MT4 - Necrobacillosis and panaritium of ruminants (W) Flashcards

1
Q

Fusobacterium necrophorum subsp. necrophorum causes necrobacillosis

A

T

  • F. necrophorum subsp. necrophorum: more virulent
  • F. necrophorum subsp. funduliforme
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2
Q

Fusobacterium necrophorum subsp. necrophorum can help the agent of foot rot in causing
disease

A

T

diseases:
* necrobacillosis
* calf diphtheria
* liver „abscesses”
* mastitis
* gingivitis, metritis, peritonitis (dog, cat)
* facilitates foot rot (synergistic effect)

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3
Q

Aerosol infection is a common form of transmission of the agent of necrobacillosis

A

F

  • wounds on mucous membranes (oral cavity, genitals)
  • wounds on the skin
  • navel infection
  • rumen parakeratosis
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4
Q

Necrobacillosis is mainly seen in young animals

A

T

lambs, calves, rabbit

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5
Q

Rumen parakeratosis can predispose to necrobacillosis

A

T

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6
Q

Insufficient disinfection of the navel can predispose to necrobacillosis

A

T

  • wound
  • neglected disinfection of navel
  • change teeth
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7
Q

Abortion is a frequent clinical sign of necrobacillosis

A

F

  • necrosis (oral cavity)
  • fever, oedema, pain
  • salivation, smell
  • painful chewing, swallowing
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8
Q

Laryngeal erosion serves as place of entry for the agent of calf diphtheria

A

T

  • virus
  • cough
  • laryngeal erosion
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9
Q

calf diphtheria is caused by Dichelobacter nodosus

A

F

F. necrophorum

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10
Q

Calf diphtheria is caused by Fusobacterium necrophorum subsp. necrophorum

A

T

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11
Q

Respiratory diseases can predispose to calf diphtheria

A

T

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12
Q

Fusobacterium necrophorum subsp. funduliforme is the agent Necrobacillosis in lambs

A

T

  • F. necrophorum subsp. necrophorum: more virulent
  • F. necrophorum subsp. funduliforme
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13
Q

Necrobacillosis can occur as a consequence of navel infection in lambs

A

T

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14
Q

Pulmonary necrosis is a frequent lesion of necrobacillosis of lambs

A

F

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15
Q

Liver necrosis is a frequent lesion of necrobacillosis of lambs

A

T

necrosis in the liver in the case of:
- naval infection
- rumen parakeratosis

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16
Q

Fusobacterium funduliforme is the causative agent of Necrobacillosis

A

F

Fusobacterium necrophorum is the causative agent of Necrobacillosis

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17
Q

Necrosis of the mucous membranes in the oral cavity is a clinical sign of Necrobacillosis

A

T

  • necrosis (oral cavity),
  • fever, oedema, pain
  • salivation, smell
  • painful chewing, swallowing
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18
Q

Focal necrosis in the kidney is a frequent post mortem lesion of Necrobacillosis

A

F

necrosis:
- oral cavity, lips
- local oedema

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19
Q

Necrobacillosis is prevented with wide vaccination

A

F

  • disinfection of the navel
  • prevention of predisposing factors
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20
Q

