguish from intestinal torsion, so open pig
  • VERY slowly
  • If bowel wall is not inflamed, check for stomach ulcer
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5
Q

Describe Chronic Ileitis?

A

Typical Diphtheriic gu
- Enlarged mesenteric LNs
- Thickened diphtheriic terminal ileum (section just before joing colon)

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6
Q

Control of Ileitis?

A
  • Hygiene – scrape through passages + straw beds always difficult
  • Ensure all-in, all-out as far as possible and maintain pig flow
  • Vaccination - injectable
  • Vaccination – oral vaccine. Usually vaccinate at weaning
    ◦ Live vaccine, so feed MUST be free from antibiotics
    ◦ Can then use water soluble tylosin to “embed” the vaccine
    Antibiotics, eg tylosin, tiamulin, OTC, CTC
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7
Q

Name of swine dysentery?

A

Brachyspira hyodysenteriae

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8
Q

Describe Swine dysenery

A

Dysenteric scour, ie blood + mucous
High mortality if not controlled
Deaths in sows and finishers
Differential – acute ileitis

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9
Q

Swine Dysentery vs Acue ileitis

A

SD in colon, not ileum
SD gives higher mortality
SD can affect sows
SD generally in older pigs

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10
Q

What are some factors do swine dysentery?

A

Massive weight loss and deaths
Biosecurity essential
Loading ramps
Clean lorries
Birds, foxes etc – disposal of carcases
Some strains resistant to some antimicrobials
BASIC BIOSECURITY

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11
Q

Dx of SD?

A

CLINICAL SIGNS
PCR (care), culture
Antimicrobial sensitivity

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12
Q

Tx for SD?

A

Tiamulin – in feed or via the water
Lincomycin or lincospectin – via water
May need to use a combination of antibiotics depending on the sensitivity
RESISTANCE HAS BEEN FOUND to both tiamulin and lincomycin

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13
Q

Controlling Swine dysentery?

A

Can learn to live with it – strategic medication, marketing

Eradication preferred
▪ Medication
▪ Depopulation/repopulation

Dysentery Charter

Responsibility to the industry

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14
Q

Diagnostic approach to enteric dx?

A

▪ Clinical history – age, stage of production, vaccination, feeding history
▪ Clinical inspection – morbidity and mortality
▪ Investigation ->
- Faecal sampling, swabs, pooled faeces samples
- Post mortem - gross appearance, intestinal samples, hiso sections
- Submission of carcase to APHA

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15
Q

Tx & control of enteric dx?

A

▪ Group / individual
▪ In feed, in water, by injection
▪ Vaccination
▪ Nutritional manipulation

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16
Q

Most common cause of Meningitis?

A

Strep suis type 2

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17
Q

Sporadic causes of Meningitis can also be caused by ….

A

◦ Glasserella parasuis - Glassers Disease
◦ consequence of pyaemia, eg from tail biting
◦ Salt poisoning/water deprivation

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18
Q

Meningitis Often precipitated by environmental conditions/stress such as….

A

◦ Large temperature fluctuations especially in autumn and spring
◦ High humidity
◦ Poor ventilation
◦ overstocking

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19
Q

CLS of meningitis?

A

Sudden death
Neurological signs
◦ Ataxia
◦ Recumbency
◦ Paddling
◦ Proprioceptive deficits
◦ Pyrexia

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20
Q

Tx for meningitis?

A

◦ Penicillin + NSAID’s
◦ Water (per os / per rectum)

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21
Q

Control of Meningitis?

A

◦ Ventilation
◦ Stocking density
◦ (Strategic antibiotic use in feed or water,
eg trimediazine, lincomycin or penicillin (pelleting reduces activity)

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22
Q

CLS of systemic strep suis?

A

Joint swelling
Lethargy
Inappetance
Seizure
Nystagmus
Sudden death

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23
Q

Diagnosis of systemic strep suis?

A

Post mortem examination
◦ Nothing
◦ Fibrin-polyserositis
◦ Endocarditis

Microbiology
◦ Heart blood
◦ Joint fluid
◦ Meningeal swab

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24
Q

What respiratory diseases do they get?

