digestive tract disorders part 3 Flashcards
Inflammation of the lining of the stomach
Gastritis
Cause of GAstritis
Mucosal barrier that normally protects the stomach from autodigestion breaks down
Hydrochloric acid, histamine, and pepsin cause tissue edema, increased capillary permeability, possible hemorrhage
Helicobacter pylori thought to be prime culprit
dietary indescretions, reflux of duodenal contents, use of aspirin, steroids, NSAIDS, alcohol, caffeine, cigarettes, food allergies, infection
S/S of Gastrtis
Nausea may or may not be present, vomiting, anorexia, a feeling of fullness, and pain in the stomach area,
pressure,
client may see blood in their emesis or note darkening of their stool color
medical diagnosis of gastritis
Gastroscopy
Laboratory studies to detect occult blood in the feces, low blood hemoglobin and hematocrit, and low serum gastrin levels ( may detect anemia) ; H. pylori can be confirmed by breath, urine, stool, or serum tests, or by gastric tissue biopsy
Medical treatment of gastritis
Oral fluids and foods withheld until the acute symptoms subside; IV fluids administered
Medications to reduce gastric acidity and relieve nausea
Analgesics for pain relief and antibiotics for H. pylori
Surgical intervention may be needed
some clients may wish to avoid spicy food/ high fats and caffeine
avoidance of alcohol and NSAIDS
Nursing assessement of Gastritis
Patient’s present illness
Pain, indigestion, nausea, and vomiting (observe color)
Determine the onset, duration, and location of pain
Note factors that trigger or relieve the symptoms
Diet, use of alcohol and tobacco, activity/rest patterns
Patient’s general appearance for signs of distress
Compare vital signs, height, weight to previous readings
Note the skin color and check turgor
Inspect abdomen for distention; palpate for tenderness
Auscultate abdomen for increased bowel sounds
nursing interventions of gastritis
Pain
Imbalanced Nutrition: Less Than Body Requirements
Deficient Fluid Volume
Ineffective Coping
Loss of tissue from lining of the digestive tract
Classified as gastric or duodenal
most occur in the duodenum by may develop at the lower end of the esophagus
Peptic Ulcer
Cause of peptic ulcer
Contributing factors: drugs, infection, stress
Most ulcers are caused by the microorganism H. pylori
when the normal balance between factors that promotes mucosal injury and factors that protect the mucosa is disrupted
S/S of peptic ulcer
Burning pain that occur 1-7h after meals , abdominal pain
Nausea, anorexia, weight loss , disrupted sleep,
single greatest risk is development of PUD is infection w/ gram negative bacterium H pylori
complications with peptic ulcer
Hemorrhage, perforation, or pyloric obstruction
medical diagnosis of peptic ulcer
Barium swallow examination, gastroscopy, and esophagogastroduodenoscopy
H. pylori can be detected by antibodies in the blood or stool so we need to eradicate the bacteria, reduce the acid levels, and by a breath test
medical treatment of peptic ulcer
Drug therapy
Diet therapy
Managing complications
Begins in the mucous membranes, invades the gastric wall, and spreads to the regional lymphatics, liver, pancreas, and colon
No specific signs or symptoms in the early stages
enlarged mass or ulcerating lesion that expands or penetrates several tissue layers
stomach cancer
The most common type of stomach cancer that rises from the granular cells in the inner layer of the stomach
adenocarcinoma
cause of stomach cancer
H. pylori infection, pernicious anemia, chronic atrophic gastritis, and achlorhydria, type A blood, and a family history
Cigarette smoking, alcohol abuse, and a diet high in starch, salt, pickled foods, salted meats, and nitrates