Hardware disease (traumatic reticuloperitonitis): clinical signs, local and/or systemic consequences, diagnosis and treatment Flashcards

1
Q

Local and/or systemic consequences

A

• Toxins, bacteria : Secondary disorders
o Nephrosis
o Myocardosis/myocarditis
o Arthritis
• Further penetration : complications
o Traumatic pericarditis, +/- pleuritis, pneumonia
o Traumatic splenitis, hepatitis, abomasitis
o Hoflund syndrome
o Seldom: coronary bleeding, subcutaneous abscess

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2
Q

Clinical (acute) signs in initial penetration

General appearance

A

sudden onset, sharp in milk yield! No movement because painful

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3
Q

Clinical (acute) signs in initial penetration

Posture

A
o Stretched head and neck
o Kyphosis (arching of the back)
o "Tucked up” (pulled-up) abdomen
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4
Q

Clinical (acute) signs in initial penetration

• Movement, behaviour

A

o Frightened glance,
o Slow, rigid, “cautious”, difficult stand up/turning,
o Defecating, urinating, lying down, getting up, and stepping

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5
Q

Clinical (acute) signs in initial penetration

sound (grunt)

A

o Stethoscope over the larynx or trachea
o Hand over the laryngeal area, but….
o More detectable in acute than in chronic (often subsides in chronic stage)!

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6
Q

Clinical (acute) signs in initial penetration

Basic clinical values

A

o T: 39,5-40,0 OC, often mildly increased
o P: 80 - 100/min
o R: >30/min, rapid, shallow, costal: too painful to breathe with the abdomen

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7
Q

Clinical (acute) signs in initial penetration

Digestive signs

A
  • Appetite, belching, rumination! (–)
  • Rumen motility! (–)
  • Rumen: small, firm, separated content, +/- mild tympany
  • Reticulum: reticular sounds! (–) missing: US to see if they are movement
  • Faeces: decreased, dry, poorly digested
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8
Q

Clinical signs in chronic cases

A
  • Feed intake and faecal output ↓
  • Milk production remains low
  • Abdominal pain become less apparent
  • T: usually returns to normal
  • Some cattle develop vagal indigestion syndrome (Hoflund syndrome) (particularly on the ventromedial reticulum is affected)
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9
Q

Clinical signs in case of pleuritis or pericarditis

A
  • Depressed
  • H: (>90 bpm) (severe tachycardia)
  • T: >40°C])
  • R: fast, shallow respiration
  • Pericardial friction rubs or gas and fluid splashing sounds (washing machine murmur): fluid accumulation in the pericardium so we can hear it
  • Jugular vein distention and congestive heart failure
  • Submandibular and brisket oedema
  • Prognosis is poor with these complications
  • Penetration of pericardium into the myocardium: sudden death!
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10
Q

Complication of traumatic reticuloperitonitis:

A

• Traumatic pericarditis
Drainage: exudate from the pericardium with fibrin, plasma, pus, bacteria, blood …
2 black lines: diaphragm at the last rib To calculate the place of puncturing

• Traumatic lienitis
o The spleen is on the upper left corner of the abdominal cavity: the reticulum is under it! so
if object vertically it can puncture the spleen

• The pin has penetrated even in the skin

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11
Q

Prognostic information for TRP

A
  • History and clinical findings
  • Abnormal general behaviour
  • Spontaneous grunting and groan
  • Sporadic onset
  • Sudden digestive disorder without change in feeding
  • Fever: signs of (acute) inflammation
  • Check the hay!
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12
Q

Methods for diagnosis

A
• Clinical examination methods: 
    o To elicit a grunt or groan
        ▪ Reticular pain probes and rectal sleeve
        ▪ Temporarily prevention of breath
    o Sensitivity of the wither’ss region 
        ▪ Kalchschmidt
• Haematology
• Rumen fluid
• Ferroscopy
• Abdominocentesis
• Laparoscopy
• Ultrasonography
• Radiography
• Endoscopy of the reticulum
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13
Q