A

EP – Enzootic pneumonia – Mycoplasma hyopneumoniae
APP – Actinobacillus pleuropneumonia
PRRS – porcine reproductive and respiratory syndrome
Glassers – Glasserella parasuis
SI – swine influenza
AR – atrophic rhinitis – Pasteurella + Bordetella
PMWS – Porcine circovirus type 2
Coronavirus

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25
Resp CLS?
Coughing ±Increased resp. effort ±Nasal/ocular discharge ±Decreased appetite ±Condition loss ±Reproductive effects ±Death
26
Resp Diagnostics?
Blood samples --> ◦ Serology ◦ PCR Nasal/tonsillar swabs Post mortem examination --> ◦ Lung pathology ◦ Histopathology
27
What causes of Enzootic pneumonia?
Mycoplasma hyopneumoniae
28
What gross lesions of enzootic pneumonia?
consolidation of tips of apical + cardiac lobes
29
Abattoir score of lungs with Enootic pneumonia?
Abattoir score of lungs ◦ Apical (cranial) – 10 x 2 ◦ Cardiac (middle)– 10 x 2 ◦ Diaphragmatic (caudal)– 5 x 2 ◦ Intermediate (accessory) – 5 x 1 Max possible score = 55
30
CLS of Enootic pneumonia
Approx 80% UK herds infected Coughing, especially in older pigs Weight loss Reduced weight gain + FCR Pale pigs Increased mortality
31
EP Control?
Biosecurity ◦ keep it out! ◦ Vet to vet ‘conveyance’ is common ◦ Isolate incoming stock ◦ AI only Vaccination of piglets prior to, or at weaning Improve ventilation Reduce stocking density Medication ◦ Water - strategic ◦ Feed – continual or pulse medication
32
What is APP?
Actinobacillus pleuropneumonia
33
What gross lesions seen with Actinobacillus?
**Lesions in centre of diaphragmatic lobe** Area of intense haemorrhage and overlying pleurisy Looks like ‘liver’ Deep abdominal cough Reduced growth rates Sudden deaths, sometimes without coughing
34
APP signs?
Can cause chronic, ‘heavy’ coughing and reduced growth, or in some cases sudden deaths
35
APP infection ?
15 main serotypes, producing 3 different toxins Infection from dam, MDA lasts 5-6 wks, 10 wks with vaccination
36
APP Dx?
Diagnosed at PME / abattoir monitoring Culture on CBA with V factor to confirm
37
APP Control?
- Ventilation + stocking density - Medication of creep or grower – usually only short period required - Vaccination of sows or piglets - Autogenous vaccines if commercial vaccines not available for that serotype
38
Name of glassers dx?
Glasserella parasuis
39
Gross lesions of Glassers dx?
- Pericarditis and pleurisy - Polyserositis, so can affect all serous surfaces, joints, meninges, pericardium, peritoneum
40
when do we see glassers?
Often occurs following immune suppression, eg with PRRS or PMW
41
Control of Glassers?
◦ Vaccination ◦ Strategic medication ◦ Check environment, especially ventilation
42
Describe Atrophic rhinitis?
Nasal distortion - seen later in life Bordetella damages the nasal membrane allowing toxigenic Pasteurella to enter and damage turbinates
43
Atrophic rhinitis effects?
Infection of piglet causes sneezing Tear staining
44
How do we diagnose atrophic rhinitis at abattoir?
Monitor by snout section at abattoir Used to be common, now gone
45
is vaccine available for atrophic rhinitis?
yes
46
What is PRRS ?
porcine respiratory and reproductive disease Colloquially called ‘blue ear’, this is not pathognomic
47
Acute outbreak of PRRS causes ...?
◦ Sick sows, abortions, deaths ◦ Very high piglet mortality ◦ Increase in other diseases, especially respiratory
48
Endemic dx with PRRS causes ...
◦ Pneumonia, with lung congestion + oedema ◦ Monitor by serology
49
What can PRRS predispose to ?
Piglet scour
50
What will we often see with PRRS?
ear tip and snout necrosis
51
What happens with first outbreak on farm of swine flu?
Everything coughs, sows, piglets, growers Widespread inappetance Some deaths
52
What happens with endemic swine flu within a farm?
Waves of coughing in finishers, with periodic increases in mortality Severity depends on the strain ‘blue flu’ = PRRS + flu
53
What is PRDC?
Porcine Respiratory Dx complex
54
Describe PRDC?
MIXED INFECTION PRRSv Coronavirus Swine Influenza virus Circovirus (PCV2)
55
Sampling techniques for resp dx?
- Nasal swabs - Pharyngeal / tracheal swabs - Oral fluids
56
Describe nasal swabbing?
