lec 6 - ligament and joint injury Flashcards

1
Q

what is the structure of ligaments

A

dense bands of collagen tissue (less uniform than tendons)
- vary in size, shape, orientation, and location

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2
Q

what is the function of ligaments

A
  • connect bone to bone
  • passive stabilisation of joints (don’t have to activate)
  • important proprioceptive function
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3
Q

what is the response to ligament injury

A
  • decreased proprioception (nerve endings damaged)
  • leads to reinjury
  • ligament scars can creep (increased laxity)
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4
Q

what does joint stabilisation depend on

A

passive, active, and neural subsystems

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5
Q

what is the passive subsystem

A

non contractile connective tissues passively stabilising joint
- ex. bones, ligaments, cartilage, etc

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6
Q

what is the active subsystem

A

controlled by the neural subsystem to provide dynamic joint stability (protects joints from injuries)
- ex. muscles and tendons

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7
Q

what happens if load exceeds the active subsystems capacity

A

load gets transferred over into the passive subsystem
- when bone/ligament injuries occur

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8
Q

what is the difference between intra articular, extra articular, and capsular ligaments

A

intra articular - within the joint (ACL, PCL)
extra articular - additional support for the joint (MCL, LCL)
capsular - thickenings of the joint capsule (ankle ATFL)

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9
Q

what is the difference in blood supply in 3 types of ligaments

A

capsular = best blood supply
intra articular = worst blood supply

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10
Q

what is the ligaments adaptation to training

A

adapt slowly to increased loading but weaken very rapidly as a result of immobilisation
- loading = increased CSA = stronger ligament
- normal everyday training = maintain mech properties
- systematic training can increase ligament strength by 10-20%

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11
Q

on a stress strain curve, when will collagen fibres in the ligament start to rupture

A

when the force causes more than a 4% change in length

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12
Q

what category of injury is most likely to occur in ligaments

A

acute sudden event injuries (identifiable event - joint in extreme position)

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13
Q

can repetitive injuries occur in ligaments

A

yes - can occur as the ligaments gradually stretched out
- ex. UCL creep in pitchers leading to injury

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14
Q

what determines a grade 1 ligament injury

A

mild, structural damage on the microscopic level
- no instability

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15
Q

what determines a grade 2 ligament injury

A

moderate, partial tear, swelling and pain
- no / limited instabilty (laxity to varying degrees)
very large range - highly variable

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16
Q

what determines a grade 3 ligament injury

A

severe, full rupture, significant swelling
- instable

17
Q

what is hemoarthrosis

A

bleeding into the joint
- caused by ACL tear (70%), peripheral meniscus tear, osteochondral injuries or fractures

18
Q

what are the 2 bundles of the ACL and their functions

A

anteromedial bundle - resists tibial anterior translation (relaxed in extension, tight in flexion)
posterolateral bundle - resists tibial rotation (tight in extension, relaxed in flexion)

19
Q

what are the three physical exams you can do for an ACL injury

A

anterior drawer test - bent at 90deg, pull anteriorly on the tibia
lachman test - bent at 20-30deg, externally rotated, pull anteriorly on the tibia
pivot shift test - tests rotational stability of the ACL

20
Q

what is the unhappy triad of knee injuries

A

ACL, MCL, meniscus

21
Q

what are possible complications of ACL injury

A

osteochondral injury
osteoarthritis in 15-20 years (long term conseqeunces)

22
Q

what is the bucket handle meniscus tear

A

part of meniscus tears and flips over on itself and can’t come bakc
- leads to locked knee in flexion (needs surgery)

23
Q

when is an ACL graft weakest and when is reinjury most common

A

weakest = 3-6 months
highest reinjury = 2 years

24
Q

where do ACL grafts come from and why

A

hamstring graft
- common in calgary to preserve patellar tendon for downhill skiing
patellar tendon
- common in soccer players because you want to preserve the hamstring tendon in sprinting sports (hamstring tears are common)

25
Q

why don’t prevention programs work in the real world

A
  • poor athlete and coach buy in
  • need training programs implemented
    (ACL injuries are still on the rise - injury rates for girls haven’t changed in 20 years - 6x greater than boys)
26
Q

what are the modifiable risk factors for an ACL injury

A

training program
- weak hip abductors and external rotators
- increased knee abduction moments during cutting and landing

27
Q

explain the glenohumeral joint

A

one of the most mobile joints in the body
- passive stabilisation provided by coracoacromial and coracohumeral ligaments
- active stabilisation from rotator cuff and biceps

28
Q

what is dislocation

A

complete separation fo articulating bones

29
Q

what is subluxation

A

partial dislocation of articulating bones

30
Q

what is the common mechanism for shoulder dislocation

A

direct blow to the posterior of the shoulder
landing on outstretched arm (anterior and fracture dislocation)

31
Q

what is the risk of shoulder dislocation recurrence

A

50-90% depending on the type of sport and shoulder dominance