TLO 2.8 Musculoskeletal Child Flashcards

1
Q

What does the skeletal system do?

A

Provides structure and support
Protects vital organs
Provides movement
Storage space: blood cell production, regulates mineral and hormonal imbalances

**Bone structural disorders require follow up until child reaches skeletal maturity

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2
Q

Pediatric musculoskeletal difference

A

Skull sutures don’t fuse until 12-18 months
Muscle tissue almost completely developed at birth
Soft tissues are resilient making dislocation and sprains less common
Infants’ bones only 65% ossified at 8 months age
Fractures in <1 year child, uncommon, consider abuse

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3
Q

Diagnostic tests

A
X-ray
Bone scan
MRI
CT scan
**non-invasive
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4
Q

Diagnostic tests, X-ray purpose, viewing, nursing

A
Purpose: detect abnormalities or determine bone age
View:
Air looks black
Fat looks dark grey
Water looks light grey
Bone looks whitish
Nursing:
noninvasive
not generally NPO
place in gown
assist young children with proper positioning
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5
Q

Diagnostic test, bone scan, purpose, nursing

A

Purpose (investigates): trauma, early stress fractures, tumors, infections (diskitis, osteomyelitis)
Radioactive material given IV and scan occurs 3-4 hours later
Nursing:
encourage fluids 2-4 hr prior helps rid radioactive material
void before scan, improves pelvic bone visualization
young children may need sedation

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6
Q

Diagnostic test, MRI, purpose, nursing

A

Purpose:
Clearly define organ structures
Show changes in soft tissue (edema, infarcts)
Magnet and radio waves create energy field that’s translated to an image
Nursing:
No NPO
Ensure no metal on pt
Is loud, can take 1 hr
Assess claustrophobia
Must remain still (kids might need sedation, NPO is sedation)

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7
Q

Diagnostic test, CT scan, purpose, nursing

A
Purpose;
Visualize boney and soft tissue details
Has more radiation exposure
X-ray beams scan in successive layers and can be shown in 3D if needed
With or without contrasts
Nursing:
Not NPO
Scary for children, sounds like loud washing machine
Place in gown
NPO if sedation
Assess allergies for dye
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8
Q

What is a sprain and strains?

A

Sprains:
Joint trauma where a ligament is stretched or partially torn
Strains:
Known as pulls, tears ruptures, result from excessive stretch of muscle
**Both manifest as:
pain, swelling, localized tenderness, limited ROM, poor weight bearing, pop sound (sprain)

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9
Q

Dislocation, what is it?

A
Force of stress on the ligament results in displacement of the bone from it's socket
Pain and joint deformity results
Most common sites:
phalanges
elbow
hip
shoulders
**ligaments bind bones together
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10
Q

Management of sprains and strains

A

Primary goal is to reduce swelling and prevent further injury
RICE: rest, ice, compression, elevation
ICES: ice, compression, elevation, support
**swelling inhibits healing by keeping ligament ends separated. First 6-12 hours most important in controlling swelling and reducing muscle damage. Elevate above level of heart

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11
Q

Sprains and strains, nursing considerations

A
Analgesics:
acetaminophen
ibuprofen (controls pain and swelling)
Distraction and play can help with pain
Healing time depends on extent of injury
Weigh bearing gradually increased as pain decreases
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12
Q

Common causes of fractures

A

Accidental trauma; falls, MVA
Non accidental trauma; child abuse
Pathologic conditions; osteogenesis imperfecta, tumors cysts
**children heal faster than adults
**weakest point of long bones is cartilage growth plat or epiphyseal plate

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13
Q

Types of fractures

A

Greenstick: partial break on one side of bone while bone bows on other side, common in forearm
Spiral: twisted break/circular break seen in child abuse
Oblique: diagonal break
Transverse: break occurs at right angle
Comminuted: bones splintered into pieces rare in kids
Compound: bone exposed through skin

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14
Q

Manifestations for fractures in children

A
Pain/tenderness at site
Decreased ROM/immobility
Deformity
Swelling
Ecchymosis, muscle spasm
Inability to bear weight
Crepitus
Erythema
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15
Q

Diagnostic test for fractures

A

X-ray: both side view to compare

**distal forearm and clavicle common fracture sites

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16
Q

Fracture management, reduction, retention

A

Goal: maintain function without affecting growth
Reduction= align
Repositioning of the bone into normal alignment, open- surgically inserting devise (rod, pins)
Retention= hold
Application of a device to maintain alignment until healing occurs, splints, casts, traction, external fixation

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17
Q

Immobilization of fractures

A

Splints
Casts
Traction: manual, skin, skeletal
External fixation

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18
Q

Purpose of traction?

