Motor Control Systems Flashcards

1
Q

Reflex

A

A simple, highly stereotyped, and unlearned response to a particular stimulus.
Monosynaptic

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2
Q

Motor plan

A

A plan for action in n.s. (Ex crawling)

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3
Q

Closed-loop control mechanisms

A

Maximize accuracy. Provides flow of info from whatever is being controlled to the device that controls it.

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4
Q

Open-loop control mechanism

A

Maximizes speed. Feedback from the output of the system is not provided to input control. Makes ballistic movements.

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5
Q

Tendon

A

Tissue that connects muscles to bones

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6
Q

Antagonist

A

A muscle that counteracts the effect of another muscle

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7
Q

Synergist

A

A muscle that acts together with another muscle.

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8
Q

Flexion

A

Pull towards body

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9
Q

Extension

A

Pull away from body

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10
Q

Primary motor cortex (M1)

A

In most posterior part of frontal cortex.

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11
Q

Nonprimary motor cortex

A

Supplementary- lateral part of frontal lobe

Premotor cortex- medial part of frontal lobe

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12
Q

Fast-twitch muscle fibers

A

Act quickly and fatigue quickly.

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13
Q

Slow-Twitch muscle fibers

A

Slower to respond but have endurance

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14
Q

Neuromuscular junction(NMJ)

A

The point where the nerve transmits ya message to the muscle fiber

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15
Q

Innervation ratio

A

The ratio expressing the number of muscle fibers innervated by a single motor axon.

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16
Q

Final common pathway

A

Consists of all motoneurons in body. Receive and integrate all motor signals from the brain and then direct movement accordingly.

17
Q

Proprioception

A

Body sense. Info about the position and movement of body sent to brain.

18
Q

Muscle spindle

A

A muscle receptor that lies parallel to a muscle and sends impulses to the cns when the muscle is stretched.

19
Q

Intrafusal fiber

A

One of the small muscle fibers that lie within each muscle spindle.
Gamma motoneurons control

20
Q

Extrafusal fiber

A

One of the ordinary muscle fibers that lie outside the spindles and provide most of the force for muscle contraction.
Controlled by alpha motoneurons

21
Q

Primary sensory ending

A

Aka anulospiral ending.

The axon that transmits info from the central portion of a muscle spindle

22
Q

Secondary sensory ending

A

Aka flower spray ending.

The axon that transmits info from ends of a muscle spindle

23
Q

Golgi tendon organ

A

One of the receptors located in tendons that send impulses to cns reporting muscle tension. Sensitive to muscle tension, how much stress on muscle. Activate reflex to drop something if will cause damage.

24
Q

Pyramidal system

A

Brain, to medulla, to spinal cord, to muscle.

Includes M1, and nonprimary motor cortex. Movements represented. Learning occurs here.

25
Q

Supplementary motor area (SMA)

A

Rigoberto of nonprimary motor cortex that receives input from basal ganglia and modulates the activity of primary motor cortex.
Initiates movements, mental rehearsal of movements

26
Q

Extrapyramidal system

A

Doesn’t go thru pyramidal structures in medulla. Goes thru basal ganglia. Involved in initiation, amplitude and direction, habitual movements, addictions, OCD. Cerebellum- skilled movements, learned motor programs, classical conditioning

27
Q

Reticulospinal tract

A

One pathway of extrapyramidal system. Starts at reticular formation. Descend to spinal cord

28
Q

Rubrospinal tract

A

Starts in midbrain’s red nuc. Sends axons down spinal cord

29
Q

Muscular dystrophy

A

Dystrophin protein mutation. Gene on X chrom.

30
Q

Myasthenia gravis

A

Autoimmune disorder. Attacks ACh neurons, lose input to muscles, they weaken.

31
Q

Polio

A

Virus. Selectively affects motor neurons. Starts at periphery, works inward.

32
Q

ALS

A

Gradually worsening paralysis. Created locked-in syndrome.

33
Q

Parkinson’s disease

A

Loss of dopamine neurons. Affects pathways to basal ganglia. Causes tremors, unable to initiate movement, lack of facial expressions. Brain compensates for loss of DA at first, allows disease to progress a lot. Most environmental factors- not genetic.

34
Q

Parkinson’s treatment

A

L-Dopa- can help release DA, decrease symptoms for a while. Stem cell research - they deteriorate. Deep brain electrical stimulation.

35
Q

Huntington’s disease

A

Single gene disorder, repeated trinucleotide. Fatal. First affects emotional, then. Motor problems.

36
Q

Cerebellar damage

A

No cerebellum is ok. If have one and damage =ataxia, decomp of movements, jerky movements, tracking deficits in eyes.

37
Q

Apraxia

A

Impairment in ability to begin and execute skilled voluntary movements even though there is not muscle paralysis

38
Q

Paresis

A

Partial paralysis

39
Q

Spasticity

A

Markedly increased rigidity in response to forced movement or the limbs