TLO 2.9 Neurological/Sensory Motor Child Flashcards

1
Q

Glasgow Coma Scale

A
Standardized scale that assesses the LOC of infants and children
Assesses:
Eye opening: 1-4
Verbal response: 1-5
Motor response: 1-6
Higher score: intact infant
Lower score: coma
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2
Q

GCS

A

Patient with GCS of 3 is either dead or in vegetative state
GCS of 15 if normal
LOC is the best indicator to determine the depth of the comatose state
Children older than 3 should be able to state their name

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3
Q

Diagnostic test EEG

A

EEG identifies electrical activity of the brain

  • regular diet no caffeine or simulants
  • clean hair
  • sleep deprived night before test
  • painless
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4
Q

Diagnostic test CT scan

A

Identifies abnormal tissue structures, bleeding, hydrocephalus
IV if contrast used
Allergies
Sedate if necessary

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5
Q

Diagnostic test MRI of head

A

Sedated if agitated or afraid
Needs to lay still
Loud clicking noises
Head will be restrained, painless

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6
Q

Increased intracranial pressure IICP

A

A rise in pressure of the brain >20 mmHg.
Many causes of ICP
Brain tumor, hematoma
Infection, trauma
Overproductions of CSF, hypoxia (cause brain swelling)
Brain aneurysms
Brain damage prenatally or during birth (Cerebral Palsy)
Hydrocephalus, spina bifida, myelomeningocele

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7
Q

ICP assessment

A
Often have altered LOC with increased ICP. 
Use GCS
Nursing assessment:
LOC
Behavior
Pupil evaluation
Motor function
Vital signs
Developmental function, delayed milestones
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8
Q

Manifestations of ICP infant

A
Infant:
Poor feeding/vomiting
Irritability/restlessness
Lethargy
Bulging fontanel
High pitched cry
Increased head circumference
Cranial suture separation
Distended scalp veins
Eyes deviated downward
Increase/decrease pain response
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9
Q

Manifestation of ICP chidren

A
Child:
HA
Diplopia
Mood swings
Slurred speech
Altered LOC
N/V especially in AM
**head circumference is most significant neuro assessment for children <2
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10
Q

Assessment ICP Behavior

A

Behavior:
Behavior changes are important early signs of ICP
Involved parents ask questions
Inability to recognize parents, follow commands, minimal response to pain is deteriorating

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11
Q

Assessment ICP Pupils

A

Pupils:
ICP rises= 3rd cranial nerve compresses causing pupil dilatation and sluggish or absent constriction
Fixed dilated: bad suggests herniation of brains
Should be equal round reactive to light, accommodation (PERRLA)

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12
Q

Assessment ICP motor function

A

Motor function:
Purposeful movement will decrease

Abnormal posturing may be seen:
Decorticate: flexion upper extremities
Decerebrate: extension of upper extremities with internal rotation of upper are and wrist

Progression from decorticate to decerebrate indicates deteriorating neuro function

Flaccid paralysis indicates further deterioration in condition

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13
Q

Nursing interventions of ICP

A

HOB 30-40 degrees: facilitates venous draining reducing ICP
Avoid prone, supine position, hip flexion, neck flexion: this will increase ICP
Monitor for fever, seizures, hypotension, hypercapnia: this can increase ICP, avoid coughing
Block care reduces stimulation: too much stimulation can increase ICP, triggering seizure
Neuro checks: provides baseline
Monitor I/O: fluids affect ICP
Have emergency equipment: cardiac arrest

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14
Q

Seizures

three major classifications

A

Caused by excessive abnormal discharge of neurons
More than 2 seizures= epilepsy

Three major classifications of seizure:
Focal: 1 part of brain may/may not alter consciousness
Generalized: entire brain involved alters consciousness
Unknown

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15
Q

Seizures

Classified according to type

A

Tonic clonic (grand mal) generalized body involvement
Atonic: abrupt loss of postural tone drop head or drop to floor and get back up like nothing happened
Myoclonic: contraction and relaxation of muscles
Absence (petit mal): no muscle activity instead eyes roll upward
Focal: symptoms on one side of body often aura present prior to

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16
Q

Seizures predisposing factors

A
Predisposing factors:
Genetic defects
Strokes, trauma
Infections
Fevers >102
Hypoglycemia
Drug withdrawal
Intrauterine and perinatal infection
17
Q

Seizures diagnosis

A
Diagnostic
Family history
EEG, most helpful
Labs to rule out infection
CT
MRI
18
Q

Seizure assessment

A

Assessment
Body parts involved
Begin and end time of seizure

19
Q

Seizure NI

A
Nursing Interventions
Protect safety
NPO
Place side lying
Seizure precautions
STay with child
Call doc if over >5 min
Do not restrain
Loosen clothing
Teach caregiver about meds
20
Q

Seizure med

A
Medications:
Carbamazepine (tegretol)
Gabapentin
Phenobarbital
Phenytoin
Valproic acid
Levetiracetam (Keppra)
21
Q

