Unit 7: 13-14 Somatic Motor System Flashcards

1
Q

What is the Motor Unit?

A

Muscles and neurons that control muscles
*Skeletal muscle
*Recruitment- turning on or off more motor units

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2
Q

What is the role of the Motor Unit?

A

generation of coordinated movements

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3
Q

What is the Spinal cord in control of in the motor system?

A

control of coordinated muscle contraction

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4
Q

What is the brain in control of in the motor system?

A

control of motor programs in the spinal cord

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5
Q

What is the Basal Ganglia (nuclei) do in the motor system?

A

initiate and terminate
*Parkinsons disease
*times overshoot or undershoot
*volition

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6
Q

What does the Cerebellum in the motor system?

A

coordinate
*proper fluid movement together

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7
Q

What are the 5 main parts of the motor system?

A

Motor Unit
Spinal cord
Brain
Basal Ganglia (nuclei)
Cerebellum

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8
Q

What is the direct motor pathway?

A

originates in the cerebral cortex.

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9
Q

What is the Corticospinal Pathway?

A

to the limbs and trunk

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10
Q

What is the Corticobulbar pathway?

A

to the head

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11
Q

What is the indirect motor pathway?

A

balance coordination, position
originates in the brain stem
not consciously aware, involuntary
*various mytones of the body

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12
Q

What pathways make up the indirect pathway?

A

Rubrospinal- tone of opposing muscle
Tectospinal- posturel of the head & neck
vestibulospinal- vestibular compensation
reticulospinal- series of midbrain nuclei (sleep to wake position) *sleep walking & paralysis

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13
Q

What pathways make up the direct motor pathway?

A

Corticospinal
Corticobulbar

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14
Q

What is Dysmetria?

A

the inability to control the distance, speed and range of motion necessary to perform smoothly coordinated movements

It can be seen in individuals with cerebellar damage due to brain trauma, brain tumors, metabolic diseases, and demyelinating or degenerative disorders.

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15
Q

What is ataxia?

A

Ataxia means without coordination.

People with ataxia lose muscle control in their arms and legs. This may lead to a lack of balance, coordination, and trouble walking. Ataxia may affect the fingers, hands, arms, legs, body, speech, and even eye movements.

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16
Q

What is kinesthesia?

A

Perception of movement over time
Kinesthetic sensation involves feeling the movement of limbs and joints. Kinesthesia examples include walking without looking at one’s feet, typing without looking at one’s hands, or moving limbs with one’s eyes closed.

17
Q

What are upper motor neurons?

A

Cell bodies in the cortex or brain stem
*Neural circuits involving BASAL GANGLIA and CEREBELLUM regulate activity of the upper motor neurons

(first-order neurons which are responsible for carrying the electrical impulses that initiate and modulate movement. Various descending UMN tracts are responsible for the coordination of movement. The major UMN tract that initiates voluntary movement is the pyramidal tract.)

18
Q

What somatic pathways are involved in upper motor neurons?

A

direct pathway
indirect pathway

19
Q

What are lower motor neurons?

A

Cell bodies in brain stem or spinal cord (SKELETAL MUSCLES)
retrieve info from upper motor neurons
*motor unit- need nerve on muscle fibers

(responsible for transmitting the signal from the upper motor neuron to the effector muscle to perform a movement. There are three broad types of lower motor neurons: somatic motor neurons, special visceral efferent (branchial) motor neurons, and general visceral motor neurons.)

20
Q

What is the lateral cerebrospinal fasciculus?

A

descussation of pyramids in medulla
*Corticospinal pathway

(The lateral corticospinal tract (LCST) is the largest descending motor pathway in the human body, it spans the entire length of the spinal cord, eventually supplying motor signals to all the skeletal muscles of our upper and lower limbs.)

21
Q

What is the anterior cerebrospinal fasciculus?

