ANATOMY - ANGLE Flashcards

1
Q

What are the borders of the anterior chamber?

A

ANTERIOR : posterior cornea
POSTERIOR: anterior iris

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2
Q

How deep is the anterior chamber?

A

3mm

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3
Q

Is the anterior chamber shallower in hypermetropes or myopes?

A

Shallower in hypermetropes - Axial length is smaller

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4
Q

How much aqueous humor is present in the anterior chamber?

A

0.25ml

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5
Q

What are the structures of the angle of the anterior chamber? (6)

A
  1. Iris Root
  2. Ciliary Body
  3. Scleral Spur
  4. Trabecular meshwork (posterior/pigmented)
  5. Trabecular meshwork (anterior/not-pigmented)
  6. Schwalbe’s line

I CAN SEE TILL SCHWALBE’S LINE

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6
Q

What is the scleral spur

A

At the junction of the limbus, internally there is a groove/indendation called the scleral sulcus.
A projection posterior from this area and is called the scleral spur

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7
Q

What is the ciliary body band?

A

Ciliary body is attached to scleral spur - a space exists here called the supraciliary space bewteen the ciliary body and sclera

Iris inserts into anterior side of ciliary body, and part of the ciliary body between iris and scleral spur is ciliary band, which is seen on gonioscopy

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8
Q

What is Schlemm’s Canal?

A

A Continuation of trabecular meshwork as a canal - it is oval in shape and is lined by endothelium

It is perforated by aqueous collector channels which branch into intrascleral and deep scleral plexi

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9
Q

What is Schwalbe’s line

A

Trabecular meshwork inserts into the periphery of the cornea at a ridge, known as Schwalbe’s line - marks the end of Descemet’s membrane of the cornea

Most anterior structure of gonioscopy

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10
Q

What is posterior embryotoxon?

A

Anterior displacement of Schwalbe’s line which is prominent on external exmination - associated with eisenfeld riger syndrome

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11
Q

What is Sampaoelsi’s line?

A

Pigmented Schwalbe’s line seen in PXF and pigment dispersion syndrome

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12
Q

What are the main age related changes seen in the trabecular meshwork? (5)

A

1) Increased pigmentation
2) Increased debris accumulation
3) Increased glycosaminoglycan deposition in extracellular space
4) Thickened basement membrane
5) Reduced endothelial cells

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