TLO 2.3 Nutrition and Fluid Balance - Child Flashcards

1
Q

Cleft Lip/Cleft Palate
Structural defect?
Causes?

A

Structure:
Facial malformations that occur during embryonic development
Most common of congenital deformities of the head and neck
Most often have both deformities
Causes:
Interaction of multiple genetic and environment factors
-exposure to alcohol, smoking, anticonvulsants, steroids (10x increase with phenytoin)

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2
Q

Cleft Lip/Cleft Palate

First sign?

A

Occurs during 7-12th week of embryonic development
US during pregnancy can reveal deformity
Cleft lip obvious at birth
Cleft palate is discovered with exam

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3
Q

Cleft Lip/Cleft Palate

Problems?

A
Inability to suck
-imbalance nutrition
-imbalanced fluid/electrolytes
-breast/bottle feeding difficulties
-growth and development
-liquids escape through nose
Risk for aspiration
Risk for chronic otitis media and hearing loss
Parental anxiety and fears
Maternal-child bonding
-emphasize positive physical features
-emphasize post op outcomes
-do not focus on defect
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4
Q

Cleft Lip/Cleft Palate

Feeding

A

ESSR method of feeding: enlarged, stimulate, swallow, rest feeding

  • special bottle systems with enlarged nipples
  • hold infant in semi upright seated position
  • try to limit feeding to 30 min
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5
Q

Cleft Lip Palate

Post op care

A
Cleft lip repair: 3-6 mo old, revision by 5 yr
Focus on protecting operative site
-prevent crying
-elbow restraints
-keep on back/side position or infant seat
Analgesia for pain
Clear liquids when awake
Suture line meticulously cleaned
Antibiotic ointment
Gentle aspiration of mouth/nose prn
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6
Q

Cleft palate repair

Post op care

A
Cleft palate repair: 6-24 mo, <1 yr recommended
4-6 weeks healing
May position on abdomen
No suction or other objects in mouth
No hard foods
Parental support access information
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7
Q

Esophageal atresia

Suspected?

A

Any infant with unexplained frothy saliva in the mouth and unexplained episodes of cyanosis should be suspected in having esophageal atresia

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8
Q

Esophageal atresia with a tracheoesophageal fistula

what is it?

A

Esophagus terminated before it reaches the stomach and a fistula occurs that represents an unnatural connection with the trachea
Results in aspiration, respiratory distress and dehydration
Surgical repair needed

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9
Q

Esophageal atresia with a tracheoesophageal fistula

clinical signs and diagnosis

A
Clinical signs:
3 "C's" chocking, coughing, cyanosis with feeding
Excessive oral secretions
Vomiting
Unable to pass an NG tube at birth
Abdominal distention

Diagnosis:
X-ray, proximal esophagus dilated with air (atresia) or abdominal distention (fistula)
Bronchoscopy and endoscopy to assess fistulas

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10
Q

Esophageal atresia with a tracheoesophageal fistula

Surgical emergency

A
Repair to ligate fistula and anastomose esophagus
Priority: prevent aspiration
-NPO until repaired
-frequent suctioning oral
-NG w/ aspiration q 5-10 min (pouch empty)
-infant in warmer/humidified O2
-IV fluids
-HOB elevated to 30
-monitor VS, pulse ox, I/O
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11
Q

Esophageal atresia with a tracheoesophageal fistula

Post op nursing

A
Monitor respiratory status
Support fluid balance and nutrition
Maintain temperature, pain relief
monitor for infection
Promote bonding with parents
chest tube patency maintained
Assess lung sounds, suction prn
Accurate I/O, gastric tubes for feeding
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12
Q

Esophageal atresia with a tracheoesophageal fistula

Evaluation

A

Infant tolerating oral feedings with 3 “C’s”?
Infant gaining weight?
Surgical site without signs of infection?
Skin intact around G tube, chest tube, incision?

