ECG 2 Flashcards

1
Q

location V5-V6

A

anterolateral wall

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2
Q

location 2, 3 Avf

A

inferior wall

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3
Q

location of I, aVL

A

high lateral wall

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4
Q

prolonged QT interval if the

A

QT interval is more than half the RR interval

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5
Q

Causes of prolonged QT interval

A
  1. Hypocalcemia
  2. hypokalemia
  3. hypomagnesemia
  4. Class 1A or 3 anti-arrhythmic drugs
  5. Hypothermia
  6. Congenital Long QT syndrome
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6
Q

hypercalcemia effect on QT interval

A

shortened

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7
Q

hypocalcemia effect on QT interval

A

prolonged

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8
Q

in hypokalemia

A
  1. prolonged QT interval
  2. T wave merges with the U wave
  3. u waves frequent
  4. T wave may be inverted
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9
Q

in mild hyperkalemia

A
  1. Tall T waves

2. Peaked & Symmetrical

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10
Q

in moderate hyperkalemia

A
  1. P &R waves flatten,
  2. QRS & T broaden,
  3. Big S waves develop
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11
Q

in severe hyperkalemia

A
  1. P & R waves gone,

2. S and T waves broaden in a “sine wave” pattern

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12
Q

abnormalities in the QT interval and the T wave due to drugs, abnormal electrolytes or other causes predispose to ____

A

arrhythmias usually though alterations in repokarization

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13
Q

There are ___ large boxes (heavy lines) between the R waves and ___ small boxes (thin lines) between the R waves.

A

6

30

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14
Q

T wave is inverted, a sign of ____

A

ischemia

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15
Q

ST elevation is a sign of ______.

A

transmural injury in an acute coronary syndrome, usually with a clot due to platelet aggregation obstructing a coronary artery.

Most commonly the injury is associated with an acute MI.
But, if the obstructed artery is quickly opened with angioplasty or a thrombolytic agent the ST elevation may partially or entirely reverse and much, or rarely all, injury avoided.

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16
Q

very small Q waves

A
  1. may not be pathological

2. But sizable Q wave is usually due to transmural necrosis.

17
Q

sizable means

A

at least one small box wide (≥0.04 seconds).

18
Q

When Q waves develop in leads which would normally be positive they give information on ___

A

localization of the infarct.

19
Q

Infarcts usually involve only the

A

left ventricle.

20
Q

Q waves in inferior leads (II,III, aVF) are due to_____.

A

inferior infarcts

21
Q

Q waves in leads V1-V4 are due to _____.

A

anterior wall infarcts

22
Q

Leads I, aVL and the anterolateral leads (V5,V6) are associated with ____

A

lateral wall infarcts.

23
Q

a transmural acute MI typically:

A

evolves over time

24
Q

acute MI early stage

A
  1. rarely seen
  2. lasts only a few minutes i
  3. giant, upright “hyperacute” T waves.
  4. T wave inverts (“ischemia”)
  5. ST segments then rise (“current of injury”).
25
Q

In acute MI,____ are usually the last ECG findings to develop

A

Q waves

26
Q

in acute MI, sometimes, the ST elevation ___

A

precedes or occurs simultaneously with the T inversion. .

27
Q

When the QT interval is altered susceptibilities to __ increase.

A

arrhythmias

This is especially likely with QT prolongation.

28
Q

commonly QT prolongation is acquired due to ____

A

electrolyte abnormalities or drugs.

29
Q

Hypokalemia: common causes

A
  1. overuse of diuretics
  2. vomiting
  3. diarrhea
  4. Alkalosis
  5. potassium losing nephropathies
  6. excess aldosterone