Neuromuscular dysfunction with endocrine and or cardiovascular disorders Flashcards
What are some examples of neuromuscular disorders that could make O2 transport hard
- cerebral lesions: CVA, CP, TBI
- spinal cord: polio, SCI
- demyelinating disease: GBS, ALS, MS
- neuromuscular junction: myasthenia gravis
- failure of contracile mechanisms of muscles: myopathy
What can NM disorders reduce in reguards to CV and O2 transport
- respiratory mm strength/endurance (MG/SCI)
- inspiratory/expiratory presssure (keep airway open/cough issues)
- lung volumes and capacity
- flow rates
How can neuromuscular disorders contribute to respirtory issues
- can cause hypoventilation
- hypoventilation can lead to airway closure
- airway closure can lead to hypoexmia
- hypoexmia can lead to hypercapnia
How can NM disease along with cardio/respirtory issues implicate physical therapy
- optimize alevor ventilation
- airway protection
- patient/family education
- exercise prescription
- movement economy
- montior patient
Implications of NM disease with endocrine disease
- disorders of thyriod, pancrea, adrenal glands, and posterior pituitary gland
- additional weakness due to endocrine dysfunction
NM signs of thyroid dysfunction: hypothyroidism
- weakness
- lethary, impaired memory and cognition
- poor concentration
- paresthesia, muscle cramps
NM signs of thyroid dysfunction: hyperthyroidism
- confusion, seizures
- weakness
- tremor, chorea, athetosis (muscles are constantly functioning)
Neuromuscular signs wtih adrenal gland hyperfunction
- weakness
- emotional lability
- depression
neuromuscular signs with adrenal hypofunction
- fatigue
- weakness
- depression
neuromuscular signs with parathyroid hyperfunction
- weakness
neuromuscular signs with parathyroid hypofunction
- fatigue
- weakness
- chorea
- athetosis
- headache
PT implications for neuromuscular and endocrine dysfunction
- effectiveness of pharmacological interventions will impact PT tx
- exercise perscription
Neuromuscular disease and obesity
- alveolar hypoventilation
- increase mass of chest wall and abdomen
- increase energy to displace mass during respiration
- impaired disphragmatic excursion
- systemic and pulmonary HTN
neuromuscular disease and anorexia
- O2 transport deficits because of general debility and metabolis catabolism
- may see cardiac effects