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NAVLE ok... > small animal cardio > Flashcards

small animal cardio Flashcards

1
Q

what is endocardiosis?

explain the pathophys

A

screwed up mitral valve

  1. with a bad mitral valve, extra fluid flows backward into the LA, increasing your pre-load.
  2. this decreases your CO and decreases the amount of blood going to the rest of the body.
  3. body interprets this as hypovolemia and turns on RAAS
  4. RAAS increases water retention an increases the flow to lungs and heart
  5. this overloads the LA
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2
Q

which valves are pre-disposed to endocarditis?

A

the L-heart: aortic and mitral valves

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3
Q

volume overload leads to what kind of hypertrophy

what kind of issues do you see this with?

A

VOLUME = ECCentric hypertrophy
seen with VALVULAR issues like mitral regurge
walls get stretched

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4
Q

pressure overload leads to what kind of hypertrophy?

what kind of issues do you see this with?

A

PRESSURE = CONcentric hypertrophy
seen with aortic and pulmonic STENOSIS
walls get thick

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5
Q

what medications treat mitral valve disease?

A

furosemide (lasix) - diuretic
- decreases preload, decreases work on the heart

enalapril (ace inhibitor)
- decreases aferload by decreasing blood pressure

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6
Q

difference between dogs and cats when it comes to showing signs of congestive heart failure?

A

dogs get pulmonary edema - fluid actually in lung tissue - in alveolar beds
cats get pleural effusion (fluid in between the lung itself and the pleura - the membrane covering the lungs)

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7
Q

clinical signs of congestive heart failure

A
  • pulmonary edema (dog) or pleural effusion (cat)
  • tachypnea or hypernea
  • syncope
  • coughing
  • rads: mainstem bronchi compression (large pulmonary vessels); alveolar pattern (air bronchograms)
  • bounding pulse
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8
Q

what are two diseases with bounding pulses?

A
  1. PDA

2. bacterial endocarditis

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9
Q

what causes increased venous pressure (which leads to jugular pulses)?

A

R-sided heart failure caused by tricuspid valve disease

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10
Q

emergency therapy for congestive heart failure

A
  • furosemide (diuretic to decrease preload)
  • anxyiolytics (opioids, diazepam, ace)
  • arteriodilators (decrease afterload): enalapril (ace inhibitor); amlodipine (Ca channel blocker acts on vasculature)
  • pimobendan (increases contractility)
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11
Q

chronic therapy for congestive heart failure

A
  • diuretics = furosemide (for pulmonary edema)
  • Ace-inhibiotr = enalapril (decrease afterload)
  • vasodilator & Ca channel blocker = amlodipine (decrease BP)
  • positive iontrope = pimobendan (increases contractility and dilates vessels)
  • beta blockers = for arrhythmias
  • Ca-channel blockers = diltiazem (slows HR)
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12
Q

bacterial endocarditis

A

usually mitral or aortic valves

  • often staph/strep/E.coli or bartonella
  • signalment: young, often large breed dog with sudden murmur after being sick
  • CS: fever, polyarthritis, hyperkinetic or bounding pulses
  • tx: abx IV for at least a week: ampicillin, baytril, azitrhomycin if its bartonella
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13
Q

what large breed dog is susceptible to DCM?

A

dobies! and danes

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14
Q

when cocker spaniels get DCM, it is often caused by a deficiency in what?

A

L-cartinine

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15
Q

explain the pathophys of DCM

A
  • heart muscle isn’t working right (poor/loss of contractility)
  • decreased CO
  • causes sympathetic activation (RAAS, hormonal, etc)
  • atrial fibrillation and VPCs are common sequelae bc conduction fibers are being pulled apart
  • end up with both forward and backward failure
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16
Q

CS of DCM

A
  • first sign can be sudden death
  • TALL “R” WAVES!!
  • ventricular arrhythmias
  • weakness, tachypnea, exercise intolerance, ascites, syncope
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17
Q

how do you treat DCM?

