Emergencies Flashcards
hyperkalaemia management
10mL 10% calcium gluconate
50mL 50% insulin + 10U actrapid
10mL 10% calcium gluconate
50mL 50% insulin + 10U actrapid
hyperkalaemia management
potassium normal range
3.5-5
3.5-5 is the normal range for
potassium
paracetamol OD
NAC - N-acetyl cysteine, ABG, serum paracetamol >4h post-ingestion, LFT, U+E, PT, INR
NAC - N-acetyl cysteine, ABG, serum paracetamol >4h post-ingestion, LFT, U+E, PT, INR
paracetamol OD
sepsis 6: give IV fluids if low BP or lactate…
> 2mmol
sepsis 6: lactate >2mmol
give IV fluids
sepsis 6: do serial lactates
corroborate ABG + VBG lactate values, if >4mmol call critical care team
sepsis 6: corroborate ABG + VBG values, if >4mmol call critical care team
lactate
sepsis 6: fluid challenge, don’t exceed
30mL/kg
sepsis 6: don’t exceed 30mL/kg
fluid challenge
ABG PaO2 NR
11-13
ABG pH NR
7.35-7.45
ABG PaCO2 NR
4.6-6
ABG HCO3- NR
22-26
ABG 11-13
PaO2
ABG 7.35-7.45
pH
ABG 4.6-6
PaCO2
ABG 22-26
HCO3-
CSF bacterial meningitis
cloudy/turbid, elevated opening P, elevated WCC (polymorphs), low glucose, high protein
cloudy/turbid, elevated opening P, elevated WCC (polymorphs), low glucose, high protein
CSF bacterial meningitis
bacterial meningitis organisms in newborns
Listeria monocytogenes, E. coli, GBS
Listeria monocytogenes, E. coli, GBS
bacterial meningitis organisms in newborns
bacterial meningitis organisms in older children
Neisseria meningitidis, Haemophilus influenzae type B, Strep pneuoniae
Neisseria meningitidis, Haemophilus influenzae type B, Strep pneuoniae
bacterial meningitis organisms in older children
bacterial meningitis organisms in adults
Neisseria meningitidis, Strep pneuoniae, Listeria monocytogenes
Neisseria meningitidis, Strep pneuoniae, Listeria monocytogenes
bacterial meningitis organisms in adults
organophosphate poisoning antedote
pralidoxime
pralidoxime is used in
organophosphate poisoning
anticholinergic intoxication symptoms
flushed, aggitated, HTN, dilated pupils, tachycardia
flushed, aggitated, HTN, dilated pupils, tachycardia symptoms of what toxicity?
anticholinergic
agent used to reverse anticholinegic effects
physostigmine
physostigmine is used in
anticholinergic intoxication
ABG interpretation: resp acidosis
pH low
PaCO2 high
ABG interpretation: resp alkalosis
pH high
PaCO2 low
ABG BE NR
-2-+2
ABG -2-+2
BE
ABG interpretation: high flow 02
expect high PaO2
ABG interpretation: expect high PaO2
when PT on high flow O2
ABG interpretation: N PaCO2 in hypoxic asthmatic
sign they’re tiring, require ITU support
ABG interpretation: sign asthmatic required ITU support
hypoxic + N PaCO2
ABG interpretation: PaO2 on O2
10kPa < FiO2 e.g. PaO2 on 40% O2 = 30kPa
ABG interpretation: PaO2 <10
hypoxic
ABG interpretation: PaO2 <8
severely hypoxic = respiratory failure
ABG interpretation: hypoxia
PaO2 <10
ABG interpretation: severe hypoxia
PaO2 <8 aka respiratory failure
ABG interpretation: T1RF
PaO2 <8 + PaCO2 4.6-6
PaO2 <8 + N PaCO2
T1RF
ABG interpretation: T2RF
PaO2 <8 + PaCO2 >6
PaO2 <8 + PaCO2 >6
T2RF
ABG interpretation: resp acidosis with metabolic compensation
pH low/N
PaCO2 high
HCO3- high
ABG interpretation: resp alkalosis with metabolic compensation
pH high/N
PaCO2 low
HCO3- low
ABG interpretation: metabolic acidosis
pH low
HCO3- low
ABG interpretation: metabolic alkalosis
pH high
HCO3- high
