Respiratory Flashcards

1
Q

what is the most common cold?

A

rhinovirus

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2
Q

Respiratory syncytial virus (RSV)

A
  • seen in kids w/ asthma or those younger than 3 yrs
  • entry is through the nose or eyes
    -symp: rhinitis, clear secretions,
    -treated with decongestants
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3
Q

what is the difference between rhino sinusitis and sinusitis?

A

sinusitis causes purulent discharge while rhino causes clear secretions only

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4
Q

rhinosinutitis (sinusitis)

A

if longer than 3 months its considered chronic
-diagnosed with physical examination ,pain in the face or head when bending down
treated with abx

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5
Q

-replicates itself in the resp epithelium
-causes highest mortality in kids and adults
-there are two types
-treated with antiviral meds if before __ hours?

A

influenza
48 hours

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6
Q

type a vs type b

A

type a: is the most common and most severe, infects multiple species, and is divided into hemagglutinin, and neurominidase
type b: only in humans, less severe

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7
Q

Pneumonia
-infectiona of ____ resp. tract
-leading cause of death in ___
-infectious & noninfectious causes are:?
-what are the two classifications?

A
  • infection of the lower resp. tract
    -elderly
    -infectious cause: bacteria, viruses
    -nonin: gastic secretions in lungs
    -classficiations:
    -community ( up to 48 hours admitted) & hospital acquired
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8
Q

that is the most common pneumonia?

A

streptococcus pneumonia which can be typical or atypical

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9
Q

Atypical vs typical pneumonia

A

typical: worse than atypical, causes severe fever, lung consolidation, purulent sputum, detected in x ray. high remits and sounds like an “e” when auscultating
atypical: sputum in between the alveoli, dry cough, headache, “a” sounds clear

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10
Q

once tuberculosis moves onto different systems of the body what is it called?

A

disseminated or miliary tuber

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11
Q

tuberculosis

A

-nurses needs n95 mask, patient in negative pressure room
-will have ghon focus present which are granulomas and surrounding tissue
-no symptoms upon first infection but on second infection.

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12
Q

upon primary infection of tuber, the person is considered infectious?

A

false, only on the second infection; only infectious if person is immunocompromised

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13
Q

a primary infection will have a + ppd test with granulomas

A

true

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14
Q

TB test

A

-check for bumps
- if more than 15 mm= positive
>10mm= recent immigrants, iv drug users, nursing homes, health care, prisons
>5 mm=recent contact with TB person, HIV +, organ transplant

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15
Q

TB treatment

A

-INH, RIF (rifampin), PZA
latent infection: 6 months of INH
active tb: 2 moths of INH, RIF,PZA and 4 months of INH

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16
Q

chemoreceptors vs lung receptors

A

-chemo monitor O2 and CO2 levels and can adjust resp rate
-lung monitor breathing pattern
-central: in brainstem monitor CO2
-peripheral: in carotid and detect O2

17
Q

what is a persons respiration rate based off of

A

their O2

18
Q
  • blockage in alveoli, no O2 in tissue
    -alveoli have O2, but no capillary perfusion (pulmonary embolism)
    -blockage in alveoli and capillary
    -collection of fluid in pleural cavity
A

-shunt
-deadspace
-silent
-pleural effusion

19
Q

clear fluid vs creamy fluid w/proteins vs pus filled

A

these are types of pleural effusions
transudate vs exudate vs empyema

20
Q

pneumothorax

A
  • air gets trapped into pleural cavity
    -spontaneous- when it happens to young people
    -traumatic
    -open (communicating): air pressure equals barometric pressure
    -tension: theres a deviation in the trachea, so air enters during inhaling leaves
  • you will see tachypnea, dyspnea
21
Q

____ of lung
-causes: surge , pneumonia
-symp: dyspnea, cough, fever
-prevented by doing deep breathing, position changes

A

atelectasis

22
Q

Asthma
what is it?
what are the medications?
triggers?

A

-chronic inflammatory disorder; IgE attaches to mast cell
-SABA ( albuterol) short acting; used during attacks
-LABA (long lasting) used to prevent attacks
-coritcosteroids
-triggers: pollen, exercise, drugs

23
Q

look at notes for the types of asthma

A
24
Q

emphysema

A

-alveoli are destroyed, due to low elastin
-this happens because elastase breaks down elastin, seen in people with this genetic, or smoking
-symp: barrel chest, weight , low breath sounds, advanced cor pulmonale (RHF)
treatment: corticosteroids, inhaled anticholinergics

25
Q

chronic bronchitis

A

-air is trapped due to mucus so low O2;productive cough for 2 years
-causes advanced lung
-treatment: vaccines, same as emphysema

26
Q

what are emphysema people called?
what are bronchitis people called?

A

pink puffers
blue bloaters

27
Q

what does pulmonary HTN causes and what is it also known as?

A

it causes right sided heart failure, known as cor pulmonale

28
Q

pulmonary embolism

A

-DVT became dislodged; bone marrow leaks from bone breakage or amniotic fluid can reach mothers circulation
risk factor: virchows triad (hypercoagulativity, endothelial injury, venous stasis (a fib))
symp: chest pain , dyspnea, tachycardia
treated with TPA, inferior vena cava filter

29
Q

Cor pulmonale

A
  • due to RV hypertrophy from pulm HTN
    -high rbc’s to compensate
    -valve murmurs present
30
Q

is cor pulmonale RV and LV hypertrophy?

A

no only RV

31
Q

Acute respiratory distress syndrome ARDS

A

-proteins leak into capillaries of alveoli
-causes: near drowning, burns
-will see white lungs