– virus isolation for ‘flu. (We don’t tend to use it for EP as it isn’t particularly sensitive). You might need smaller swabs – and it’s worth checking with the receiving lab that you are using the right type of swab and medium before you go).
57
Describe Pharyngeal/ tracheal swabs?
– testing for EP by PCR on throat swabs is more likely to give a positive result in the early stages of an outbreak. Not easy to do and requires either a high level of manual dexterity or another pair of hands and a serviceable pig gag
58
Orla fluids testing?
we can test for PRRS using oral fluids. Ropes are hung in the pens, the pigs allowed to chew them and then the fluids squeezed out of the ropes into tubes – tested via PCR.
59
Rectal Prolapse reasons
- Low fibre - 'grist' of food too fine - Overcrowing - Inadequate feed space - Cold - Excess coughing - More in males
60
Tx for rectal prolapse?
- If left in pen will become traumatised - Remove to recovery area - Can put into hospital – many will drop off on own - Surgical replacement pointless - May result in strictures – “barrel” pigs
61
Treatment ofor parasites?
- Dependent upon type of parasites - Injectable - ivermectin - In-feed – ivermectin, flubendazole - In-water – fenbendazole, flubendazole - Coccidiostats – oral dose / inject with piglet iron
62
What other conditions can they get?
Gastric ulcers Intestinal torsion Ringworm Clostridia Tail biting
63
Erysipelas?
-Skin ‘diamonds’ – often feel as much as see -‘diamonds’ may scab later -Pyrexia -Inappetance -Reduced fertility in sows
64
Erysipelothrix rhusiopathiae - consequences & Tx?
Sometimes skin lesions scab off May get endocarditis + joint lesions Treatment – penicillin by injection
65
Control of Erysipelas?
◦ Vaccination. Very cheap. Booster every 6months ◦ Can be given at weaning ◦ Maternal antibody lasts 10 – 12 weeks so may have to vaccinate finishers
66
Systemic signs of Erysipelas?
Lethargy Inappettance Pyrexia Skin lesions Joint swelling Congested extremities Sudden death
67
Diagnosis for Erysipelas
Clinical signs Post mortem examination ◦ Nothing ◦ Endocarditis ◦ Polyserositis Microbiology ◦ Joint fluid ◦ Endocarditis lesion ◦ Heart blood
68
What is Greasy Pig?
Exudative epidermitis Staph hyicus -> occurs in weaners and growers
69
When does Greasy Pig happen?
in association with ◦ poor ventilation ◦ Damp conditions ◦ fighting ◦ Skin damage, eg mange
70
Tx for Greasy Pig?
Penicillin, lincomycin by injection Topical disinfection NSAID’s B vitamins ‘marmite disease’ Managing environment
71
Pityriasis rosea?
Looks a bit like circular patches of ringworm Autosomal recessive Leave to recover
72
'sraw' rash ? (biting flies)
- SUMMER xarm humid - May only be a small rahs on live pig but quite severe after processing Tx: wait a week, NSAIDs Control: env + fly control
73
What is milk spot?
Ascaris suum
74
Describe Milk spot
- Very difficult to eradicate in a continuous flow scrape through system - Milk spot = exposure in previous 6 wks - Can cause total obstruction of small intestine in weaner/growers
75
Describe Ascarids lesions?
Lesions extends into substance of liver
76
What is important with ascarids?
it's the severity of milk spot that's important ie number of milk spots per affected liver
77
Hernias?
- Growth rate will be poor so sell early - Some abattoirs require FCI (= food chain information) cert if sent for slaughter - If skin is ulcerated, they are unfit to be transported
78
Hernia welfare prosecution?
if skin is ulcerated -> both owner and driver of transport
79
Mycoplasma arthritis
M hyosynoviae - Infection from dam to piglet; carried by piglet to finisher weight - Onset of disease often associated with stress -Sudden onset acute lameness
80
Tx for mycoplasma arthritis?
tylosin, lincomycin, tiamulin, NSAID
81
Kyphosis?
Congenital and progressive Leads to depressed growth Hereditary, so carefully monitor gilts Slaughter early
82
Excess mounting ?
Minimise disturbance during the day Maintain lower light levels – but within legal limits Reduced riding = improved welfare Split sexing in finishing phase Castration
83
What else can we do abotu excess mounting?
Improvac GnRH vaccination ◦ 2 doses 4 wks apart, ◦ 2 nd dose 6 wks prior to slaughter. ◦ Pigs finish one week earlier