ABG interpretation: metabolic acidosis with resp compensation
pH low/N
HCO3- low
paCO2 low
ABG interpretation: metabolic alkalosis with resp compensation
pH high/N
HCO3- high
PaCO2 high
pH low
PaCO2 high
resp acidosis
pH high
PaCO2 low
resp alkalosis
pH low/N
PaCO2 high
HCO3- high
resp acidosis with metaboic compensation
pH high/N
PaCO2 low
HCO3- low
resp acidosis with metabolic compensation
pH low
HCO3- low
metabolic acidosis
pH high
HCO3- high
metabolic alkalosis
pH low/N
HCO3- low
PaCO2 low
metabolic acidosis with respiratory compensation
pH high/N
HCO3- high
PaCO2 high
metabolic alkalosis with respiratory compensation
ABG interpretation: BE
high BE = suggestive of metabolic component 1’ or compensatory
syncope triggers
emotion, pain, exercise
emotion, pain, exercise can be triggers for
syncope
syncope duration
seconds
syncope may be preeceeded by
nausea, pallor, sweating
nausea, pallor, sweating may present pre-
syncope
seizure triggers
light, stress, EtOH
light, stress, EtOH may be triggers for
seizure
causes of resp acidosis
resp depression (opiates), GBS (inability to adequately ventialte), asthma, COPD
opiates, GBS, asthma, COPD may be causes of
resp acidosis
causes of resp alkalosis
anxiety (panic attack), pain, hypoxia, PE, pneumothorax
panic attack, pain, hypoxia, PE, pneumothorax may be causes of
resp alkalosis
causes of metabolic acidosis
use the anion gap to calculate: +ve ions - -ve ions
increased acid producion/ingestion (raised anion gap) vs decreased acid secretion/HCO3- losses (low)
NR anion gap
4-12
high anioin gap indicates
increased acid production/ingestion: DKA, lactic acidosis, aspirin OD
DKA, lactic acidosis, aspirin OD may be causes of
metabolic acidosis, raised anion gap
low anion gap indicates
reuced acid secretion/renal/GI HCO3- losses: diarrhoea, ileostomy, proximal colostomy, renal tubular acidosis, Addison’s DZ
diarrhoea, ileostomy, proximal colostomy, renal tubular acidosis, Addison’s DZ may be causes of
metabolic acidosis with a reduced anion gap
causes of metabolic alkalosis
diarrhoea, vomiting, loop + thiazide diuretics, HF, nephrotic S, cirrhosis, Conn’s
diarrhoea, vomiting, loop + thiazide diuretics, HF, nephrotic S, cirrhosis, Conn’s may be causes of
metabolic alkalosis
causes of T2RF
COPD, pneumo, rib #, obesity, MND, GBS, opiates
COPD, pneumo, rib #, obesity, MND, GBS, opiates are all causes of
T2RF
T1RF causes
pulmonary oedema, bronchoconstriction (asthma), PE
pulmonary oedema, bronchoconstriction (asthma), PE are causes of
T1RF
Cullen’s sign
periumbiical oedema + bruising suggestive of retroperitoneal bleeding
periumbiical oedema + bruising
Cullen’s sign, suggestive of retroperitoneal bleeding
Grey-Turner sign
bruising on the flanks bx, suggestive of retroperitoneal bleeding
bruising on the flanks bx
Grey-Turner sign, suggestive of retroperitoneal bleeding
McBurney’s sign
deep tenderness over McBurney’s point (1/3 -/- ASIS to umbilicus), suggestive of acute appendicitis
deep tenderness over the point 1/3 -/- ASIS to umbilicus)
McBurney’s sign, suggestive of acute appendicitis
Rovsing’s sign
palpation of the LLQ causes pain in RLQ, suggestive of acute appendicitis
palpation of the LLQ causes pain in RLQ
Rovsing’s sign, suggestive of acute appendicitis
needlestick injury immediate managment
correct disposal of sharps, squeeze wound, wash under tap, don’t scrub