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13
Q

Esophageal atresia with a tracheoesophageal fistula

Parent care

A

Parents anxiety levels with care responsibilities
Demonstrate G tube feeding and care (tube bath in 1-2 wks)
Report drainage, leakage of formula, s/s of infection
Demonstrate skin care of wounds
CPR trained
Home care resources

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14
Q

Gastroesophageal Reflux and Disease: GER and GERD
What is it?
Causes?

A

Gastroesophageal Reflux: GER
Regurgitation of gastric contents back into the esophagus: 50% of infants <2 mo have GER, physiologic usually resolves by 1 yr

Gastroesophageal Reflux Disease: GERD (pathologic)
Represents resulting symptoms or tissue damage
Becomes a disease when complication occur
-growth failure
-bleeding
-dysphagia develops
-GERD is associated with: apnea, bronchospasm, laryngospasm and pneumonia

Causes: inappropriate transient relaxation of the lower esophageal sphincter (LES)

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15
Q

GERD s/s

A
Symptoms:
vomiting or spitting up after meals
hiccupping
apnea, coughing, choking
recurrent otitis media
weight loss/failure to thrive
irritable, discomfort, ad pain
severe cases: hematemesis or melena and anemia
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16
Q

GERD diagnosis

A

H/P
UGI series/barium swallow study
24 hr intraesophageal pH monitoring study- gold standard
endoscopy with biopsy, US, CT

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17
Q

GERD nursing interventions

A
Monitor airway
CPR education
Provide education on nutrition/fluids
Position upright after feeding, 1hr
Small frequent feeding (rice cereal)
Teach signs of dehydration and when to seek help
Observe for GI bleeding
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18
Q

GERD evaluation

A
Patent airway
Swallowing without respiratory distress
Retaining feeding with regurgitation of <10ml
Gaining weight
Balanced I/O
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19
Q

GER/GERD colaborative care and medications

A

Care:
No therapy if thriving and no resp complications
15% require surgery

Medications:
H2-receptor antagonists: Pepcid (famotidine), Zantac (ranitidine)
Proton pump inhibitors: Prilosec (omeprazole), Protonix (pantoprazole)

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20
Q

Gastroenteritis causes

A

Infectious agent in GI tract
Viral, bacterial and parasitic pathogens
Spread by fecal-oral route
-contaminated food or water
-spread person to person in close contact
**Giardia is the most common pathogen in daycare
**
Rotavirus is the most common viral cause of diarrhea (29% of deaths <5 yr)
Most severe in 3 mo to 24 mo

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21
Q

Gastroenteritis NI

A

Obtain accurate history, length of symptoms, frequency and consistency of stools blood/mucus
Assess for poor hydration
Maintain fluid balance: I/O, weight, electrolytes, K+, NA+
Maintain isolation: teach prevention of spread
No antidiarrheal
Report worsening symptoms: bloody stool, ab distention/pain, persistent elevated temp

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22
Q

Pyloric Stenosis

what is it?

A

Circular area of muscle around pylorus hypertrophies and obstructs gastric emptying
Most common symptom is progressive projectile vomiting (no bile)
Dehydration is most serious outcome
NI aimed at preventing dehydration

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23
Q

Hypertrophic Pyloric Stenosis

what is it?

A

Pyloric sphincter is thickened causing narrowing
-outlet obstruction and dilation, hypertrophy and hyperperistalsis of the stomach
Develops at 2-5 wk

