Brainscape's Knowledge GenomeTM

Browse over 1 million classes created by top students, professors, publishers, and experts.


See full index

GI II > Oral cavity infections > Flashcards

Oral cavity infections Flashcards

1
Q

Glucomannan or mannoprotein are virulence factors of this pathogen facilitating adhesion

A

Candida albicans

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Function of aspartyl proteinases of C albicans

A

Hydrolyze host proteins involved in defense

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

Gram positive, oval, yeast-like budding cells with pseudohyphae. PAS positive and culture on SDA. Demonstration of germ tube.

A

C albicans

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

C albicans on CHROMagar

A

Light green/mint

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

C tropicalis on CHROMagar

A

Dark blue

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

C krusei on CHROMagar

A

Purple

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

Treatment of mild to moderate C albicans infections

A

Local application of clotrimazole, miconazole, or nystatin

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

Treatment of severe C albicans infection

A

Fluconazole PO or IV

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Characteristics of pseudomembranous type C albicans infection

A

Red erosions with a raw, bleeding surface when scraped

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Characteristics of erythematous type of C albicans infection

A

Flat, red, occasionally sore areas

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

dsDNA virus with an icosahedral core that replicates in the host nucleus and obtains its envelope form the host nuclear membrane.

A

HSV 1 and 2

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Spread of HSV from lytic to latent lesions

A

Syncytia formation

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Finding in host cell suggestive of HSV infection

A

Intranuclear inclusion bodies

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Virus that encodes a protein that down-regulates MHC-1 antigen presentation and CTL recognition

A

HSV

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Method by which HSV escapes immune system

A

Coats itself with IgG via Fc and complement receptors

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Typical clear lesions followed by ulcers of the lips, mouth, and/or pharynx

A

HSV

17
Q

Recurrent mucocutaneous lesions of the mouth and lips

A

HSV 1

18
Q

Treatment of HSV

A

Acyclovir
Valacyclovir

19
Q

Microscopy of Giemsa stain and TZANCK smear show multinucleated giant cells and Cowdry type A inclusion bodies

A

HSV

20
Q

Cell culture findings in HSV infection

A

Syncytium formation in 1-3 days

21
Q

Possible complication of HSV infection in immunosuppressed

A

Risk of disseminated life-threatening disease

22
Q

Non-enveloped ssRNA virus that is resistant to heat, detergents, and acids. Causal agent of hand-foot-mouth disease.

A

Coxsackie A virus (type 16)

23
Q

Transmission of Coxsackie A virus

A

Fecal-oral route
Direct contact

24
Q

Child presents with shallow yellow ulcers surrounded by red halos in the oral mucosa of the soft palate. Also has thick-walled gray vesicles with erythematous base on hands, feet, and/or buttocks

A

Hand-foot-mouth/Coxsackie A

25
Q

Medication to avoid in Hand-foot-mouth disease

A

Steroids

26
Q

Adolescent pt presents with fever, malaise, sore throat, painful swallowing, and tender vesicles at the junction of the hard and soft palate and on the tonsillar pillars.

A

Herpangina

27
Q

Causal agent of herpangina

A

Coxsackie A virus

28
Q

Vincent’s organisms

A

Fusobacterium species
Borrelia species

29
Q

Treatment of acute necrotizing ulcerative gingivitis

A

Penicillin and metronidazole
Dental hygiene

30
Q

Pt presents with abrupt onset of fever, malaise, severe mouth pain, and anorexia. Examination shows erythematous, edematous gingiva with friable necrotic punched-out craters in interdental papillae.

A

ANUG/trench mouth

31
Q

Whitish, corrugated, non-painful plaques on the lateral border of tongue

A

Oral hairy leukoplakia

32
Q

Causal agent of oral hairy leukoplakia

A

EBV

33
Q

Causal agents of dental caries

A

Viridans groups of Strep (mutans, salivarius, mitis, sanguis)

34
Q

Product of Strep viridans that adheres to teeth and results in plaque formation

A

Dextran

35
Q

Common causal organism of osteomyelitis of jaw

A

Actinomyces israelii

36
Q

Strictly anaerobic, non-acid fast, gram positive, filamentous, thin rods that colonize the upper respiratory tract, GIT, and female genital tract

A

Actinomyces israelii

37
Q

Initially hard, redish, non-tender tissue swelling over the jaw with fibrosis and swelling. Draining sinus tracts develop, containing sulfur granules

A

Osteomyelitis of jaw caused by actinomyces israelii

38
Q

First choice treatment for osteomyelitis of jaw

A

Penicillin
Drainage and debridement

39
Q

Secondary choices for treating osteomyelitis of jaw

A

Carbapenems
Macrolides
Clindamycin

GI II (31 decks)

Brainscape helps you reach your goals faster, through stronger study habits.
© 2024 Bold Learning Solutions. Terms and Conditions