A
  • positive iontrope = pimobendan (increase contractility) AND digoxin (increases contractility and slows AV node conduction)
  • Ace-inhibitor = Enalapril (decrease afterload to increase circulation)
    +/-
  • Ca channel blocker = diltiazem (slow HR; centrally acting)
  • beta blocker = sotalol (good for arrhythmias)
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18
Q

heartworm disease complications (other stuff it causes)

A
  • leads to R-sided CHF
  • cor pulmonale (heart disease caused by primary lung disease. pulmonary hypertension increases cardiac afterload and leads to R-sided hypertrophy)
  • chronic inflammatory disease –> glomerulonephritis
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19
Q

what is the infective stage of heartworm?

A

L3

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20
Q

heartworm tests for dogs vs cats

A

the test looks for FEMALE antigen
cats can have single sex infections; so cats should have ANTIGEN tests
dogs should have Ab tests

cats = antigen
dogs = antibody
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21
Q

treatment for HWD

A

doxycycline for wolbachia
melarsomine/immiticide

2 month protocol: doxy for 1 month, injection of adulticide; wait one month; then have the 2nd injection; repeat HW test 6 months post treatment

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22
Q

HW prevention

A

ivermectin

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23
Q

what diseases can cause systemic arterial hypertension? (8)

A
  1. CKD
  2. cushings
  3. hyperthyroidism
  4. pheochromocytoma
  5. diabetes
  6. liver dz
  7. hyperaldosteronism
  8. intracranial lesions
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24
Q

what are some clinical signs or evidence of systemic hypertension (4 main things)

A
  1. progression of chronic kidney disease
  2. eyes: retinopathy, acute blindness
  3. brain: encephalopathy or stroke
  4. heart and vessels: left ventricular hypertrophy
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25
Q

how do you treat systemic hypertension

A
  1. Ca channel blockers: amlodipine and diltiazem
  2. Ace inhibitors - enalapril
  3. beta blockers - atenolol, propranolol
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26
Q

what are some things that cause pulmonary hypertension (4 things)

A
  1. increased pulmonary blood flow
  2. increased blood viscosity (polycythemia)
  3. increased pulmonary vascular resistance
  4. luminal narrowing
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27
Q

how do you treat pulmonary hypertension?

A

sildenafil (viagra!)

tadalifil (cialis)

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28
Q

what breed of dog is susceptible to mitral valve dysplasia?

A

bull terriers

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29
Q

what breed is susceptible to sub-aortic stenosis?

A

newfoundlands and goldens

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30
Q

what does the murmur of a sub-aortic stenosis sound like?

A

systolic crescendo/decresendo murmur that is loudest over left heartbase

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31
Q

explain the pathophys of sub-aortic stenosis

A
  1. there is an outflow tract obstruction
  2. causes a pressure overload in the Left ventricle
  3. leads to a CONcentric hypertrophy
  4. poor ventricular filling and coronary perfusion leads to a lack of oxygen for the heart
  5. mycardiocytes die
  6. leading to arrhythmias
  7. causes a secondary mitral regurgitation
  8. eventually leads to low output failure which can result in death
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32
Q

clinical signs & diagnosis of sub-aortic stenosis

A
  1. rads show big LV

2. echo shoes CONcentric hypertrophy (from pressure overload)

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33
Q

treatment of sub-aortic stenosis

A

beta-blockers! causes relaxation and decreases afterload and arrhythmias

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34
Q

prognosis for sub-aortic stenosis

hint: what does this disease predispose dogs to?

A

px not good; 50% die suddenly

predisposes to infective endocarditis!!

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35
Q

what breed is predisposed to pulmonic stenosis?

A

bulldogs! and boxers

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36
Q

what is the pathophys of pulmonic stenosis?

A

it’s basically the same as sub-aortic stenosis but on the right side of the heart.

there is some obstruction that causes a pressure overload (CONCENTRIC hypertrophy) which leads to a Right ventricular hypertrophy with secondary dilation and right atrial enlargement.

