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EPI PQ > MT4 - Avian Salmonellosis + Fowl typhoid (R) > Flashcards

MT4 - Avian Salmonellosis + Fowl typhoid (R) Flashcards

1
Q

Salmonella Infantis is most frequent serotype in the case of asymptomatic carriage in poultry

A

T

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2
Q

Salmonella reduction programs aim the reduction only for S. Enteritidis and S. Typhimurium in breeding poultry flocks

A

F

S. Enteritidis, S. Typhimurium + S. Infantis, S. Hadar, S. Virchow

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3
Q

Salmonella reduction programs aim the reduction only for S. enteritidis and S. typhimurium in broiler flocks

A

T

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4
Q

In the salmonella reduction programs ELISA tests are used to recognize the carrier animals.

A

F

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5
Q

Fowl typhoid occurs nowadays mainly in large scale farms

A

F

Found in backyard flocks, wild birds, zoo birds and pet birds

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6
Q

Atrophy of the ovaries is a postmortem lesion of fowl typhoid

A

T

pathology:
- Omphalitis, unabsorbed yolk sac
- Focal inflammation of heart muscle
- enteritis (haemorrhages, fibrin)
- inflammatory-necrotic foci: lungs, liver & spleen
- heart muscle necrosis
- enlargement of the lymphoid patches, ulcer
- atrophy of the ovaries

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7
Q

Fowl typhoid can occur in hens and chicken

A

T

  • poultry
  • (water fowl),
  • other bird species

at any age

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8
Q

Fowl typhoid is caused by Salmonella Avium.

A

F

S. Gallinarum / S. Pullorum

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9
Q

The agent of fowl typhoid does not kill the embryo

A

F

Ratio of dead eggs increases; hatchability drops

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10
Q

No postmortem lesions can be seen in the case of fowl typhoid

A

F

pathology:
- Omphalitis, unabsorbed yolk sac
- Focal inflammation of heart muscle
- enteritis (haemorrhages, fibrin)
- inflammatory-necrotic foci: lungs, liver & spleen
- heart muscle necrosis
- enlargement of the lymphoid patches, ulcer
- atrophy of the ovaries

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11
Q

Germinative infection can happen in the case of fowl typhoid

A

T

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12
Q

Animals infected with the agent of fowl typhoid can be recognised by using plate agglutination test

A

T

bacteriology: PCR
Antibodies:
- Serum plate agglutination
- Tube agglutination
- ELISA

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13
Q

Lesions of fowl typhoid is limited to the gut

A

F

affect various organs: liver, spleen, and other internal organs

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14
Q

asymptomatic carriage of salmonella by birds is generally limited to the gut

A

T

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15
Q

Focal inflammation and necrosis can be seen in the liver in birds with fowl typhoid

A

T

pathology:
- Omphalitis, unabsorbed yolk sac
- Focal inflammation of heart muscle
- enteritis (haemorrhages, fibrin)
- inflammatory-necrotic foci: lungs, liver & spleen
- heart muscle necrosis
- enlargement of the lymphoid patches, ulcer
- atrophy of the ovaries

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16
Q

fowl typhoid has a double peak death curve:

A

T

At young age: double mortality curve

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17
Q

fowl typhoid can be seen sporadically in zoo birds in Europe

A

T

sporadic:
- Asia, South America
- Backyard flocks
- Wild birds
- zoo birds and pet birds

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18
Q

omphalitis is a clinical sign of fowl typhoid:

A

T

Day-olds, young chicks:
- Weak,omphalitis
- diarrhea („white diarrhea”)
- Spread legs –> enlargened liver Respiratory signs
- arthritis

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19
Q

fowl typhoid is caused by salmonella gallinarum:

A

T

S. Gallinarum / S. Pullorum

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20
Q

arthritis can be a clinical sign of fowl typhoid:

A

T

Day-olds, young chicks:
- Weak,omphalitis
- diarrhea („white diarrhea”)
- Spread legs –> enlargened liver Respiratory signs
- arthritis

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21
Q

the agent of fowl typhoid can infect newly hatched chicken in the hatchery

A

T

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22
Q

The lesions of fowl typhoid are limited to the gut

A

F

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23
Q

Isolation of the agent of fowl typhoid from dead eggs prove infection of the herd with the agent

A

T

embrionic infection: Dead eggs, omphalitis

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24
Q

Fowl typhoid can be spread by aerogenic infection

A

T

Per os or aerogen infection, but usually spread through the ingestion of contaminated food or water

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25
Q

In the case of fowl typhoid there is a peak of death cases between days 3 and 5

A

T

At young age: double mortality curve
2‐5th days (vertical infection)
21‐28th days (horizontal infection)

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26
Q

Fowl typhoid is an acute disease of poultry

A

F

chronic

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27
Q

Fowl typhoid is caused by Salmonella Gallinarum

A

T

S. Gallinarum / S. Pullorum

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28
Q

Fowl typhoid is more frequent in water fowl than hens.

A

F

more common in chickens and turkeys rather than waterfowl

29
Q

The susceptibility to fowl typhoid is increasing with age

A

F

susceptible at any age

30
Q

Fowl typhoid can spread with germinative infection.

