Previous exam 2010 Flashcards
What is mechanism of action of Vasopressin, what receptor types, location, physiological effect
causes peripheral vasoconstriction which increases blood pressure
second messenger system–acts on peripheral vasculature
Antidiruetic
increases calcium
Outline 6 limitation for a language barrier for obtaining onset
.
CANMEDS role for CVICU full with only 2 discharges but 8 cases booked. How do you manage?
.
List 4 CANMEDS roles and how each applies to the above situation
MR valve morpholoy thrombus High wilkins score Calcification
Pt has a DDD AICD for LVEF and VT and needs a bowel resection. Gen Surg Consult
What happens when you put magnet on?
If the pt has VT while magnet on what should be done
If the pt has a DDD pacemaker , what mode is the pacemaker in while magnet is on
List 2 options you advise the Gen surg team to do regarding safe cautery use for this case
Magnet stops the anti-tach
Bipolar
magnet—
Pt has HR 140 after pacemaker implant (Dd pacemaker mediated tachycardia)
What are 3 conditions required for this diagnosis
1 parameter you can change
If you change this parameter what is the consequence
retrograde conduction
Must have atrial sensing
Dual chamber
Risk of AV block**
loss of av synchrony
Describe function of following pacing modes AOO AAI DDI DDD VOO
Pacing
Sensing
Respond to sensing
Identify 3 acute aortic syndromes
Acute aortic dissection
Intra mural hematoma
Penetrating athersclerotic ulcer
Patient is hypertensive, chest pain, CT diagnosis IMH
What is the mechanism
What is the natural history
What would you do
Bleeding in the media without intimal tear secondary to vasavasorum rupture
Spontaneous or secondary to plaque rupture
Can heal or progress to dissection
With acute presentation more chance that will progress into dissection so manage as a dissection
What are 5 surgical techniques to avoid SAM
Avoid excessive under sizing of annuloplasty
Ensure posterior leaflet height of less than 15 mm
Resection of excessive septal hypertrophy
Chords to anterior leaflet
mitral valve replacement
*Avoid inotropes
Avoid Hypovolemia
Avoid tachycardia
Increase afterload
Pt needs redo CABG with patent LITA, occluded OM, stenotic RCA. 5 adverse event you consent this patient
Death Stroke MI Bleeding AKI
Radial artery use.
What’s the 1 most important prognostic factor
>5 year patency rate: Radial to LAD? Radial to RCA?
Ensuring you have a high grade lesion to avoid competitive flow (greater 90% on right and > 70% on left
Endovein vs open vein
List 4 advantages
List 4 limitations
less morbidity
wound complications are less
time, patency,
List 3 risk factors for recurrence/residual lesion after coarctation repair
end to end repair
subclavian flap
aortic arch hypoplasia
Peforming a TGA and notice that neonatal cerebral infrared spectrometer has changed. You’re on CPB.
List 4 management steps
Change position of cannula
avoid kinking/check pressure
Arch dissection after completion of a ? on CPB. Assume central cannulation. with a clamp proximal to the innominate
Outline 5 management steps
Iatrogenic aortic arch dissection
3 distinct mechanisms how NO works
inhibits angiotensin II
Increase cGMP leading to vasodilatation
Improve oxygenation by dilatation of ventilated area
Anti platelet effect
Inhibition of proliferation of smooth muscle
3 potential limitations of NO administration
inhaled
toxic byproducts
abnormal
List 3 requisite anatomical features of complete AV canal defect
common av valve
lack septum
Pt with TOF and has developed severe PI years after initial repair. List 5 indication for pulmonary valve replacement
Right ventricular dysfunction
Right ventricular end diastolic
Severe PI
Symptoms of TR
Ischemic MR. Recent literature states certain subgroups benefit from reverse remodelling
List 4 preoperative predicators that suggest improved remodeling after CABG + IMR repair
LVEDD < 65
LVESD < 50
Low shericity index