lec 19 - exercise in hot and cold Flashcards

1
Q

what is thermoregulation

A

process of maintaining a normothermic body temp

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2
Q

what are the 4 mechanisms of heat loss

A

radiation - heat loss directly to environment
convection - heat transferred from skin to colder air or water
conduction - heat loss via direct contact
evaporation - breathing and sweating

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3
Q

what does the body do it it needs to warm up

A

vasoconstriction (decreases blood flow to the skin)
thermogenesis

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4
Q

what are the two types of thermogenesis

A

non shivering - metabolic heat production asscoiated with metabolic burning of brown fat (adipose tissue)
shivering - generates heat as well
— non shivering occurs right before shivering

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5
Q

what does the body do it it needs to cool down

A

vasodilation (increases blood flow to tthe skin)
sweating

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6
Q

what are heat illnesses

A

result of increased heat production and impaired dissipation of heat
mild = heat edema, rash, syncope, cramps
major = heat exhaustion/stroke

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7
Q

what is heat edema

A

normal core temp
peripheral vasodilation to produce heat loss leads to pooling of fluid in extremities (soft tissue swelling)

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8
Q

what is the treatment for heat edema

A

elevation of extremities
hydration and salt intake
compression socks
no diuretics! - can make us lose more blood volume through urine
lasts 7-14 days

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9
Q

what is heat rash

A

normal core temp
sweating saturates the skin and clogs the sweat glands
- obstruction leads to leakage of sweat into the epidermis or dermis
blister like rash on skin

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10
Q

what is the treatment for heat rash

A

(risk of secondary infection from open wounds)
cool the skin adn prevent sweating
loose and light clothing
shower in cool water (let skin air dry)
mild anti-inflammatory lotion (not sunscreen)
lasts 2-3 weeks

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11
Q

what is heat syncope

A

normal core temp
occurs iwth orthostatic hypotension resulting from peripheral vasodilation and venous pooling
low BP = fainting

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12
Q

what causes heat syncope

A

standing still after exercise
older peopel with poorer blood flow

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13
Q

what is the treatment for heat syncope

A

supine in cool location
elevate legs
hydration

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14
Q

what are heat cramps

A

normal or elevated core temp
no clear cause (possible sodium / electrolyte depletion)
occur usually after 2+ hours of prolonged exercise
occur in the active muscle groups

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15
Q

what is the treatment for heat cramps

A

removal from cramps
hydration
light stretching / massage
cooling with ice
seek medical help if they don’t subside in an hour
no training for a few hours after the cramps subside

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16
Q

what is heat exhaustion

A

elevated core temp (37-40)
normal mental and neurologic status
sometimes tachycardia or hypotension
malaise, fatigue, dizziness, heavy sweating, headache, vomiting, cold or clammy skin

17
Q

what is the goal of heat exhaustion treatment

A

avoid progression to heat stroke
pull athlete asap and get symptoms calmed down so nothing progresses

18
Q

what is the treatment for heat exhaustion

A

removal from heat
supine position and elevate legs
cool the body (cold packs on neck, armpits, and groin)
hydration
seek medical help if not improvign within the hour

19
Q

what is heat stroke

A

elevated core temp (40+)
CNS disturbance - irritability, ataxia, confusion, coma, seizures
may or may not be preceded by mild symptoms

20
Q

what are the two types of heat stroke

A

classic = environmental heat waves (can be from physical exertion or not)
exertional = intrinsic heat production (not necessarily hot environment)

21
Q

what is the goal of heat stroke treatment

A

lower body core temp to <38.9 within 30 mins
(any longer can lead to long term consequences)

22
Q

what is the treatment for heat stroke

A

assess ABCs
reduce heat as quickly as possible (cold water immersion)
call medical assistance asap
cool first - transport second

23
Q

what are some complications from heat stroke

A

seizures
hypotension
arrhythmias
damage of vital organs (kidney/liver)
rhabdomyolysis (life threatening from the breakdown of muscle fibres and leaking their contents into the blood)

24
Q

what is the most important precursor to heat illnesses

A

dehydration - biggest risk factor

25
Q

what are the IOC acclimatisation guidelines

A

min 1 week (optimal = 2 weeks) of acclimatisation before competing in a hotter and more humid environment
- recommendation based off of where the individual is coming from

26
Q

what is wet bulb globe temperature (WBGT)

A

measure of the heat stress in direct sunlight
temp + humidity + wind speed + sun angle + cloud cover
can predict environmental heat stress

different from heat index (measured in shade)

27
Q

how are body weight and dehydration related

A

<2% BW loss = mild dehydration (occurs commonly in athletes and gen pop)
>3% BW loss = athlete should be restricted until BW recovers with hydration

28
Q

what is hypoatremia

A

too much water in the system
serum sodim levels <130 mmol/L
symptoms = nausea, vomiting, altered mental status, seizure, coma, death

29
Q

what is the RTS protocol for heat illnesses

A

mild forms = within 24 hours
heat exhaustion = next day if symptoms are no longer present (gradual return = ideal)
heat stroke = within 1 month

30
Q

when does hypothermia occur

A

when heat losses exceed heat production
core body temp <35
can also occur after hard exercise (vasodilation occurs for too long and the body loses too much heat)

31
Q

what is the body’s response to losses in heat

A

shiverign to increase metabolism and heat
superficial blood vessels constrict reducing skin heat loss

32
Q

what are the 3 stages of hypothermia

A

mild = feeling cold, shivering, incoordination, apathy
moderate = changes in behaviour and appearance, loss of shivering, confusion, slurred speech, decreased physiological functioning
severe = major metabolic and physiologic abnormalities, asystole by 18 deg

33
Q

what is the treatment for hypothermia

A

passive core rewarming
move to warm and dry place
remove wet clothing (use dry blankets)
don’t apply heat to extremities (can send cold blood to the core)
warm fluids

34
Q

what are the 3 stages of frostbite

A

frostnip = superficial skin is frozen
mild frostbite (superficial) = freezing of the skin and subcutaneous tissue
severe frostbite (deep) = freezing of the tisues below the skina dn adjacent tissues (muscle, tendon, bone, etc)

35
Q

what is superficial frostbite

A

involves dermis and/or shallow subcutaneous tissue
skin is red, white, cold, painful, blistering
usually recovers without permanent damage
increased susceptibility to cold injury

36
Q

what is deep frostbite

A

involves subdermal tissue
skin is white, grey, hard, insensitive, becomes black
leads to loss of tissue / body part

37
Q

what is the treatment for frostbite

A

rewarming with warm water immersion as long as there is no risk of refreezing
pain meds / antibiotics
important to check for other conditions (hypothermia)

38
Q

what are examples of external and internal risk factors for cold injury

A

external = cold weather, rain, wind, wet clothes
internal = previous cold injury, body size/comp, sex, medical conditions