A

T

31
Q

Fowl typhoid has a death peak on days 3-5

A

T

At young age: double mortality curve
2‐5th days(vertical infection)
21‐28th days (horizontal infection)

32
Q

Fowl typhoid has no effect on hatching %

A

F

33
Q

Unabsorbed yolk sack is a post mortem lesion of fowl typhoid.

A

T

pathology:
- Omphalitis, unabsorbed yolk sac
- Focal inflammation of heart muscle
- enteritis (haemorrhages, fibrin)
- inflammatory-necrotic foci: lungs, liver & spleen
- heart muscle necrosis
- enlargement of the lymphoid patches, ulcer
- atrophy of the ovaries

34
Q

Fowl typhoid is a frequent disease in large scale farms

A

F

35
Q

Fowl typhoid is caused by Salmonella Enteritidis

A

F

Salmonella Gallinarum

36
Q

Aerogenic infection occurs in the case of fowl typhoid

A

T

Per os or aerogen infection

37
Q

Arthritis is a clinical sign of fowl typhoid

A

T

Day-olds, young chicks:
Weak, omphalitis
diarrhea („white diarrhea”) Spread legs – enlargened liver Respiratory signs
arthritis (legs and wings)

38
Q

Germinative infection is important in the epidemiology of fowl typhoid

A

T

39
Q

Salmonella gallinarum/pollorum is the causative agent of fowl typhoid.

A

T

40
Q

No post mortem lesions can be seen in the case of fowl typhoid

A

F

pathology:
- Omphalitis, unabsorbed yolk sac
- Focal inflammation of heart muscle
- enteritis (haemorrhages, fibrin)
- inflammatory-necrotic foci: lungs, liver & spleen
- heart muscle necrosis
- enlargement of the lymphoid patches, ulcer
- atrophy of the ovaries

41
Q

After recovery from fowl typhoid the animals do not remain carriers

A

F

can remain carriers of Salmonella Gallinarum, shedding the bacteria intermittently in their feces without showing clinical signs of the disease.

42
Q

Fowl typhoid generally does not occur in large scale poultry farms

A

T

Found in backyard flocks, wild birds, zoo birds and pet birds

43
Q

Salmonella avium is the causative agent of fowl typhoid

A

F

S. Gallinarum / S. Pullorum

44
Q

Fowl typhoid is mainly seen in large scale poultry farms

A

F

Found in backyard flocks, wild birds, zoo birds and pet birds

45
Q

Salmonella gallinarium is the agent of fowl typhoid.

A

T

46
Q

In case of Salmonella Gallinarum, germinative and aerogenic infection can happen.

A

T

47
Q

In case of fowl typhoid, the death curve peak at age 5-6 weeks

A

F

At young age: double mortality curve
2‐5th days (vertical infection)
21‐28th days(horizontal infection)

48
Q

In fowl typhoid there are two peaks during the epidemic

A

T

49
Q

Fowl typhoid can’t spread in a germinative manne

A

T

50
Q

Fowl typhoid can be diagnosed locally with agglutination methods.

A

T

bacteriology: PCR
Antibodies:
- Serum plate agglutination
- Tube agglutination
- ELISA

51
Q

Fowl typhoid is caused by Salmonella avium

A

F

S. Gallinarum / S. Pullorum

52
Q

The agent of fowl typhoid is shed in the faces in high number

A

T

53
Q

Asymptomatic carriage of Salmonella can occur in poultry

A

T

54
Q

Arthritis can be a clinical sign of Salmonellosis in birds

A

T

55
Q

Fowl typhoid have an exponential loss curve.

A

F

56
Q

Fowl typhoid can infect the ovaries.

A

T

atrophy of the ovaries

57
Q

White diarrhoea is a sign in young chickens with fowl typhoid.

A

T

Day-olds, young chicks:
- Weak, omphalitis
-diarrhea (white diarrhea)
- Spread legs – enlargened liver
- Respiratory signs
- arthritis (legs and wings)

58
Q

We can use slide agglutination to detect fowl typhoid.

A

F

bacteriology: PCR
Antibodies:
- Serum plate agglutination
- Tube agglutination
- ELISA

59
Q

We can use tetracyclines to treat fowl typhoid

A

T

Antibacterial treatment (economical aspects!) antibiotics:
- tetracyclin
- fluoroquinolones
- sulfonamides

60
Q

Fowl typhoid is caused by facultative pathogenic Salmonella

A

F

Obligate pathogen !!

61
Q

Rotting eggs are an important clinical sign of fowl typhoid.

A

F

62
Q

Dead eggs can be seen in fowl typhoid

A

T

63
Q

Fowl typhoid can be seen only in adult birds

A

F

any age

64
Q

Decreased hatchability is an important clinical sign of fowl typhoid

A

T

Ratio of dead eggs increases; hatchability drops

65
Q

Fowl typhoid propagates by germinative infection

A

T

66
Q

Fowl typhoid is more frequent in water fowl than hens.

A

F

67
Q

Fowl typhoid is usually a disease of waterfowl

A

F

68
Q

The highest infection rate of fowl typhoid is between days 8-10

A

F

2-5th days = vertical infection
21-28th days = horizontal infection

69
Q

The susceptibility to fowl typhoid increases with age

A

F

EPI PQ (81 decks)

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