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24
Q

Hypertrophic Pyloric Stenosis clinical findings

A
Projectile vomiting
Vomiting after feeding
Emesis is nonbilious
Dehydration
Metabolic alkalosis
Growth failure
Olivelike mass palpated when the stomach is empty
Lethargic
Eventually malnourished
25
Pyloric Stenosis diagnostic tools
``` History Clinical findings US of pyloric channel Upper GI if US negative Laboratory findings reflect fluid and electrolyte depletion ```
26
Pyloric Stenosis nursing assessment
Weight on admission H and P Observe for signs of dehydration Signs of electrolyte imbalance Ab assessment for distention, tenderness, bowel sounds, presence of pyloric mass Assess family for knowledge deficit regarding diagnosis, treatment, support, ability to participate in care
27
Pyloric Stenosis preoperative
``` NPO Weight, daily IV fluids VS I/O Labs Elevate HOB NG tube: amount, color, type of drainage Skin/mouth care Respiratory distress ```
28
Pyloric Stenosis therapeutic management and post op care
Pyloromyotomy: incision of pyloric muscle to release obstruction Laparoscopy: shorter surgical time, rapid postop feeding, quicker d/c ``` Post-op: feeding 4-6 hr post op small amounts of clear fluid breast/formula as tolerated monitor I/O daily weight ```
29
Celiac disease | what is it?
Chromosomal variation that result in child's inability to digest gluten (gliadin) Immune mediated enteropathy of proximal small intestine Resulting in severe GI mucosal changes that continue on exposure Teach about diet (no wheat, rye, barley, oats) Replace with corn or rice Vitamin supplements may be needed, especially fat soluble and folate
30
Celiac disease causes
Unknown Inherited predisposition with environmental factors Genetic markers is almost 100% of cases
31
Celiac disease pathophysiology
Inflammatory reaction is activated by gluten Cytokines released contribute to intestinal damage Fat, calorie, carb and vitamin deficiencies
32
Celiac disease diagnosis
Immunoglobulin A (IgA) deficiency, IgG is used Jejunal biopsy for ulceration in GI Monitoring reaction to a gluten free diet Symptoms often relieved in 1 wk after gluten free diet
33
Celiac disease clinical findings
``` 3-6 mo after introduction of grains to diet Diarrhea: steatorrhea (greater amounts of fat in feces, characterized by frothy foul smelling fecal that floats) Growth failure Ab distention Vomiting Anemia Irritability Anorexia Muscle wasting Edema Folate deficiency **Crisis: profuse watery diarrhea and vomiting ```
34
Celiac disease NI
Teach diet restriction (no wheat, barley, rye, oats) Monitoring of all food labels -gluten and lactose free diet -diet high in calories and protein with simple carbs -avoid high fat, high fiber foods Monitor weight, growth Physician monitoring: anemia, osteomalacia (softening of bones, def in Vit D and calcium) Teach disease process, life long condition Refer to support group
35
Minimum Urine Output
Infants/toddlers: >2-3 ml/kg/hr Preschoolers/young school age: >1-2 ml/kg/hr School age/adolescents: 0.5-1 ml/kg/hr
36
Infant nutrition | Breast feeding benefits
Bacterially safe Always fresh Correct temperature Maturation of the GI tract, immune factors lower gastroenteritis, neonatal necrotizing enterocolitis, celiac disease Antibodies against otitis media, resp illness: RSV, pneumonia, UTI, bacteremia and bacterial meningitis Decrease allergies Decreased incidence of SIDS Protective affect against childhood lymphoma/diabetes Enhanced cognitive development Analgesic effect for painful procedures (heel stick)
37
Infant nutrition | Formula kcals
Formula: 85-100 kcal/kg/day - birth to 6 mo: weight doubles between 4-7 mo - 6-12 mo: weight triples
38
Infant nutrition | Breastfeeding consideration
Vitamin D deficiency (risk for rickets) 400 IU of vit D: shortly after birth until taking 1 L/day of fortified formula Cobalamin (vit B12) deficiency Contributes to infant neurologic impairment Caused by inadequate maternal ingestion
39
Do not feed infants?
Goat's milk Skim or low fat milk Condensed milk Raw or pasteurized milk from any source
40
Infant | Solid foods
Consult with pediatrician prior to starting, usually around 4-6 mo Infants cannot easily digest foods Higher incidence of food allergy associated with early start Iron fortified cereal until 18 mo Introduce 1 food at a time Detect allergies/intolerance Finger food introduced 8-10 mo
41
Infant nutrition | Toddler considerations
