Ch 15 Learning Disabilities Flashcards
Definition of LD
unexpected under achievement in academics
arise in childhood
neurobiologically based, developmental
occurs on a continuum, not discrete, dichotomous
Types of LD
Reading disability
Math disability
Written expression disability
NLD that is common in which communities
Turner syndrome
22q11.2 deletion
congenital hydrocephalus
T or F? individuals may grow into their deficits
true
learning deficits may not fully manifest until a later age
% of people with LD
15-20
% of students with LD and need special education in school
13-14% (> 8 million)
% of students who have special education are classified as having SLD?
50%
Cutoff percentiles for LD diagnoses
25%ile (low achieving)
10%ile (extremely low achieving)
% of students with a primary LD have reading and language processing related LD
85%
male to female ratio with LD
1.5x more likely for males
heritability for reading LD
0.3-0.9
heritability for math LD
0.5-0.8
43%
LD is a developmental lag or functional deficit?
functional deficit that persists over time despite intervention efforts
do not spontaneously remit or normalize with time or age (does not grow out of it)
How does Anxiety amplify academic difficulties with LD?
interaction between limbic and frontal systems
anxiety floods frontal systems with excessive dopamine
pushes the system to impairing side of the inverted U shaped curve
Diagnosis of LD - 4 models
Aptitude achievement discrepancy model
Low achievement model
Intraindividual differences model
RTI model
Problems with RTI
no research support for adults
little consensus about how to define inadequate response to appropriate instruction
how to ensure fidelity to implement tiers of intervention
DSM impairment in Reading specifiers
Word reading accuracy
Reading rate or fluency
Reading comprehension
how many genes linked to dyslexia?
13 candidate genes
- 4 of them identified in speech and language d/o
children with genetic risk for development of dyslexia display differences in brain response and orientation to speech sounds as early as
a few days to 6 mos of age
postmortem studies of pts with dyslexia showed
- unexpected symmetry of the planum temporale, polymicrogyria of the L planum temporale, cortical dysplasia in L hemisphere
- L occipitotemporal cortex, middle part of left fusiform gyrus (rapid processing of words - visual word form area)
- alterations in structure (gray and white matter volume) in L hemisphere and R cerebellum
- difference in white matter microstructure in L
- underactivation in Wernicke’s area, angular gyrus, striate cortex
- over activation in inferior frontal gyrus
neural signature of dyslexia
overactivation in inferior frontal gyrus
underactivation in Wernicke’s area, angular gyrus, striate cortex
% of school aged children with Reading disability
10-15%