Pathogenesis of the abomasal displacement, general and local consequences of abomasal displacement

Question 1

Pathogenesis of abomasal displacement

Answer 1

Nutritional abnormalities (e.g. milk production needs: grain↑, fibre↓)
↓
Increased VFA in abomasum
↓
Hypomotility or atony of the abomasum
↓
Dilatation of the abomasum
↓
Displacement (left or right)

Question 2

Local consequences of abomasal displacement

Answer 2

• For both LDA and RDA
    o Abomasal motility decreases
        ▪ Abomasal passage disturbed and/or stopped
        ▪ Reflux syndrome
    o Abomasal ulceration→peritonitis

Abomasal digestion ↓

Question 3

Local consequences of abomasal displacement

Reflux syndrome (“intraruminal vomitus”)

Answer 3

• Consequence in the rumen: H+ and Cl- increase (physiologic abomasal pH: 2-4)
• HCl rarely causes severe acidosis (HCl-acidosis), due to the buffering effect of saliva,
ammonia and proteins
• Cl- is not absorbable from the rumen; its concentration can be determined in the rumen fluid photometrically or by other machines→Normal value: <30 mmol/L