Methods for diagnosis

Reticular pain probes

A

• Back grip:
o A fold of skin is pulled up with both hands, which forces the back to sink. The stretch will
be painful on the reticular region so excreted on the glands and the cow will grunt
• Pain percussion:
o Heavy rubber hammer toward the area of the reticulum (A) for percussion of the region
(B)
• Pole test:
o Two assistants hold the pole in place (A) and then pull the pole upwards against the cow’s abdomen (B)
o Can’t do it alone
o Start it cranially
• If the foreign body is suspected→at least the back grip, the pole test and pain percussion should be used
• Knee-elbow probe:
o You have to bring yourself into a sitting position and push your el on your knee and if you left up your hand you can pull your hands towards the reticular regions = incudes pain

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14
Q

Methods for diagnosis

Temporarily prevention of breathing

A

• Rectal sleeve placed over the mouth and nose temporarily prevent the animal from breathing
→ apnoea
→ strong diaphragmatic contractions
→ pain

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15
Q

Methods for diagnosis

Sensitivity of the wither region (Kalchschmidt)

A

• Touch, displace or pull the skin, or pull the hair gently

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16
Q

Methods for diagnosis

Haematology

A

• Haematocrit ↑
• Leucocytosis (neutrophilia): WBC number increases
• Acute cases: neutrophilia
o Differential leukocyte count is a better diagnostic indicator for peritonitis (only in the first three days) than the total leukocyte count commonly with a left shift
• Chronic cases: normalisation not, but moderate leukocytosis, neutrophilia and monocytosis
• Fibrinogen ↑
• Total protein↑: albumin + globulin +++
• Glutaraldehyde coagulation test (GCT) test is used to estimate the level of immunoglobulins
and fibrinogen but today more refractometry or biochemistry machine
• Gel formation: sensitivity (87.9 - 97.8%), coagulation time of 3 min and for 6 min

17
Q

Methods for diagnosis

Rumen fluid

A

• Results of rumen fluid analysis are not pathognomonic for TRP!

18
Q

Methods for diagnosis

Ferroscopy

A

• Metal detector: scanning of the ventral and lateral thoracic and abdominal wall → ferromagnetic foreign bodies
• False negative results:
o Dorsal penetration of the reticulum and nonmagnetic foreign bodies (copper or
aluminium)
• False positive results:
o Non-perforating ferromagnetic foreign bodies (bolt nuts) and magnets
• Compass: can be used to identify magnets that had been given previously by the producer or
veterinarian

19
Q

Methods for diagnosis

Abdominocentesis

A

• Under ultrasonic guidance: hypodermic needle or spinal needle with stylet
• Amount, colour, transparency, odour and consistency of the sample
• Refractometer: specific gravity
• Cytology and bacteriological examination
• Exudate:
o Cloudy, heterogeneous
o Echogenic sediment (bacteria and inflammatory cells) o Watery to viscous
o Smelly
o Flecks of fibrin
o Specific gravity >1.015 protein content >30 g/l

20
Q

Methods for diagnosis

Laparoscopy

A

• Useful for the detection of inflammatory changes
87
• Before removing foreign bodies: the area, degree and direction of penetration. This information → prognosis:
• Simple ventral penetration: favorable
• Deep anterior penetration: unfavorable (pericarditis)
• Cranio-medial penetration: questionable (vagus indigestion) = Hoflund syndrome

21
Q

Methods for diagnosis

Ultrasonography

A

• 3.5 to 5.0 MHz linear or convex transducer
• Ventral aspect of the thorax on both sides of the sternum and both sides of the lateral thorax
up to the level of the elbow
• Reticular motility: left ventral thoracic region and for 3 minutes without moving the
transducer

22
Q

Treatment and prevention of traumatic reticuloperitonitis

Treatment

A

• Conservative therapy
o Antibiotics (penicillin, oxytetracycline, ceftiofur)
o Diet
o Reticular magnet (?)
o Elevation of the cranial body, immobilisation
• Surgery
o Laparorumenotomy

23
Q

Treatment and prevention of traumatic reticuloperitonitis

Prevention (most important)

A
  • Removal of foreign objects

* Reticular magnet