Nutrition/Metabolism (final) Flashcards

1
Q

8 amino acids our bodies can’t produce

A

tryptophan, methionine, valine, threonine, phenylalanine, leucine, isoleucine, and lysine

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2
Q

fat soluble vitamins

A

A, D, E, K

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3
Q

vitamin A deficits

A

(retinol)

night blindness, dry skin, and embryo development defects in pregnant women

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4
Q

vitamin D deficits

A

(anti-rachitic factor)

rickets in children, osteomalacia in adults

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5
Q

vitamin E deficits

A

(anti-sterility factor)

hemolysis of RBC’s

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6
Q

vitamin K deficits

A

(coagulation factor)

bleeding and prolonged clotting times

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7
Q

vitamin C deficits

A

(ascorbic acid)

joint pain, poor tooth/bone growth, scurvy, and increased susceptibility to infection

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8
Q

vitamin B1 deficits

A

(thiamine)

Beriberi, vision disturbances, loss of memory, heart enlargement, tachycardia

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9
Q

vitamin B2 deficits

A

(riboflavin)

dermatitis, light sensitivity, and blurred vision

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10
Q

vitamin niacin deficits

A

(nicotinamide)

pellagra, photosensitivity, rough skin

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11
Q

vitamin B6 deficits

A

(pyridoxine)

anemia, seborrhea, and lesions around eyes/mouth

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12
Q

biotin vitamin deficits

A

scaly skin, muscle pains, elevated blood cholesterol levels

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13
Q

vitamin B12 deficits

A

(cyanocobalamin)

pernicious anemia and weight loss

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14
Q

folic acid vitamin deficits

A

(folacin)

megaloblastic anemia, GI disturbances, spina bifida, low birth weight, and neurological defects

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15
Q

calcium deficit

A

muscle tetany, osteomalacia, osteoporosis, rickets

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16
Q

chlorine deficit

A

alkalosis and muscle cramps

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17
Q

sulfur deficits

A

none known

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18
Q

potassium deficits

A

muscular weakness, paralysis, tachycardia, heart failure

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19
Q

sodium deficits

A

muscle cramping and covulsions

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20
Q

magnesium deficits

A

neuromuscular problems, tremors, irregular heartbeat, sudden cardiac death

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21
Q

phosphorus deficits

A

rickets and poor growth

22
Q

fluorine deficits

23
Q

cobalt deficits

A

vitamin B12 deficits (pernicious anemia)

24
Q

chromium deficits

A

adult diabetes

25
copper deficits
rare
26
iodine deficits
hypothyroidism, cretinism in infants, myxedema
27
iron deficits
iron deficiency anemia
28
zinc deficits
loss of taste and smell, growth retardation, depressed immunity
29
manganese and selenium deficits
not known
30
hyponatremia
not enough sodium neurological dysfunction, mental confusion, coma, twitching, irritability, decreased blood volume, decreased blood pressure, circulatory shock
31
hypokalemia
not enough potassium | cardiac arrythmias, cardiac arrest, alkalosis, hypoventilation
32
hypomagnesemia
not enough magnesium | tremors, convulsions
33
hypochloremia
not enough chloride | metabolic alkalosis due to bicarbonate retention
34
hypocalcemia
not enough calcium | tetany
35
what does insulin do
stimulates glucose uptake, amino acid uptake, glucose catabolism, glycogenesis, lipogenesis, protein synthesis inhibits gluconeogenesis
36
what does glucagon do
stimulates glycogenolysis, gluconeogenesis
37
what does epinephrine do
stimulates glycogenolysis, gluconeogenesis, lipolysis
38
what does growth hormone do
stimulates amino acid uptake, protein synthesis, lipolysis, gluconeogenesis
39
what does thyroxine do
stimulates glucose uptake, glucose catabolism, protein synthesis, lipolysis
40
what does cortisol do
stimulates lipolysis, gluconeogenesis, PROTEIN BREAKDOWN
41
what does testosterone do
stimulates protein synthesis
42
gluconeogenesis
converts amino acid and glycerol to glucose
43
normal blood glucose levels
90 mg/100 mL
44
if blood glucose levels go down
1. glucagon releasing cells of pancreas activated, release glucagon targeting the liver 2. liver breaks down stored glycogen and releases glucose to blood (glycogenolysis) 3. blood glucose levels rise
45
if blood glucose levels rise
1. insulin secreting cells of pancreas activated, release insulin in blood 2. liver takes up glucose and stores it as glycogen. body cells also start uptaking glucose. (glycogenesis) 3. blood glucose levels fall
46
cellular respiration steps
glycolysis, krebs cycle, electron transport chain
47
glycolysis
``` a catabolic pathway that occurs in cytosol oxidizes glucose (6 C) into two pyruvate molecules (3 C) ```
48
krebs cycle
a catabolic pathway that occurs in mitochondrial matrix | complete glucose oxidation by breaking down pyruvate derivative (acetyl CoA) into CO2
49
what do glycolysis and krebs cycle produce
a small amount of ATP by substrate level phosphorylation | NADH by transferring electron from substrate to NAD+
50
electron transport chain
located at inner membrane of mitochondrion - accept energized electrons from NADH and FADH2 - this process produces most of the ATP (90% 34 ATP)
51
after 1 turn of the krebs cycle
3 NADH 1 FADH2 1 ATP (2 carbons oxidized and leave as CO2)