A

decussation in spinal cord
*corticospinal pathway

(The anterior corticospinal tract (also called the ventral corticospinal tract, “Bundle of Turck”, medial corticospinal tract, direct pyramidal tract, or anterior cerebrospinal fasciculus) is a small bundle of descending fibers that connect the cerebral cortex to the spinal cord.)

(The anterior corticospinal tract is an example of a descending motor tract. It comprises of upper motor neurons which helps adjust the posture by controlling the voluntary movements of the axial or truncal body musculature. Within the brain, it runs in close proximity to the lateral corticospinal tract.)

22
Q

What is Hyporeflexia?

A

Decreased muscle stretch reflex
*Damage to lower motor neuron
*FLACCID PARALYSIS
*same (IPSILATERAL) side
*corticospinal pathway

(Hyporeflexia happens when your skeletal muscles have a decreased or absent reflex response. An absent reflex response is also called areflexia. A reflex is an involuntary (automatic) action your body does in response to something. Reflexes protect your body from things that can harm it.)

23
Q

What is Hyperreflexia?

A

the presence of hyperactive stretch reflexes of the muscles.
*Damage to upper motor neuron
*SPASTIC PARALYSIS
*opposite (CONTRALATERAL) side
*corticospinal pathway

24
Q

Critical thinking question:

A person suffered a CVA (stroke) and now has
difficulty moving her right arm, and she also has speech problems.
What areas of the brain were damaged by the stroke?

A) Right parietal lobe
B) Left parietal lobe
C) Left frontal lobe
D) Right frontal lobe

A

C) Left frontal lobe

*Speech: left lateralized
*Right arm: left lateralized
*Frontal lobe: motor, broca’s area arm

25
Q

The Corticobulbar pathways is…

A

*Descending
*IPSILATERAL vs CONTRALATERAL
*Jaw devastates completely
* Skeletal muscle in the head (nine pairs of cranial nerves)

26
Q

Critical thinking question:

How will a doctor determine whether a patient with facial paralysis is due to lesion to upper motor neuron (stroke) or lower motor neuron (CN VII: facial nerve)? Is it a stroke or facial nerve paralysis (Bell’s palsy)?

A

*Stroke: paralysis –> bilateral (paralysis in random parts of face)
*Bell’s Palsy: lesions –> no muscles work (Facial nerve CNV II)

(To differentiate between upper motor neuron (UMN) and lower motor neuron (LMN) facial paralysis, doctors may conduct a thorough physical examination and use various diagnostic tests:

  1. Physical Examination:
    • UMN Signs: If the forehead is spared (due to bilateral UMN innervation), but the lower face is affected, it suggests UMN lesion.
    • LMN Signs: If the entire half of the face is affected, including the forehead (due to unilateral LMN innervation), it suggests LMN lesion.
  2. Electromyography (EMG): EMG can help determine the extent and location of nerve damage. In Bell’s palsy (LMN lesion), EMG typically shows decreased nerve conduction.
  3. Imaging:
    • MRI: Magnetic resonance imaging can help identify structural abnormalities such as tumors or strokes.
    • CT Scan: Computed tomography can also identify structural issues like fractures or tumors but may be less sensitive for strokes.
  4. Blood Tests: Blood tests can rule out infections or other systemic conditions that might cause facial paralysis.
  5. History and Symptoms: The timing of onset and associated symptoms (such as weakness in other parts of the body) can provide clues. Bell’s palsy often presents with a rapid onset of symptoms and may be associated with a viral illness.

In summary, a combination of physical examination, EMG, and imaging studies can help differentiate between UMN (stroke) and LMN (Bell’s palsy) facial paralysis.)

27
Q

Critical thinking question:

Bell’s Palsy vs Upper Motor Neuron Lesion?

A

*Stroke: paralysis –> bilateral (paralysis in random parts of face)
*Bell’s Palsy: lesions –> no muscles work (Facial nerve CNV II)

28
Q

*** add questions from objective and summary

A