Seizure treatment, daily meds

A
Phenytoin (Dilantin):
Oral care important
Causes gingival hyperplasia
Soft toothbrush
Floss after each meal
See dentist 3-6 mo

Valproic Acid (Depakote):
Monitor for generalized weakness
Do not crush/split med
Effects platelets, monitor for bleeding

  • *Most meds cause dizziness/drowsiness
  • *Do not stop med abruptly
  • *Monitor liver function
22
Q

meningitis

A

Inflammation of the meninges of the brain
Results from pathogen entering CNS and causing toxic response

Bacterial: medical emergency, droplet precautions 24 hour of antibiotic treatment

Viral: self limiting, lasts 7-10 days

Diagnosis: Lumbar puncture to obtain CSF

23
Q

Meningitis lumbar puncture

A
Insertion of spinal needle into subarachnoid space of lower spinal canal
Purpose:
Detect infection
Detect increase pressure
Insertion of medication

CSF findings:
Clear/colorless: normal
Cloudy/milky yellow: bacterial meningitis

24
Q

Lumbar puncture Nursing responsibilities

A

Informed consent
Child lying on side, knees to chest
Practice position with child
Monitor cardiorespiratory status during procedure
Lay flat 8-12 hours after to avoid spinal HA
Label specimens 1, 2, 3
Walk to lab

25
Q

Meningitis manifestations Infants/preschool

A
Poor feeding/poor sucking
Irritability/lethargy
Bulging anterior fontanel
V/D
Bulging fontanels/high pitched cry
Hyperthermia or hypothermia
Seizures
26
Q

Meningitis manifestations Children/adolescent

A
Severe HA
Photophobia
Nuchal rigidity
Fever
Altered LOC
Lethargy
Irritability
Anorexia/V/D
Drowsiness
27
Q

Meningitis Kernig and Brudzinski sign

A

Kernig: leg easily extended in supine position but when flexed toward abdomen pain presents

Brudzinski: supine child bends head forward to chest hip and knee flexion will involuntarily occur in meningitis

28
Q

Meningitis nursing care

A
Assist with LP
VS, neuro checks q 15 min
S/S of increase ICP
I/O
NPO if decrease LOC
Droplet precautions
Potential complications:
Hydrocephalus
Vision/hearing loss
Delayed G&D
Seizures

Prevention:
Meningococcal vaccine age 11-12 or by age 18
H. Influenzae B (Hib) vaccine as part of normal routine

29
Q

Viral meningitis

A

Most common type of meningitis
Less severe than bacterial
Self limiting, usually resolves by 7-10 days, some receive antiviral meds
S/S same as bacterial but less severe

30
Q

Common viruses that cause vial meningitis

A
Non-polio enteroviruses:
Mumps
Measles
Influenza
West Nile
31
Q

Spina Bifida what it is? types? risks?

A

Congenital neural tube defect where there is an incomplete closure of the vertebrae and neural tube during fetal development

Two types:
Spina bifida cystica (more severe)
Spina bifida occulta

Risks
Genetic predisposition
Folic acid deficiency

32
Q

Spina bifida results in?

A

90% lesions at or below L2

Results in:
Paralysis, partial paralysis
Varying sensory defects
Clubfeet, scoliosis, contractures and hip dislocations may also be seen r/t impaired intrauterine development
Hydrocephalus also can occur
73% develop latex allergies
33
Q

Spina bifida Occulta

A

Occurs between L5-S1 usually
Vertebrae fail to completely close
Usually no sensory or motor deficits

Clinical manifestation in lower lumbar or sacral area:
Dimple or small tuft of hair
Hemangioma or lipoma

34
Q

Spina bifida Cystica

A

More extensive defect with sensory and motor impairments
Visible defect seen on infant r/t failure of vertebrae to completely fuse

Meningocele:
Saclike protrusion containing meninges and CSF

Myelomeningocele:
Saclike protrusion filled with CSF, meninges, nerve roots and spinal cord (more extensive)

35
Q

Spina bifida Cystica nursing care

A

Sac protrusion measured and assessed
Head circumference, palpate fontanel (increased ICP risk)
Avoid pressure on sac, infant in prone position
Maintain foot and leg alignment, reduce tension on sac

36
Q

Spina bifida surgical care

A
Pre-op:
Prevent infection, cover sac
Prone position
Avoid fecal contamination
Maintain warmth
Adequate hydration (until NPO)
Post-op:
Monitor for hydrocephalus
Respiratory status
Wound care as ordered
Monitor incision s/s infection
37
Q

Hydrocephalus what is it?

A

Caused by imbalance between production and absorption of CSF
Excess CSF accumulates causing brain to be compressed against the skull
Skull becomes enlarged if sutures are open and s/s of increased ICP if sutures are closed

38
Q

Hydrocephalus risk factors

A
Risk factors:
Myelomeningocele
Neonatal meningitis
Intrauterine infection
Older children: tumor, hemorrhage, meningitis
39
Q

Hydrocephalus assessment infant

A
Infant assessment:
Rapid head growth
Bulging anterior fontanel
Irritability
Poor feeding
Distended scalp veins
Widely separated cranial sutures