Aphasia Classification Flashcards
list cortical aphasias
broca’s, wernicke’s, conduction, anomic
transcortical motor, transcoritcal sensory, global, mixed
list subcortical aphasias
anterior capsular-putaminal aphasia, posterior capsule-putaminal aphasia, global capsular-putaminal aphasia, thalamic aphasia
list nonfluent aphasia
broca’s, transcortical motor, global
most commonly affected areas in broca’s
left lateral frontal, pre-rolandic, suprasylvian region extending into adjacent subcortical preiventricular white matter
aphemia
possible symptom where person has an isolated loss of ability to articulate words with intact ability for spoken and written language
lesions limited to broca’s area cause what in brocas aphasia
mild deficits in prosodu and mild agraphia with occasional deficits in word finding pauses and mild dysrthria
broca’s aphasia deficits
langauge expression and relatively intact comp
most commonly affected areas in transcortical motor
areas connecting the supplementary motor cortex and brocas’ area; areas anterior and superior to broca’s area; supplementary motor cortex; white matter pathways underneat the supplementary motor area
most common affected bloody supple of transcortical motor
anterior cerebral artery or anterior branches of middle cerebral artery
nonfluent type of aphasia with intact repition skills
transcoritcal morot
most commonly affected areas in global aphasia
infarcts in both divisions of middle cerebral artery; left fronto-oarieto-temporal zone of lang
most sever form of aphasia with a geneeralixed effct on comminication skills
global aphasia
global aphasia deficits
deficits in both comp and expression
list fluent aphasias
wernicke’s, conduction, transcortical sensory, anomic
most commonly affected areas in wernickes aphasia
posterior 3rd of superior temporal gyrus
primary temproal lobe lesion deficits in wernickes
deficits in understanding individual isoleted spoken words but deficits in understanding words in context
more posterior temporal lobe lesion
wernickes aphasia
wernickes aphasia deficits
-vision
-more deficits in written lang and lang in context
-less deficits with isolated words
-lang comp
most commonly affected areas in conduction aphasia
supramarginal gyrus, undelrying white matter pathways, wernicke;s area, laeft insult, and aud cortex
caused either due to isolated lesions in alternative white matter bundle in inferior parietal lobe OR a combination of lesion affecting the left primary auditory cortex, insula, and underlying white matter
conduction aphasia
conduction can be classified into 2 types
afferent: damage in temporal lobes leading to impaired rep and memory deficits
efferent: parietal and insular lesions causing phonemic deficits
fluent type of aphasia with impaired repetition
conduction
most commonly affected areas in anomic aphasia
angular gyrus, second temporal gyrus
T/F: anomia is a very common symptom associated with different types of aphasia
TRUE
anomic aphasia is caused due to
lesions of angular gyrus or second temporal gyrus
anomic aphasia damage to left ingerior frontal region deficits
action naming
anomic aphasia damage to left temporal region deficits
nouns
fluent type of aphasia with anomia as the primary symptom
anomic aphasia
most commonly affected areas in transcortical sensory aphasia
posterior parieto=temporal, sparing wernickes area, bilateral lesions
transcortical sensory aphasia can often co-exist with
alzheimer’s disease
fluent type of aphasia with intact rep skills
transcortical sensory aphasia
Anterior Damage to Internal Capsule and Putamen symptoms:
-severe form of dysarthic articulation
-mild rep problems
-mod naming or word-finding probs
-some aud comp probs
-severe writing and mod reading probs
Posterior Capsular Putamen Damage symptoms:
-severe aud comp deficits
-fluent speech
-significant naming and word-finding probs
-mod reading and writing probs
anterior and posterior damage to thalamus sympomts:
-global aphasia
-nonfluent and extremely limited spontaneous speech
-severely impaired aud comp
-significant reading, writing, repetition, and naming probs