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37
Q

given the pathophys of pulmonic stenosis, what is an unusual clinical sign of this disease?

A

the murmur is best heard over the LEFT heartbase (systolic crescendo/decresendo) despite the disease being a R-sided heart disease

(sounds very similar to sub-aortic stenosis)

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38
Q

how do you treat pulmonic stenosis?

A

balloon vulvuoplasty most common!

- causes pulmonic insufficiency, but that is better to have than stenosis

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39
Q

explain the pathophys of a PDA (patent ductus arteriosus)?

A

it is a shunt connecting the aorta (L-side) and pulmonary artery (R-side).

  1. blood is continuously pumped thru this patency in the aorta (L-side) to the pulmonary artery (R-side)
  2. this leads to overload of L atria, increases pressure on the right
  3. this will eventually reverse the shunt
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40
Q

what does PDA cause?

A

volume overload of pulmonary vasculature

overload in LA and LV

41
Q

what breeds are predisposed to PDA?

A

female maltese!!!

42
Q

what does a PDA murmur sound like?

A

continuous left basilar murmur

43
Q

how do you diagnose a PDA?

A

enlarged LA and LV

ductus bump on rads

44
Q

how do you treat a PDA?

A
  1. transcatheter (coils, amplatz ductual occluder)

2. surgical ligation (cylindrical vs funnel shaped)

45
Q

what is an odd sign of a reversed PDA?

A

after it reverses, if the shunt is located after the branches that go to the head, you get oxygenated blood going to the head and mixed blood going to the caudal party - leads to a blue butt!

46
Q

who is predisposed to VSDs?

A

CATS!

47
Q

what does the murmur of a VSD sound like?

A

systolic plateau murmur that is loudest on the RIGHT side

48
Q

clinical signs of VSD?

A

holosystolic murmur heard best on the right side

creates functional pulmonic stenosis bc you are pushing more blood thru the valve.

49
Q

how do you treat a VSD?

A

Ca Channel blocker - amlodipine: decreases afterload and decreases pressure in aorta so that there is an increase in forward flow

50
Q

what is a type of atrial septic defect we see and what species does it occur more often in?

A

fossa ovalis

dogs

51
Q

what are some signs of a severe atrial septal defect?

A

RA or RV enlargement due to volume overload (an ECCENTRIC hypertrophy)

52
Q

what breed do we see mitral valve dysplasia in?

A

bull terriers - males

53
Q

what is the pathophys of mitral valve dysplasia?

A

congenital disease in which you have dysplastic chordae tendoninae so the valves dont grow correctly.

54
Q

what are clinical signs and tx of mitral valve dysplasia?

A

similar to degenerative valve disease but we are seeing htem in YOUNG dogs.
- regurgitation and stenosis

tx: valve replacement

55
Q

what breed is predisposed to tricuspid dysplasia?

A

male golden retrievers - super young little babies

56
Q

pathophys of tricuspid dysplasia

A

the tricuspid is dysplastic - it comes from the right atrium and leads to R-heart enlargement

57
Q

px of tricuspid dysplasia

A

guarded to poor

58
Q

what breed is predisposed to tetralogy of fallot?

A

keeshonds!!

59
Q

what are the magical 4 things that make up tetralogy of fallot?

A
  1. VSD
  2. pulmonary stenosis
  3. overriding aorta (dextropositioned)*
  4. R-ventricular hypertrophy
60
Q

what does the murmur of tetralogy of fallot sound like?

what are the CS?

A

holo-systolic RIGHT sternal murmur (due to VSD) OR a systolic ejection murmur at LEFT base (due to pulmonic stenosis)

CS: chronic hypoxia leads to increased erythropoietin and utlimately polycythemia!! syncope, cyanosis, exertional weakness

61
Q

how do you diagnose tetralogy of fallot`

A

rads: underperfused lungs - reduced pulmonary vasculature (hardly see any vessels)

62
Q

how do you treat tetralogy of fallot?

