Inner Eye Disorders Flashcards

1
Q

Thyroid Orbitopathy

occurs in who?

3 disorders

A
  1. Hyperthyroid (Graves)
  2. Euthryoid
  3. Hypothyroid
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2
Q

Thyroid Orbitopathy

presentation

A
  • bi or unilateral proptosis
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3
Q

Uveitis

describe

A

emergency inflammation of uveal tract

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4
Q

Uveitis

differentiate:
* anterior
* posterior
* intermediate
* panuveitis

A
  • anterior chamber structures
  • retina, choroid
  • vitreous body only
  • all structures involved
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5
Q

Uveitis

sx

6 components

A
  1. blindness, distorted/blurred vision
  2. painful
  3. redness
  4. photophobia
  5. uni or bi lateral
  6. acute, < 3 mo, sudden onset
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6
Q

Uveitis

PE Findings

6

A
  1. small/constricted pupil
  2. pupil reacts poorly to light
  3. iris becomes difficult to see
  4. redness
  5. WBCs settled in bottom of anterior chamber
  6. decreased IOP
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7
Q

Uveitis

tx

3 components

A
  • immediate referral to ophthalmology
  • steroids
  • exacerbated if give drugs for tx glaucoma
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8
Q

Uveitis

complications

4

A
  1. visual loss
  2. cyst-like formations on macula
  3. glaucoma
  4. cataracts
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9
Q

Episcleritis

describe

A

inflammation of the episclera which is the thin layer of vascular elastic tissue between the sclera and conjunctiva

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10
Q

Episcleritis

PE Findings

3 components

A
  • red, vascular injection of conjunctive w/ enlarged blood vessels beneath the conjunctiva
  • mild pain/discomfort
  • diffuse/nodular inflammation
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11
Q

Episcleritis

Tx

4 components

A
  1. refer to ophthalmology
  2. warm compress
  3. steroids (topical)
  4. NSAIDs (drops/PO)
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12
Q

Nystagmus

describe

2 components

A
  • involuntary rhythmic eye motion/oscillation
  • results from dysfunction in the vestibular system, brainstem, cerebellum
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13
Q

Nystagmus

describe vestibular nystagmus

A
  • results from dysfunction of the labryinth (Meinere’s disease), vestibular nerve, or vestibular nucleus in the brainstem
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14
Q

Nystagmus

signs/sx of vestibular nystagmus

5 components

A
  • nausea
  • vertigo
  • tinnitus
  • hearing loss
  • sudden head movements worsen sx
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15
Q

Nystagmus

describe jerk nystagmus

A
  • characterized by slow drifting off of a target followed by a quick corrective jerk
  • can be downbeat, upbeat, horizontal, or torsional
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16
Q

Nystagmus

signs/sx associated with jerk nystagmus

A
  • some pts are unaware they have it
  • others have: blurred vision, oscillopsia (environment oscillates)
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17
Q

Nystagmus

describe up and downbeat nystagmus

A
  • downbeat: occurs from lesions near the craniocervical junction (Chiari malformation, basilar invagination)
  • Upbeat: damage to the pontine tegmentum from troke, demyelination, or tumor.
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18
Q

Nystagmus

describe gaze evoked nystagmus

A
  • when the eyes are help eccentrically in the orbits, they tend to drift back to primary position
  • the pt will correct this, causing a quick oscillation to continue to target
  • normal in many pts in extreme vision fields
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19
Q

Nystagmus

what can exaggerate gaze-evoked nystagmus?

5 components

A
  1. drugs (sedatives, anti-convulsants, alcohol)
  2. muscle paresis
  3. myasthenia gravis
  4. demyelinating disease
  5. cerebellopontine angle, brainstem, cerebellar lesions
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20
Q

Strabismus

pathophys

A
  • problems w/ eye muscles, the nerves that transmit information to the muscles, or the control center in the brain that directs eye movements
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21
Q

Strabismus

describe the misalignment of the eyes

A
  • axis are not aligned
  • misaligned may be constant or intermittent
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22
Q

Strabismus

risk factors

3 components

A
  1. family hx
  2. refractive error (farsighted)
  3. medical conditions (down syndrome, stroke, cerebral palsy)
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23
Q

Strabismus

differentiate extropia and esotropia

A
  • extropia: deviating eye turns outward
  • esotropia: deviating eye turns inward
24
Q