needlestick injury risk assessment of transmitted blood
safe sex, IVDU, country of origin, tattoos/piercings abroad, previous STIs, blood transfusions abroad
if a high risk needlestick start PEP within
1h
risk of HIV transmission /1000 needlestick injuries
3
duration of PEP
28/7
tests after needlestick injury
3/12 later = HIV, Heb A + C
PEP SE
N, V, diarrhoea, headaches, dizziness
post-needlestick precautions
document in notes, incident form, occupational health, no exposure prone proceedures, protected sex, no blood donations
ecstasy clinical features
aggitation, anxiety, confusion, ataxia, tachycardia, HTN, hyponatraemia, hyperthermia, rhabdomyolysis
management of ecstacy
supportive, dantrolene (for hyperthermia)
aggitation, anxiety, confusion, ataxia, tachycardia, HTN, hyponatraemia, hyperthermia, rhabdomyolysis describe the clinical features of
ecstacy
features of opioid misuse
rhinorrhoea, needle tract marks, pinpoint pupils, drowsiness, watering eyes, yawning
complications of opioid misuse
viral infection (HIV, hep B, C) 2’ to sharing needles, bacterial infection (IE, septic arthritis, septicaemia, necrotising fasciitis) 2’ to injecting, VTE, respiratory depression, cravings, crime, prostitution, homelessness
management of opioid OD
IV naloxone
rhinorrhoea, needle tract marks, pinpoint pupils, drowsiness, watering eyes, yawning are features of
opioid misuse
neonatal resuscitation at birth
dry baby, remove wet towels, start the clock
neonatla resuscitation w/i 30”
assess tone, RR, HR
neonatal resuscitation w/i 60”
if gasping/not breathing open airway + give 5 rescue breaths
paediatric basic life support: unresponsive start with
5 rescue breaths
paediatric life support: compressions ratio
15:2 (if >2 rescuers, if not 30:2)
CI to thrombolysis
active internal bleeding, recent haemorrhage/trauma/Sxs, coagulation/bleeding disorders, intracranial neoplasm, stroke <3/12 ago, aortic dissection, recent head injury, pregnancy, severe HTN
thrombolysis examples
altepase, tenecteplase, streptokinase
SE of thrombolysis
haemorhage, hypotension
active internal bleeding, recent haemorrhage/trauma/Sxs, coagulation/bleeding disorders, intracranial neoplasm, stroke <3/12 ago, aortic dissection, recent head injury, pregnancy, severe HTN are CI of
thrombolysis
altepase, tenecteplase, streptokinase are examples of
thrombolytic agents
sinister headache (2+ symptoms do a CT head)
V >1 w/o cause, new neuro deficit, reduced GCS, coughing/sneezing/positional headaches, progressive headache with fever
classification of croup
mild = occasional cough, no stridor, mild effort of breathing, well child moderate = frequent cough, stridor at rest, recession, mildly stressed child severe = frequent cough, prominant stridor, marked recession, significant distress, tachycardia, hypoxia
croup management
ABCDE, 0.15mg/kg PO dexamethasone, high flow O2, nebulised adrenaline
choking management
“are you choking?”, A: encourage cough (if mild), 5 back blows, 5 abdo thrusts, repeat, BCDE
NR for anion gap
10-18
N anion gap in metabolic acidosis
GI HCO3- loss (D, ureterosigmoidostomy, fistula), RTA, acetazolamide, NH4Cl injeciton, Addison’s
[ABCD = Addison’s, Bicarb loss, Cl, Drugs]
raised anion gap in metabolic acidosis
lactate (shock, hypoxia, burns, metformin), ketones (DKA, EtOH), urate (renal failure), acid poisoning (salicylates, MeOH)