A

periodic phlebotomy

medical management of CHF (furosemide, enalapril, amlodipine, pimobendan, beta blockers, diltiazem)

hydroxyurea (goes to bone marrow and decreases production of RBCs)

63
Q

what is the surgical option of treatment for tetralogoy of fallot?

what is the ultimate px of tetralogy of fallot?

A

subclavian artery transposition to create a PDA - gets more oxygenated blood thru the system

middle aged if mild

64
Q

what ECG abnormalities will you see with a hyperK?

A
  • no P waves!
  • TAAAALL T waves!
  • QRS complex wide and bizarre
  • low HR
65
Q

which breed is predisposed to sick sinus syndrome (screwed up SA node)?

A

schnauzers

66
Q

what is the rate at which the SA, AV and myocardial muscle cells operating at?

A

SA node is fastest: 80 - 120 bpm
next is AV node: 60 - 80 bpm
ventricular myocardial cells: 40 - 60bpm

67
Q

boxer is predisposed to what cardiomyopahty

A

arrhythmogenic Right Ventricular complexes (ARVC)

68
Q

effect of beta 2 adrenergic receptors on the heart?

A

work on sympathetic system - relaxes it
slooooow down the HR
dilate vessels

in contrast to Beta-1 receptors which speed things up

69
Q

effect of alpha 2 adrenergic receptors on the heart?

A

increase diastole

  • INCREASE HR
  • increase contractility
70
Q

atrial premature contractions (APCs)

A

a P wave that comes early
its due to another part of hte atria depolarizing instead of the SA node

will be a fast, irregular rhythm

tx: sloooow down HR: positive ionotrope - digoxin

71
Q

atrial fibrillation

A

ecoptic pacemaker: signal is coming from different pacemakers

  • irregularly irregular rhythm
  • more P waves than QRS complexes
  • treat when BP drops below 80 bpm: want to SLOW HR: beta blockers (esmolol), Ca channel blockers (diltiazem), digoxin
  • causes: anything that causes LA enlargement - DCM, mitral endocardiosis, HCM
72
Q

what do positive ionotropes do?

A
  • decrease atrial arrhythmias but increase ventricular arrhythmias
  • slows conduction thru AV node to slow HR
73
Q

ventricular premature complexes (VPCs)

A
  • wide and bizarre QRS
  • when they look different its bc they are coming from different places
  • treat in hospital with lidocaine (Na channel blocker)
74
Q

ventricular tachycardia

A

arrhythmia coming from ventricle (so SA and AV node are both not working). defined as >3 consecutive VPCs

  • premature complex: decreasing R-R interval
  • REGULAR rhythm
  • watch for R-on-T!! risk of ventricular fibrillation!
  • tx: treat ventricular arrhythmia by giving lidocaine up to 3 times then try procainamide. or you can try just slowing down HR a bit by giving esmolol or diltiazam.
  • if dog is unconscious defibrillate (defibrillation works in a-fib and v-tach)
75
Q

what are class1a drugs?
drug type
what they treat
examples of drugs

A

fast Na channel blockers

treat: V-tach, supraventricular tachycardias
ex: procainamide

76
Q

what are class 1B drugs?
drug type
what they treat
examples

A

Na channel blockers

treat: VPCs, supraventricular tachycardias, v-tach
ex: lidocaine

77
Q

what are class 2 drugs?
drug type
what they treat
examples

A

beta blockers - increase conduction time

treat: supraventricular tachycardias and v-tach
ex: atenolol, esmolol, propranolol

78
Q

what are class 3 drugs?
drug type
what they treat
ex

A

prolong action potential and refractory period

treat: used to prevent v-tach and sudden death
ex: sotalol (dirty beta blocker), amodarone

79
Q

class 4 drugs
drug type
what they treat
ex

A

Ca channel blockers, slow (L-type) channel inhibitors,

treat: supraventricular tachycardia
ex: diltiazem

80
Q

what drug options are there fore tachycardia?