Strabismus

differentiate hypotropia from hypertropia

A
  • hypotropia: deviating eye turns downward
  • hypertropia: deviating eye turns upward
25
# Strabismus asssocaited sx | 3
* diplopia (double vision) * suppression (diminished sensitivity within visual field of deviating eye) * amblyopia (eyes not working together)
26
# Strabismus describe accommodative esotropia
* due to uncorrected farsightedness causing eye to turn inward * sx: double vision, closing/covering one eye when using nearsighted vision, tilting/turning head
27
# Strabismus describe intermittent exotropia
* person cannot coordinate both eyes together * sx: HA, difficulty reading, eye strain, closed eye w/ farsightedness/sunlight
28
# Strabismus dx | 5
* corneal light reflex * cover/uncover test * retinal exam * ophthalmic exam * visual acuity
29
# Strabismus emergent causes? | 5
1. orbital fractures 2. cellulitis 3. tumors 4. meningitis 5. increased IOP
30
# Strabismus dx for emergent causes?
* orbital CT (ocular infiltrative process, tumors, abscesses, cellulitis) * Brain MRI (evaulate CN roots + brainstem)
31
# Strabismus tx | 4 components
* can spontaneously resolve as muscles strengthen * prism lenses (alter light enterance to eye to re-train them) * vision therapy * eye muscle surgery
32
# Amblyopia describe generally
* lack of development of clear vision in one/both eyes that cannot be fixed with glasses
33
# Amblyopia pathophys
* brain cannot combine the images from the eyes
34
# Amblyopia this is a lazy eye
just FYI
35
# Amblyopia causes | 3
1. early strabismus 2. early myopia/presbyopia 3. visual problems (ex: congenital cataracts)
36
# Amblyopia tx
* patching of "good eye" to force the lazy eye to work * vision therapy
37
strabismus vs amblyopia
* strab: cross eyed * ambl: lazy eye
38
# Glaucoma generally describe
abnormal flow of fluid around lens that causes increases in IOP and then damages the optic nerve causing diminished vision and eye irritation
39
# Glaucoma which type of glaucoma is usually asx?
chronic
40
# Glaucoma epidemiology | age, race, common
* increasing age * AAs at largest risk * family hx increases risk * chronic open angle is most common
41
# Open-Angle Glaucoma what increases IOP in open angle?
IOP is elevated due to reduced drainage of aqueous fluid through the trabecular meshwork
42
# Open-Angle Glaucoma goal for tx of open angle?
reduce the pressure by reducing the amount of fluid present in the eye
43
# Open-Angle Glaucoma sx/signs | 6 components
* no sx in early stages * progressive bilateral loss of peripheral vision * tunel vision * preserved visual acuities until advanced disease * cupping of optic discs * elevated IOP
44
# Closed-Angle Glaucoma predisposing factors | 3
1. shallow anterior chamber (farsightedness/short stature) 2. Asian/Inuits 3. enlarged lens
45
# Closed-Angle Glaucoma what causes angle closure?
pupillary dilation
46
# Closed-Angle Glaucoma associated sx
* HA * red eye * pain in eye
47
# Closed-Angle Glaucoma dx
* measure IOP (elevated) * gonioscopy (allows you to observe narrow chamber angle)
48
# Closed-Angle Glaucoma PE findings | 5
* red eye * cloudy cornea * pupil dilated, non-reactive to light * IOP > 50mmHg * globe firmness
49
# Closed-Angle Glaucoma complication of acute
severe and permanent vision loss if not treated within 2-5 days of sx onset
50
# Closed-Angle Glaucoma prophylaxis for acute
laser peripheral iridotomy to the unaffected eye
51
# Closed-Angle Glaucoma Tx | 4 components
* goal: reduce IOP * Acetazolamide * osmotic diuretics (glycerin, urea, or mannitol) if no response to acetazolamide * Pilocarpine drops after IOP decreases
52
# Closed-Angle Glaucoma surgical options | 3
* laser pepheral iridotomy * surgical peipheral iridectomy * cataract surgery
53
# Glaucoma which BB can be used? describe what is does?
* timolol * reduces aqueous humor which decreases IOP
54
# Glaucoma which topical prostaglanding agonist can be used? how?
* "prosts" * increases outflow of aqueous humor to decrease IOP
55
# Glaucoma which alpha-2 adrenergic agonist can be used? describe?
* brimonidine * dilates pupil to increase aqueous humor outflow and decrease IOP
56
# Glaucoma which carbonic anhydrase inhibitor can be used? describe?
* zolamides * decreases aqueous humor volume to decrease IOP
57
# Glaucoma glaucoma screening guidelines
* everyone at age 40 * age 40-60 w/out risk factors: every 3-5 yrs * age 40-60 w/ risk factors: every 1-2 yrs * age > 60 y/o: every 1-2 yrs