``` Finger foods, 8-10 mo, encourage self feeding Healthy foods Nutritional snacks 2x day Growth rate slows Appetite decreases Weight quadruples by 2.5 yr Double height at age 2 Multivitamin only if poor dietary intake Nutritious foods- My Plate -fruits 100% juice, 4-6 oz/day -vegetables -whole grains -low fat dairy and nonfat dairy (milk 16-24 oz/day) -fish, beans, lean meats ```
42
Nutritional pre school considerations
``` Frequent small meals Healthy foods and snacks Focus on weekly food intake vs daily Increase Kcals Nutritional considerations same as toddlers Gain 2-3 kg per year Birth weight doubles by 4 yr ```
43
Nutritional adolescent considerations
Encourage food pyramid Monitor adequate intake Supplement as needed ``` Nutritional issues Milk replaced by soft drinks Fast food Body image Busy schedule Eat away from home more frequently Peer pressure ```
44
Pubertal growth spurt
Females: 10-14 yr Weight gain 7-25 kg 95% of height by menarche Males: 11-16 yr Weight gain 7-30 kg 95% of height by 15 yr
45
Physiological principles related to fluid and electrolyte balance in children
Younger the child, higher the proportion of water content in their body to body mass Younger the child, higher the proportion of extracellular fluid to intracellular body fluid Extracellular body fluid is more easily depleted than intracellular body fluid. This leaves the child at higher risk for fluid depletion when they get sick Younger children can't tell their caregivers that they are thirsty
46
Dehydration | Isonatremic
Primary form in children Electrolyte and water are lost in approx. equal amounts Loss from extracellular compartment -reduces the plasma volume and circulating blood volume -affects skin, muscles and kidneys Shock is greatest risk to life Sodium remains 138-145 mEq/L
47
Dehydration | Hyponatremic
Dehydration occurs when electrolyte deficit exceeds the water deficit (sodium <135 mEq/L) Shock if frequent finding
48
Dehydration | Hypernatremic
Dehydration result from water loss in excess of electrolytes loss Most dangerous and requires specific fluid therapy (intake of large amounts of solute or high protein NG feedings) Serum sodium >150 mEq/L
49
Assessment for dehydration key factors
``` History of acute or chronic fluid loss Clinical symptoms Weight Serum electrolyte levels -decreased bicarb, K+, glucose -sodium level may be normal in Isonatremic Urine specific gravity >1.020 ```
50
Dehydration management goal
Goals: correct fluid and electrolyte imbalance treat underlying cause
51
Dehydration NI
Teach parents prevention of dehydration -extra fluid during hot weather, avoid overdressing -rest periods after high energy play Treat fluid replacement (Pedialyte) -plan water in large amounts is dangerous Monitor weight Monitor I/O -stools/emesis: frequency, type, amounts, consistency Monitor VS
52
Food allergens | Two catagories
Food allergy or hypersensitivity - reactions involving immunologic mechanisms - usually immune globulin E (IgE) - reaction immediate or delayed - mild to severe= anaphylactic reaction Food intolerance - reactions involving known or unknown nonimmunologic mechanisms - i.e. lactose intolerance
53
Food allergy clinical manifestations
Systemic - anaphylactic - growth failure Gastrointestinal - ab pain - vomiting - cramping - diarrhea Respiratory - cough - wheezing - rhinitis - infiltrates Cutaneous - urticaria (hives) - rash atopic dermatitis
54
Food allergens diagnosis
``` Health history Elevated IgE Skin tests Radioallergosorbent test (allergy test done on blood) Anaphylaxis occurs in 5-30 min Asthma symptoms: wheezing, dyspnea Death can occur w/o treatment ```
55
Obesity defined
Body mass index >95th percentile for youth of the same age and gender Most common nutritional problem among US children
56
Obesity predisposing factors
Intake of high caloric, fatty foods Lack of outside physical activity Inactivity related to television, computers and video games -63% of 8-18 yr have TV in room, watching average of 4 hours per day -minority and low socioeconomic status populations watch over 4 hours/day
57
Obesity potential complications
``` Psychosocial Pulmonary GI Renal Musculoskeletal Neurological Cardiovascular Endocrine Diabetes HTN ```
58
Obesity NI
``` Parental education Do not use food as reward Scheduled meals/snacks Healthy choices Preplan meals Avoid fast food Be a role model Plan activities with children Routine pediatrician check ups ```