Bovine Diseases 3 Flashcards

1
Q

Prevention of Mastitis:

A
  • Good milking technique:
  • Make sure the milking machine is working properly. Improper vacuumed will damage teat canals and/or force milk back up the teat canal.
  • Milk the cows with mastitis last.
  • Keep the cows with mastitis separate from the healthy cows.
  • Cull cows with chronic mastitis.
  • Treat all cows at the end of lactation with a “dry cow” antibiotic treatment.
  • Maintain good records to catch mastitis early, when it is curable.
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2
Q

Good milking technique:

A

o Pre-dip teats before milking with antiseptic.
o Wipe off teats with a clean towel for each cow.
o Dip teats again after milking with an antiseptic dip that seals the teat ends

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3
Q

Causes of abortion in cattle:

A
  • Bacterial: Brucellosis, Salmonellosis
  • Viral: BVD, Bovine Herpes virus
  • Protozoal: Neospora
  • Nutritional: toxic plants, deficiencies, ect.
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4
Q

Diagnosis of abortion:

A

Abortion kits
• State Vet Pathology labs have kits for diagnosis.
• Fetal membranes and fetus should be submitted as soon as possible.
• Many causes of abortion are zoonotic.
• Many causes of abortion are contagious.

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5
Q

Milk Fever or Periparturient Hypocalcemia:

A
  • A problem with calcium regulation.
    * Fetal skeletal growth and milk production consumes a lot of calcium.
    * High dietary calcium before parturition can decrease the action of the parathyroid gland.
  • Occurs in late gestation or right after parturition.
  • More common in high producing dairy cows and Jersey & Guernsey cows.
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6
Q

Clinical Signs of Milk Fever:

A
  • Anorexia
  • Tremors
  • Low blood calcium
  • Flaccid paralysis
  • Bradycardia
  • Decreased rumen contractions
  • Death
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7
Q

Calcium gluconate intravenously

A

 Must be given slowly to prevent cardiac arrest.

 Often combined with phosphorus and magnesium.

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8
Q

Calcium gluconate subcutaneously.

A

 Used in combination with IV calcium.

 Contains dextrose. Use aseptic techniques.

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9
Q

Calcium gluconate gel orally:

A

 Comes in a huge tube.

 Dose is one tube per cow.

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10
Q

Calcium Supplementation

A

o Calcium gluconate intravenously:
o Calcium gluconate subcutaneously.
o Calcium gluconate gel orally:

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11
Q

Prevention of Milk Fever:

A
  • Feed quality grass hay prior to calving instead of alfalfa.
  • Avoid mineral supplements with high amounts of calcium.
  • Supplement the diet with highly digestible protein and energy feed.
  • Recent research has linked hypocalcemia with pregnacy toxemia (ketosis).
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12
Q

Pregnancy Toxemia or Ketosis

A

(Also called acetonemia)
• Occurs in fat or thin cattle during the last month of gestation or first 60 days of lactation.
• Occurs in muliple fetus pregnancies.
• Cattle are deprived of feed or unable to ingest adequate energy due to large uterus or heavy lactation.
• Often combined with hypocalcemia.

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13
Q

2 Types of Ketosis:

A

Wasting Ketosis

Nervous Ketosis

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14
Q

Wasting Ketosis:

A
  • Rapid weight loss.
  • Anorexia: refuses grain at first.
  • Decreased milk production.
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15
Q

Nervous Ketosis:

A
  • Bizzare behavior of rapid onset.
  • Hyperesthesia
  • Tremor
  • Ataxia
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16
Q

Clinical signs of ketosis

A
  • Anorexia
  • Rumen stasis
  • Scant feces
  • Weakness
  • Recumbancy
  • Ketonuria
  • Metabolic acidosis
  • Ketonia: can smell ketones on breath.
  • Fatty liver syndrome
17
Q
  • Delivery of the fetus.
  • Intravenous fluids with glucose & bicarbonate.
  • Insulin: helps get glucose into the cells.
  • Propylene glycol orally as a drench.
  • Corticosteroids: increase glucose metabolism.
  • Treatment is often unsuccessful.
A
  • Delivery of the fetus.
  • Intravenous fluids with glucose & bicarbonate.
  • Insulin: helps get glucose into the cells.
  • Propylene glycol orally as a drench.
  • Corticosteroids: increase glucose metabolism.
  • Treatment is often unsuccessful.
18
Q

Prevention of pregnancy ketosis:

A
  • Avoid overfeeding or underfeeding cattle in late gestation and the dry period.
  • Supplement the dry cow ration with a high quality concentrate 4 weeks prior to parturition.
  • Insure adequate energy consumption at parturition and during the first 2 months of lactation.
  • Gradually increase the amount of concentrate to avoid rumen acidosis.
19
Q

Failure of Passive Transfer(FPT):

A
  • Ruminants must have colostrum to receive maternal antibodies.
  • Gut closes to absorbtion of antibodies in 24 to 36 hours after birth.
  • Absorbtion of antibodies by the gut gradually decreases over the first 24 hours.
  • The first milking of colostrum has the most antibodies.
20
Q

Causes of FPT:

A

Inadequate consumption of colostrum

Poor quality colostrum

21
Q

Inadequate consumption of colostrum:

A

o Calf needs at least 2 liters in the first 12 hours (4 liters is best).
o Some calves are weak and can’t suckle.
o Some teats are hard to reach or suckle.
o Some cows won’t allow the calf to suckle.

22
Q

Poor quality colostrum

A

o Pre-milking the dam will decrease antibodies.
o Some cows don’t have quality colostrum.
o Feeding colostrum from later milkings instead of first milking colostrum.

23
Q

Prevention of FPT:

A
  • Milk the cow and feed the colostrum to the calf in the first few hours of life.
  • Feed at least 2 liters in the first few hours.
  • Bottle feeding is best, but if the calf won’t nurse, give the colostrum by stomach or esophageal tube.
  • In beef cattle, make sure the calf is suckling and the dam has milk.
24
Q

Diagnosis of FPT:

A
•	Serum IgG < 500 mg/dl
o	Test 8 to 12 hours after birth.
•	Several tests available.
•	Total Protien< 5.0 g/dl
o	Dehydration can give false elevations in TP.
o	Practical and easy test.
25
Q

Treatment of FPT:

A
  • Blood transfusion from the dam.
  • Plasma transfusions.
  • Oral Immunoglobulin does not work! The gut is closed to absorbtion.