Trigger - HF 1 Flashcards

1
Q

RUQ pain, SOB, Orthopnea, and PND are symptoms of what

A

ACUTE heart failure

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2
Q

Edema, Anorexia, Abdominal distention and fatigue are common symptoms of what

A

CHRONIC heart failure

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3
Q

can be caused by thyrotoxicosis, severe anemia and sepsis

A

high output HF

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4
Q

what type of HF is EF of 46%

A

Type a - HFpEF borderline

Type I - HFrEF (EF </=40%)
Type II - HFpEF (EF >/= 50%)
Type a - HFpEF, borderline (EF 41-49%)
Type b - HFpEF, improved (EF >40%)

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5
Q

JVD, hepatic congestion, ascites, LE edema

A

R sided HF s/s

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6
Q

when do you see isolated R sided HF

A

rare, only when lung disorder is present!

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7
Q

DOE, PND, Orthopnea, fatigue, cyanosis

A

Left sided HF

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8
Q

These classes quantify functionality

A

NYHA

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9
Q

These classes CANNOT change and describe the evolution of HF

A

ACC/AHA

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10
Q

How would you classify someone with structural HF that has symptoms occasionally

AHA classification

A

Class C

A = At risk but no disease or symptoms.
B = Structural disease but no S/S.
C = Structural dsease with prior or current S/S
D = Refractory HF that requires specialized interventions (Usually class IV patients)

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11
Q

what causes RAAS system activation in HF

A

poor renal perfusion d/t poor CO.

leads to fluid retention and sodium dilution -> hyponatremia

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12
Q

increases ventricular contractility of the heart and HR.

also leads to vasoconstriction and enhanced venous tone, increasing preload

A

NE release. (d/t activation of SNS)

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13
Q

how does plasma concentration of NE correlate with HF severity

A

inversely correlated

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14
Q

kidney rxn to HF

A

Increased proximal tubular sodium reabsorption, which contributes to sodium retention in HF.

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15
Q

stimulated by increased beta - q adrenergic activity

A

RAAS system

also stimulated by decreased glomerular filtration

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16
Q

this peptide rises in early HF

A

ANP

17
Q

this is released in response to high ventricular filling pressures

A

BNP

18
Q

this Reduces SVR and central venous pressure and increases natriuresis to reduce afterload.

A

BNP

19
Q

effects of angiotensin II and catecholamines

A

promote myocyte loss and result in cardiac remodeling

20
Q

venous return and EDV (end-diastolic volume) is considered what

A

preload

21
Q

force generated at any given EDV

A

contractility

22
Q

aortic impedance, SVR, and wall stress

A

Afterload

23
Q

4 common PE findings in HF

A

Resting sinus tach
narrow PP (< 25 mm Hg)
Diaphoresis
Peripheral vasoconstriction

24
Q

What causes myocytes to develope more AT receptors and therefore result in apoptosis

A

RAAS system

25
Q

if a patient has edema with a 4 mm depression that resolves in 10 seconds what is her classification

A

2+ moderate pitting edema

  1. mild pitting (2mm depression)
  2. moderate pitting (4mm depression that disapears in 10-15s)
  3. moderate/severe - (6mm depression that last >1 min)
  4. severe - (8mm depression lasting >2 min)
26
Q

what does pulsus alternans suggest

A

Pathognomonic finding in severe LV failrue

27
Q

what does a laterally discplaced apical impulse suggest

A

LV enlargement

28
Q

what does parasternal lift of RV on precordial palpation suggest

A

pulmonary HTN

29
Q

is S4 associated with systolic or diastolic HF

A

diastolic

S3 - systolic
S4 - diastolic

30
Q

4 HF findings in CXR

A

Pulmonary vascular congestion
KERLEY B lines
cardiomegaly
Pleural effusions

31
Q

released in response to atrial volume expansion

A

ANP

32
Q

what is the BEST test for HF evaluation

A

BNP and NT-ProBNP

33
Q

when do we do stress testing in HFrEF

A

R/o CAD

34
Q

donnneeee

A