clinical tx of arrhythmia 4 Flashcards

1
Q

Ventricular Tachyarrhythmias: when is defibrillator needed?

A
  1. secondary prevention
  2. primary prevention
  3. medication and ablation are inferior to internal defibrillator, which is the ONLY tx shown to confer mortality benefit in these situations
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2
Q

Secondary prevention:

A

When the patient has had a sudden cardiac arrest due to VT or VF without a reversible cause (ischemia, drugs, electrolytes).

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3
Q

Primary prevention:

A

When the patient has not had a cardiac arrest but is at significant risk.
1. ischemic heart disease, low ejection fraction < 35% despite medical therapy.
2. ischemic heart disease, EF 35-40%, and inducible VT at EP study.
3. certain structural heart diseases with high risk of cardiac arrest:
hypertrophic cardiomyopathy, cardiac sarcoid, congenital heart disease, ARVC.

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4
Q

Currently, the best available treatments for sudden cardiac death is

A

bystander basic life support and early defibrillation with an external defibrillator.

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5
Q

other tx for SCD: acute therpy

A
  1. BLS
  2. defibrillator timing
  3. first responder AED
  4. Biphasic waveform
  5. Public AED
  6. pre-defibrillation CPR
  7. induced hypothermia
  8. antiarrhythmics
  9. vasopressin in asystole
  10. early ACLS
  11. thrombolysis in PEA
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6
Q

Tachyarrhythmia Take Home Points

A
  1. Tachyarrhythmias are divided into supraventricular arrhythmias and those coming from the ventricle.
  2. Any unstable tachyarrhythmia: SHOCK.
  3. Treat and reverse the underlying causes.
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7
Q

Only available treatments for sudden cardiac death:

A

basic life support and early defibrillation. YOU- as a potential bystander- can make a difference.

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8
Q

Most sudden deaths from ventricular arrhythmias occur in

A

general population before they can be risk stratified.

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9
Q

There are a variety of treatment options for SVTs and they should be

A

individualized to a patient’s characteristics, and the risks and benefits should be weighed.

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10
Q

Decisions regarding implantable defibrillators

A
  1. structural heart disease

2. what is risk of sudden death with the arrhythmia

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11
Q

SVT: treatment is

A

individualized according to the specific mechanism so

MAKE THE DIAGNOSIS (Adenosine to see p waves).

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12
Q

Chronic Management of AF

A
  1. rhythm control
    a. To achieve and maintain sinus rhythm
  2. rate control
    a. To achieve resting heart rate 60-80 /min
    b. To reduce undue increase of heart rate during exercise
  3. prevent thromboembolism
    a. Anticoagulation or anti-platelet treatment, based on risks of stroke
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13
Q

Rhythm-control strategy

A

Try rhythm-control first for patients with AF:

  1. who are symptomatic
  2. who are younger
  3. presenting for the first time with lone AF
  4. secondary to a treated/corrected precipitant
  5. with congestive heart failure
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14
Q

Rate Control should be used for

A
  1. Patients unsuitable for cardioversion, like
    a. Comorbidities
    b. decompensated heart failure
  2. All patients with rapid AF initially to relieve symptoms
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15
Q

control achieved in slowing:

A
  1. resting HR

2. HR during exercise

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16
Q

rhythm control: cardioversion is more likely to succeed if

A
  1. Recent onset AF
  2. No structural heart disease
  3. Successful treatment of precipitating causes: eg thyrotoxicosis, infection
  4. Young age
  5. Acute onset AF eg MI , Acute heart failure
17
Q

rhythm control: cardioversion is associated with

A

increased risk of thromboembolism during procedure

  1. Therefore warfarin, with INR 2-3, for 3 weeks prior to DC shock
  2. Or if onset of AF within 48 hours – IV heparin
  3. IF AF > 48 hours, transesophageal echocardiogram to rule out thrombus before cardioversion
  4. All patients maintained on anticoagulation for at least 4 weeks after cardioversion