A

Reduce muscle spasm
Position distal and proximal bone ends in realignment
Immobilize the fracture site until realignment has been achieved and healing has taken place to permit casting or surgical fixation

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19
Q

Skin traction, what is it?

A

Noninvasive and well tolerated
Best for kids <15kg or <3 years of age
Applied to pelvis, spine, extremities (long bones)
Use external devices (foam, straps, bandages) applied to skin attached to pulleys and weights
Not appropriate if skin infections, open wounds, extensive tissue damage, abnormal sensations in extremities

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20
Q

Skeletal traction, what is it?

A

Exerts greater force than skin traction
Tolerated for longer periods of time
Maintains alignment of bony fragments and assists in proper healing
Traction maintained by metal device inserted into bone
Osteomyelitis most serious complication

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21
Q

External fixation device, what is it?

A

Used to treat:
Complex fractures
Length bones
Correct angular deformities involving bones and soft tissue

Allows for periodic changes in alignment with external device with pines inserted into bone
Infection is risk r/t pin sites, 50% develop problems

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22
Q

Assessment and care of immobilizing devices

A

Neurovascular status: 5 P’s

  • pain unrelieved by meds
  • pallor
  • pulselessness or lack of cap refill
  • paresthesia
  • paralysis
23
Q

Assessment of neurovascular status?

A
Neurovascular competency can be assessed using 5 P's or CMTS
Color
Movement
Temperature
Sensation
24
Q

What is osteomyelitis and risk factors?

A

Bacterial infection of bone

Open fractures/trauma
Soft tissue injury, cellulitis
External fixation or skeletal traction devices