Wounds can predispose to Necrobacillosis

A

T

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21
Q

Navel infection can occur in the case of Necrobacillosis

A

T

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22
Q

Lesions of Necrobacillosis can be seen frequently in the oral cavity

A

T

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23
Q

Rough, sticky feed can predispose to Necrobacillosis

A

T

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24
Q

Necrosis in the liver can be a post mortem lesion of Necrobacillosis

A

T

necrosis in the liver in the case of:
- naval infection
- rumen parakeratosis

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25
Necrobacillosis can only be seen in lamb
F | lambs, calves, rabbits
26
In the case of Necrobacillosis infection can happen through wounds
T | through wound or neglected disinfection of the navel, change of teeth
27
In the case of Necrobacillosis, aerogenic infection is common
F | wounds, navel infection, rumen parakeratosis
28
Lesions of Necrobacillosis are frequently found in or around the oral cavity
T
29
Fusobacteria are highly resistant, they remain viable in the environment for several weeks
F
30
Wounds and abrasions can predispose to Necrobacillosis.
T
31
Incorrect disinfection of the naval area can lead to Necrobacillosis
T
32
Wounds on the oral mucosa can predispose animals to Necrobacillosis.
T
33
The clinical signs of Necrobacillosis can frequently be seen in young animals
T | lambs, calves, rabbits
34
Necrobacillosis is a generalized disease with high fever and depression
F - fever, oedema, pain - salvation, smell - necrosis (oral cavity) - painful schewing, swallowing
35
Vaccines containing attenuated agents are widely used to prevent Necrobacillosis.
F | no vaccine, disinfection of navel , prevention of predisposing factors
36
Necrobacillosis is caused by Dichelobacter nodosus
F - F. necrophorum subsp. necrophorum: more virulent - F. necrophorum subsp. funduliforme
37
Footrot is the main sign of Necrobacillosis.
T? F. necrophorum causes footrot
38
The lesions in Necrobacillosis are restricted to the mouth
F
39
Necrobacillosis occurs mainly in young animals
T | lambs, calves, rabbit
40
There is no treatment for Necrobacillosis
F - surgery - disinfection - antibiotics
41
Umbilical disinfection has an important role in the prevention of Necrobacillosis.
T
42
Necrobacillosis in lambs can be seen as a result of an umbilical infection
T
43
Fusobacterium necrophorum subsp. necrophorum strains can cause Necrobacillosis of calves
T
44
You can find necrosis in the mouth in case of Necrobacillosis in lamb
T
45
You can find liver abscesses in case of Necrobacillosis
F
46
Foot rot is more severe in cattle than in sheep
F
47
The hoof can be detached in the case of foot rot
T | leg: hoof detached, foul smell, necrosis
48
Virulent foot rot can be treated with antibiotics
T virulent foot rot: - local treatment: surgery + antibiotic ointment - parenteral: penicillin, oxytetracycline, lincomycin, erythromycin
49
Foot rot occurs only in sheep
F sheep, goats, cattle, and occasionally other hoofed animals
50
The agent of foot rot can produce proteases; they are virulence factors
T virulence factors: - proteases - haemolysin (leukotoxin)
51
the causative agent of foot rot is dichelobacter nodosus
T
52
the main virulence factors of dichelobacter nodosus are toxins
F virulence factors: - fimbria - proteases, keratinase
53
the warm and wet environment can predispose to foot rot
T
54
Fusobacterium necrophorum subsp. Necrophorum can help the agent of foot rot in causing disease
T? synergetic bacteria: - F. necrophorum - P. melaninogenica - Spirochaetes, Streptococcus, Staphylococcus
55
Keratinsase production is a virulence factor of the agent of Foot Rot
T virulence factors: * fimbria * proteases, keratinase
56
Foot Rot cannot be prevented by vaccination
F Vaccine: 3 months long protection
57
Production of keratinase is a virulence factor of Dichelobacter nodosus
T
58
Morbidity of foot rot is very low, 1-5%.
F | morbidity ~100%
59
Foot bath in zinc sulphate can be used for the treatment of foot rot
T benign foot rot: foot bath - formalin - sterogenol - zink sulphate - copper sulphate
60
Dichelobacter nodosus causes foot rot in sheep
T | ruminants
61
Dichelobacter nodosus is helped by other bacteria in the pathogenesis of foot rot
T
62
Foot rot cannot be prevented with vaccination
F
63
Dichelobacter nodosus can produce keratinase
T virulence factors: - fimbria - proteases, keratinase
64
Foot rot has more severe clinical signs in goats than sheep
F
65
Exotoxins of the causative agent are responsible for the lesions of foot rot
F
66
Extracellular enzymes of the causative agent are responsible for the lesions of foot rot
T Extracellular enzymes produced by the causative agents, such as Fusobacterium necrophorum and Dichelobacter nodosus, play a crucial role in causing the lesions associated with foot rot.
67
Footbath with formalin can be used for prevention or for treatment of milder cases of foot rot
T benign foot rot: foot bath - formalin - sterogenol - zink sulphate - copper sulphate
68
Toxins are the main virulence factors of Dichelobacter nodosus
F virulence factors: - fimbria - proteases, keratinase
69
Warm and wet climate predispose animals to foot rot
T
70
The hoof wall can be detached in the case of foot rot
T - interdigital dermatitis - pain = lameness - lags behind the flock - leg: hoof detached, foul smell, necrosis
71
Animals with softened hoof wall are more susceptible for panaritium (foot rot).
T
72
Contaminated, dirty bedding is a predisposing factor for foot rot.
T predisposing factors: - warm, rainy weather - wet pastures, mud, stony soil - old bedding - missed foot trimming
73
In the development of foot rot, fimbriae of the pathogen have an important role
T virulence factors: * fimbria * proteases, keratinase
74
Treatment of foot rot is using foot bath containing formalin and sterogenol
T benign foot rot: foot bath - formalin - sterogenol - zink sulphate - copper sulphate
75
For treatment of panaritium, formalin foot baths are recommended.
T benign foot rot: foot bath - formalin - sterogenol - zink sulphate - copper sulphate
76
Foot rot is a very rare disease in Europe
F | widespread
77
Fimbria and extracellular enzymes are the virulence factors of the agent of foot rot
T
78
Dichelobacter causes Necrobacillosis
F - F. necrophorum subsp. necrophorum: more virulent - F. necrophorum subsp. funduliforme
79
The causative agent of foot root can produce exotoxins
F
80
The causative agent of foot root can produce enzymes
T
81
Foot rot can be prevented by foot bath
T
82
Animals with soft horn are more predisposed to panaritium
T
83
Strongly contaminated bedding is a predisposing factor to panaritium.
T
84
The fimbriae of the pathogens play an important role in the development of panaritium
T
85
To treat severe foot rot we use parenteral antibiotics
T - individual surgery, hoof paring - dry environment, clean bedding benign foot rot: - foot bath virulent foot rot: - local treatment: surgery + antibiotic ointment - parenteral: penicillin, oxytetracycline, lincomycin, erythromycin