Neurodevelopmental Flashcards

1
Q

Attention deficit/hyperactivity disorder

A

ADHD
maladaptive levels of excessive activity, inattention, and impulsiveness that interferes w/ functioning or development
3 to 1 boys to girls

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Two categories of symtpoms

A

inattention vs. hyperactivity and impulsivity (specify)
most symptoms before age 12, must have 6 as a child, 4 as an adult
must be across all situations

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

Causes

A

genetics (mixture), neurotransmitter dopamine
neurobiological- smaller brain volume
psychological and social factors- negative responses, poor self image

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Treatment

A

biological interventions
effects of medications
psychosocial intervention
comibined - recommended

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Biological interventions

A

stimulants vs. nonstimulants
trial and error approach
does ridilin work?

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

effects of medication

A

reduce hyperactivity and impulsivity

increase attention and focus

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

Psychosocial intervention

A

improve academic performance and social skills, decrease disruptive behavior
parent training
behavioral interventions before medications

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

Autism Spectrum Disorder

A

Persistent deficits in social communication, social intervention, and restricted, repetitive patterns of behavior/activities
DSM-IV separate disorders
DSM-5 spectrum

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Severity

A

level 1 requiring support
level 2 requiring substantial support
level 3 requiring very substantial support

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Neurocognitive disorders

Note* New chapter

A

affect multiple cognitive processes - learning, memory, consciousness
most develop later in life
three classes

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

Delirium

A

impared cognition and consciousness that develops rapidly, lasts hours to days
confusion, disorientation, attention/memory/language deficits
subtypes - basically etiology
causes- drug intoxication, drug withdrawal, head injury, high fever/infections
treatment- education, coping, reasurrance

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Major neurocognitive disorders

A

gradual deterioration of brain functioning, affects memory, language, judgement, and other advanced cognitive processes

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

mild neurocognitive disorders

A

new to DSM 5

focuses on attention to early stages of cognitive decline

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Initial symptoms

A

memory impairment, visuospatial skills deficits, agnosia (inability to recognize objects), facial agnosia

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

common side effects

A

delusions, depression, agitation, aggression

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

later symptoms

A

continued cognitive decline, assisted living activities

17
Q

Alzheimer’s disease

A

Involves distinct pattern of structural
decline in brain w/ multiple cognitive deficits.
Neurofibrillary tangles = bundles of twisted neurons.
Amyloid plaques = deposits of amyloid protein and clumps of
dead neurons.

18
Q

Neurocognitive Disorder Due to Alzheimer’s Disease

A

Develops gradually & steadily
Significant social & occupational impairments
Range of cognitive deficits
Difficulty w/ planning, organizing, or abstracting information

19
Q

The 3 A’s

A

Aphasia – difficulty w/ language
Apraxia – Impaired motor functioning
Agnosia – Failure to recognize objects

20
Q

Genetic influences

A

Chromosomes – 1, 12, 14, 19, 21
Chromosome 14 – early onset
Chromosome 19 – late onset

21
Q

Deterministic genes

A

Preseniln-1, Presenlin-2
β-amyloid precursor protein
APP gene on chromosome 21
APP produces amyloid in plaques

22
Q

Neurobiological influences

A
Neurofibrillary tangles
 Tau
 Amyloid plaques
 Mechanisms that may account for
 Amyloid precursor protein (APP)
 ApoE gene (ApoE2, ApoE3, ApoE4)
 ApoE4 on chromosome 19
23
Q

Psychosocial/social influences

A

Influence onset & course (not direct cause)

Lifestyle factors

24
Q

Treatments

A

3 areas of focus
 Prevent certain conditions
 Delaying onset
 Cope w/ advancing deterioration

25
Q

Psychosocial treatments

A
Main emphasis
 Coping strategies
 Behavioral interventions
 Implementing technology
Cognitive stimulation
 Teach adaptive skills
 Memory enhancement
 Memory wallet
26
Q

Biological

A

 Medication
 Cholinesterase-inhibitors
 New drugs target beta amyloid plaques