A

digoxin - increases contractility, decreases speed of normal conduction

diltiazem - central acting Ca channel blocker, slows conduction thru AV node

81
Q

what drugs ares used in emergency situations for sinus tachycardia?

A

fast Na channel blocker: procainamide

Beta blocker: amiodarone

82
Q

in what 3 instances do you treat V-tach

A
  • when you have a run of complexes ( > 3)
  • when they are different shapes (coming from different locations)
  • when you have R-on-T phenomenon
83
Q

what are some mechanical (non-medical) things you can do to treat V-tach?

A

vagal maneuver - push on both eyeballs, stimulates vagal tone and slows or blocks re-entrant supraventricular tachycardia temporarily

precoridal thumps - punch the area where you feel precordial thrill, can disrupt the reentrant circuit

84
Q

ARVC (arrhythmogenic Right Ventricular Cardiomyopathy)

A
  • aka boxer cardiomyopathy
  • causes fibrotic myocarditis
  • will see large number of VPCs
  • risk of going into ventricular tachycardia
  • dx: holter monitor
  • tx: beta blockers to decrease HR (sotalol)
85
Q

slow irregular bradyarrhythmias are a result of what?

A

increased vagal tone due to GI disease, resp disease or vagosympathetic trunk masses

86
Q

how do you diagnose and treat a sinus bradycardia?

A

atropine challenge
if responsive, the HR will increase by 150%

tx with a placemaker

87
Q

sinus arrest or atrial standstill

A

sinus node stops firing so the AV node takes over

  • no P for every QRS
88
Q

sick sinus syndrome

A

aka “tachycardia-bradycardia syndrome”: its a condition where the SA node is not working properly

  • will see episodic weakness or syncope
  • tx with pacemaker + beta blocker
89
Q

who is the poster child of sick sinus syndrome?

A

female schnauzers

90
Q

1st degree AV block

A

long P-R interval with no dropped beats

no real tx necessary

91
Q

2nd degree AV block: Mobitz type 1

A

aka Wenckenback

- P-R interval increases until you get a P without a QRS

92
Q

2nd degree AV block: Mobitz type 2

A
  • P-R interval remains the same

- tx with a pacemaker

93
Q

3rd degree AV block

A

no relationship between Ps and QRS complexes

  • the P-R intervals will constantly vary with no pattern
  • often has ventricular escape beats or AV nodal beats
  • tx w/ pacemaker
94
Q

what breed of cat is predisposed to HCM?

A

maine coons
rag dolls
persians

95
Q

HCM pathophys

A

progressive disease - a type of idiopathic Left Ventricular hypertrophy

  • progressively higher LV filling pressure
  • leads to high LA and pulmonary vein pressure
  • leads to progressive LA dilation
  • which causes pulmonary congestion and edema
96
Q

what are clincal signs of HCM?

A
  • thrombus formation (think saddle thrombus)
  • outflow obstruction
  • atrial fibrillation
  • L-sided heart failure
  • thickened walls
97
Q

how do you treat HCM?

A

want to slow the heart down!

  • beta blockers: atenolol
  • Ca channel blockers: diltiazem (central), amlodipine (peripheral vasodilator)
  • Ace inhibitor: benazepril/enalapril (decreases afterload)
  • super low doses of aspirin to decrease platelet function to lessen the formation of thrombus
98
Q

restrictive cardiomyopathy

A

left ventricular endomyocardial fibrosis
systolic murmur
gallop rhythm
dx: echocardiograph; shiny lines in heart

99
Q

peritoneo-pericardial diaphragmatic hernia

A

congenital disease

cats: DLH, persians, himalayans
dogs: weimeraners

  • presents like heart disease
  • dx: rads
  • tx: only needed if animal shows clinical signs via sx

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