Spinal Cord Injury Flashcards
What is the most effective device for minimizing
cervical motion?
Halo-thoracic immobilization devices.
Hines RL, Marschall KE. Anesthesia & Co-Existing Diseases.
6th ed. Philadelphia, PA: Elsevier-Saunders; 2012: 256.
What is the median age of the patient who suffers a
spinal cord injury?
25
Nagelhout JJ, Plaus KL. Nurse Anesthesia. 5th ed. St. Louis,
MO: Elsevier Saunders Company; 2014: 924.
What is the leading cause of death in patients with
spinal cord injury at the scene of the injury?
respiratory failure from muscle weakness and aspiration
Nagelhout JJ, Plaus KL. Nurse Anesthesia. 5th ed. St. Louis,
MO: Elsevier Saunders Company; 2014: 924.
Fibers from what nerve roots innervate the
diaphragm?
C3, C4, C5
Hines RL, Marschall KE. Anesthesia & Co-Existing Diseases.
6th ed. Philadelphia, PA: Elsevier-Saunders; 2012: 258.
What is the incidence of cervical spine injury in all
major trauma victims?
Between 1.5% and 3% of all major trauma victims have a
cervical spine injury
Hines RL, Marschall KE. Anesthesia & Co-Existing Diseases.
6th ed. Philadelphia, PA: Elsevier-Saunders; 2012: 255.
The likelihood of a cervical spine injury is considered
minimal if what five criteria are met?
1) No tenderness over the midline cervical spine area, 2) no
focal neurologic deficits, 3) normal sensorium, 4) no intoxication
with drugs or alcohol, and 5) no other painful injury that could
distract from the patient’s evaluation of symptoms.
Hines RL, Marschall KE. Anesthesia & Co-Existing Diseases.
6th ed. Philadelphia, PA: Elsevier-Saunders; 2012: 255-256.
What are the symptoms of acute spinal cord injury?
Below the level of injury, the patient will exhibit flaccid paralysis,
total absence of sensation, and loss of temperature regulation
and spinal cord reflexes.
Hines RL, Marschall KE. Anesthesia & Co-Existing Diseases.
6th ed. Philadelphia, PA: Elsevier-Saunders; 2012: 255.
What are the symptoms of spinal shock?
Decreased blood pressure due to loss of preload (there is
significant dilation of the capacitance vessels). If the injury is at
or above T1-T4, the loss of sympathetic innervation to the heart
can result in bradycardia.
Hines RL, Marschall KE. Anesthesia & Co-Existing Diseases.
6th ed. Philadelphia, PA: Elsevier-Saunders; 2012: 257-258.
How does the skin of patients with spinal shock
compare to that of patients with hemorrhagic shock?
Because spinal shock disrupts the compensatory
vasoconstrictive response, patients with spinal shock will exhibit
warm, pink extremities whereas patients with hemorrhagic
shock will exhibit cool, clammy extremities.
Nagelhout JJ, Plaus KL. Nurse Anesthesia. 5th ed. St. Louis,
MO: Elsevier Saunders Company; 2014: 926
What monitoring techniques should be instituted for
patients with spinal shock?
In addition to standard monitors, an arterial line should be
placed to allow for moment-to-moment observation of blood
pressure as well as ease-of-access for arterial blood gas
analysis. A pulmonary artery catheter may also be used to
facilitate management of fluid and drug therapies.
Barash PG, Cullen BF, Stoelting RK, Cahalan MK, Stock MC,
Ortega R. Clinical Anesthesia. 7th ed. Philadelphia, PA:
Lippincott Williams and Wilkins; 2013: 1507
What is autonomic hyperreflexia?
Autonomic hyperreflexia may appear after spinal shock has
resolved and represents the return of spinal cord reflexes. It is
a massive sympathetic discharge that occurs in response to a
cutaneous or visceral stimulation below the level of transection.
In normal patients, the sympathetic activity that results from this
stimulation is overridden by inhibitory impulses from higher
central nervous system centers. In the spinal cord injured
patient, the sympathetic outflow is isolated from the inhibitory
feedback loop and the sympathetic discharge is unopposed.
Hines RL, Marschall KE. Anesthesia & Co-Existing Diseases.
6th ed. Philadelphia, PA: Elsevier-Saunders; 2012: 259.
With what spinal cord injury levels is autonomic
hyperreflexia most common?
About 85% of patients with a spinal cord injury above T6 will
exhibit autonomic hyperreflexia, but it is uncommon in patients
with lesions below T10.
Hines RL, Marschall KE. Anesthesia & Co-Existing Diseases.
6th ed. Philadelphia, PA: Elsevier-Saunders; 2012: 259.
What are the signs of autonomic hyperreflexia?
Hypertension, bradycardia, and dysrhythmias.
Nagelhout JJ, Plaus KL. Nurse Anesthesia. 5th ed. St. Louis,
MO: Elsevier Saunders Company; 2014: 927.
What types of stimuli may precipitate autonomic
hyperreflexia?
Bladder catheterization, defecation, childbirth, distention of the
bladder or rectum, and cutaneous stimulation.
Nagelhout JJ, Plaus KL. Nurse Anesthesia. 5th ed. St. Louis,
MO: Elsevier Saunders Company; 2014: 927.
What precautions can you take to prevent or treat
autonomic hyperreflexia during the perioperative
period?
Don’t allow stimulation from surgical incision or other
manipulation to occur before adequate general or regional
anesthesia has been established. Have immediate access to
short-acting vasodilators such as nitroprusside to treat sudden,
severe hypertension. Monitor the patient closely in the recovery
area as autonomic hyperreflexia may not occur until the effects
of anesthetic drugs wear off.
Hines RL, Marschall KE. Anesthesia & Co-Existing Diseases.
6th ed. Philadelphia, PA: Elsevier-Saunders; 2012: 259-260.