Bacterial found: Staphylococcus aureus

25
Osteomyelitis manifestations
``` Fever, irritability, lethargy, feeding difficulties Pain, warmth, ,erythema Tenderness over site of infection Favoring affected extremity Limited ROM Painful joint with movement Odor possible ```
26
Osteomyelitis diagnosis and labs
MRI, CT, Bone scan, US Labs: Elevated ESR and CRP, WBC, blood culture
27
Osteomyelitis nursing interventions
Pain management Monitor and document neurovascular status Admin IV antibiotics Provide wound care Maintain nutritional status, high calories, protein, frequent small meals
28
Scoliosis, what is it?
Lateral deviation/curvature of spine >10 degree Curvature causes rotation of vertebral bodies of spine Worsening curvature leads to: Rib cage deformities= compromised respirator function Distortion of intrathoracic and abdominal organs that does not affect organ function generally
29
Scoliosis manifestations
``` Visible curvature of spine Rib hump when child bends forward Asymmetric rib cage Uneven shoulder or pelvic heights Scapular prominence Hip and leg length discrepancy ```
30
Scoliosis management
Curves <25 degree require long term monitoring | Curves >25 degree require brace treatment
31
Scoliosis nursing interventions for brace treatment
Doesn't correct curve but prevents progression Wear fitted t-shirt under brace to protect skin Remove for bathing, swimming, exercise only Re check size as child grows Analgesics for discomfort
32
Scoliosis surgical intervention
Recommended for curvatures reaching 40-50 degrees Goal is to reduce size of curvature and obtain solid fusion using metal rod and bone grafts (ribs, iliac crest, vertebral body) May not completely correct spine but stabilized spine that centers head, shoulders and truck over the pelvis
33
Scoliosis surgical interventions/complications
Significant blood loss common r/t vascular nature of vertebrae and spinal column Injury to spinal cord Infection
34
Scoliosis surgical pre op care and teaching
Routine teaching (resp, circulation, pain) Remain flat/log rolling technique Express feelings/anxiety PCA, foley, chest tube (if anterior approach)
35
Scoliosis surgical post op care
Neurovascular checks q 2hr for 48 hr then q 4hr Pain management Teach log roll, turn q 2hr Ambulate post op day 2-3 and d/c day 4-5 Respiratory status, inspirex, cough and deep breathing Encourage self care activities as appropriate Bed flat unplug bed controls Bowel function Wound healing/sit
36
Hip Dysplasia, what is it?
Developmental dysplasia of the hip (DDP) described variety of disorders that present different ages and forms -if present at birth called congenital -develops after birth called developmental Etiology: unknown Instability of hip joint occur as result of laxity of ligaments holding head of femur in acetabulum
37
Hip dysplasia risk factors
``` First bone child Female Breech position Low levels amniotic fluid in utero African and Chinese decent low incidences Native American high incidences ```
38
Hip dysplasia manifestations, older infants
Older infants: asymmetry of gluteal folds limited ROM in affected hop abduction present when supine with knees and hips flexed femur on affected side appears shorter s/s range from lax ligaments to contractures and stiffness in affected hip
39
Hip dysplasia manifestations, older child
``` Older child: Any abnormalities in gait need evaluation Child may: Limp Toe walking or waddling gait ```
40
Hip dysplasia management
Pavlick harness: provides hip flexion, abduction, external rotation Bryant's traction: releases muscles and tendons to manipulate hip joint Closed or open hip reduction surgery Spica cast post op immobilization
41
Hip dysplasia teaching
Don't straighten legs or swaddle together No car seats that restrict hip position Check skin under harness daily Check neurovascular status on feet Cast care, management elimination, keep dry Adapt clothing for weather Never leave child unattended, kids still move, safety issue Change positions q 2 hr How to apply Pavlick Harness and only remove as provider order Protect skin, use onesie
42
Legg-Calve Perthes disease, what is it?
Cause is unknown but accepted as growth disorder Involves femoral head progresses through 5 stages over 1-2 year period -epiphysis shows ischemia -reduction in size and increased femoral head density -bone weakens and dies causing femoral head collapse -avascular bone reabsorbed and begin to form new bone -femoral head reform result in final healing **disease is self limiting (runs its course generally)
43
Legg-Calve Perthes disease manifestations
Pain in hip worsens with movement Shortening of affected side, indicates collapsing of femoral head Muscle wasting of thigh and buttocks Unilateral hip involvement Permanent deformity if healing process is incomplete (femoral head protrudes outside acetabulum)
44
Legg-Calve Perthes disease diagnosis
X-ray Bone scan or MRI can revel necrosis and irregularity of femoral head **more common in boys than girls ages 4-9 year
45
Legg-Calve Perthes management
Initial treatment: anti-inflammatory meds ROM exercises and bedrest After 7-10 days if still unable to abduct hip: traction or abduction braces surgery to place femur securely into acetabulum
46
Clubfoot/talipes equinovarus manifestations
Plantar flexed foot with inverted heel and adducted forefoot unilateral or bilateral defect cannot be manipulated into neutral position readily apparent at birth
47
Clubfoot diagnostics and therapeutic management
Ultrasounds prenatally ``` Therapeutic: stared ASAP serial stretching, manipulation and casting surgery recurrence is common f/u until skeletal maturity ```
48
Clubfoot nursing care
``` Same as child with cast Clear parental instructions -bathing -time feeding with manipulations to allow distraction -encourage normal development ```
49
Muscular Dystrophy, what is it?
Progressive degenerative diseases that affect muscle cells or specific muscle groups causing weakness and atrophy. * *most identified early childhood * *includes >30 types but Duchenne MD is most common. affects limb and trunk muscles first
50
Muscular Dystrophy pathophysiology
Muscle fibers leak creatinine kinase and take on excess Ca+ causing more damage Muscles degenerate and replace with fat and connective tissue Muscle death causes weakness and wasting
51
Muscular Dystrophy manifestations
Progressive, symmetric muscle wasting and weakness without loss of sensation Symptoms become acute age 3-7 (hypertrophied muscles) Waddling wide based gait, can't climb stairs Gowers sign, use hands to walk up legs to stand erect Walking ability lost age 9-12 years Shortened life span r/t affect on GI, brain, cardio Cardiopulmonary complications common cause of death
52
Muscular Dystrophy diagnosis
EMG and muscle biopsy Genetic screening Physical findings Serum creatinine kinase (CK) elevated early stages of disease then decrease as muscle decreases
53
Muscular Dystrophy therapeutic management
Goal to maintain ambulation and independence as long as possible No effective treatment Manage symptoms Prevent contractures: ROM, bracing, PT, weight control