Schizophrenia Flashcards

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1
Q

Positive symptoms of schizophrenia include ?

A
  • Hallucinations - unusual experiences that have no basis in reality,(auditory + visual)
  • Delusions - irrational/false beliefs that have no basis in reality
    (Delusions of persecution and delusions of control)
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2
Q

Negative symptoms of schizophrenia?

A
  • Avolition- severe lack of motivation to carry out everyday tasks
  • Speech poverty- a reduction in the amount and quality of speech
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3
Q

define Diagnosis ?

A
  • identification of the nature of an illness or other problem by examination of the symptoms
    EG: someone reporting hearing voices
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4
Q

define Classification?

A
  • The action or process of classifying something: the classification or diseases according to symptoms
    EG: hallucinations
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5
Q

Reliability and validity in diagnosis and classification of schizophrenia, including reference to co-morbidity, culture and gender bias and symptom overlap. AO1

A
  • Reliability refers to consistency, whether we can gain consistent results when classifying and diagnosing Sz.
  • extent which diff classification systems agree upon how Sz should be classified and the extent to which 2+ health pros would agree on same diagnosis, regardless of time period or culture, measured by inter-rater reliability.
  • Validity refers to accuracy, extent which we are measuring what we intend to measure
    EG: are the classification systems accurately outlining the signs and symptoms of Sz + are health pros’ accurately diagnosing Sz?
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6
Q

AO3 for reliability and validity

A
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7
Q

Expressed Emotion AO1

A
  • High levels of expressed emotion can cause stress in patient, this is a primary explanation for relapse in patients with Sz .
    Expressed emotion is characterised by:
  • Verbal criticism and occasional violence towards the patient
  • Hostility towards the patient, including anger and rejection
  • Emotional over-involvement in their life
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8
Q

Neural correlates AO1

A
  • abnormalities within specif brain areas maybe associated with development of Sz.
  • enlarged ventricles , meta-A, raz&raz
  • over half ppl tested w Sz had increase ventricle size compared to control group
  • (EnlV) associated w damage to central brain areas+pre-frontal cortex associated w negative symptoms
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9
Q

AO3 for genetic theory
(Study)

A

RTS from Tierney

  • studied 155 adopted kids w bio mothers w Sz
  • Found: concordance rate of 10% compared to mum w no Sz 1%
  • significant support for role of genetics as expl of Sz as role of SLT not factor as kids adopted
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10
Q

genetic theory into schizophrenia AO1

A

sz is hereditary + passed on from one gen to the next through genes.
* Therefore, a person born 2 a genetic predispo (likelihood) to sz. several maladaptive ‘candidate’ genes such as PCM1, are involved (polygenic) which incl a person vulnerability to developing sz.
* Studies show 108 separate genetic variations are associated in the risk of developing sz. Gottesman studied 40 twins found concord rate for mz twins 48% only 17% dz twins. closer genetic link to somebody with sz.the more chance of developing sz

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11
Q

Dopamine Hypothesis (another neural correlate) – AO1

A

Hyperdopaminergia in subcortex:

  • High levels/activity of dopamine in
    central areas of brain EG; Broca’s area maybe associated with aud hallucinations.

Hypodopaminergia in cortex:

  • Low levels of dopamine in prefrontal
    cortex (thinking and decision making) been associated with negative symptoms of Szsuch as avolition
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12
Q

Overall - Biological explanation of schizophrenia - AO3

A

Scientific methods

  • theory based on emp+obj techniques such as gene mappin+brain scans, used to identify parts of brain linked to Sz, EnlV
  • Thus increases overall int val, raise status

Criticised for biological determinism

  • indiv controlled by internal factors EG: high lvls of dopamine in the subcortex inevitably causes auditory hallucinations
  • therefore negl role of FW and choice, leaving indiv, feel no control over Sz beh

Practical applications

  • principles of theory, Sz caused imbalance of dopamine -> treatment drug therapy
  • typical,atypical antipsychotics, balance dopamine lvls in brain red symptoms of Sz like hallucinations and delusions
  • Thus bio expl import part of appl psych
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13
Q

Family dysfunction AO1

A
  • idea that individual develops Sz bc been raised in dysfunctional family environ
  • family is dysfunctional in way they
    communicate w each other as if have high levels of interpersonal conflict.
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14
Q

Family dysfunction - AO3

A

RTS double bind comm (DBC) by Berger

  • found schizophrenics remember more instances of DBC from mum, during childhood, than non-Sz.
  • provides clear support for confused comm in schizophrenics’ childhood (DBC) + family dysfunction as expl of Sz.

However

  • DBC theory criticised as info on childhood exper based on retrospective data (patient thinking back to childhood).
  • could mean there are inaccuracies in recall as long period of time passed.
  • reduces the int val of research into the DBC as explanation of schizophrenia.

Practical applications

  • principles of expl, Sz caused by faulty fam commun -> treatment family therapy
  • effective treating Sz by therapist meet w patient+fam try alter rship patterns
  • helps red stress, prevent relapse of Sz
  • therefore FD , imp part appl psych
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15
Q

Schizophrenogenic mother AO1

A
  • idea: Sz caused by patients early experience of schizophrenogenic mother
  • schizophrenogenic mother is cold, controlling, rejecting, emotionally
    unresponsive + builds family climate characterised by tension and secrecy.
  • leads->distrust develops to paranoid delusions (+ symptom) in Sz.
  • father in such families is often passive.
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16
Q

Double Bind communication AO1

A

Bateson et al (1956)

  • argues that Sz due to faulty communication patterns that exist in families = double blind communication
  • occurs when parent communicates verbal message which is not matched w their non-verbal message, so the child receives mixed messages.
  • EG: a father may be verbally loving but emotionally rejecting,
  • conflicting, confusing forms of communication cause Sz.
  • child feel cannot do right thing become increasingly anxious = withdrawing + avoiding social contact – signs of avolition ( - symptoms) the mixed messages result in disorganised thinking and paranoid delusions
17
Q

AO3 for cognitive explanations for Schizophrenia

A

RTS

  • stirling et al
  • compared 30 patients w Sz w 18 non-patient on a range of cog tasks (stroop test)
  • Sz patients took x2 longer to complete task as control, couldnt supress auto resp of sayin word than colour
  • supporting friths theory of cent cont dysfunction

soft determinism

  • caused by internal factors (faulty perceptions), suggest indivs have choice over beh (unlike bio approach suggest we have no choice)
  • could be beneficial as indivs may feel in control and feel it possible alter these thought proc
  • making cog expl of Sz more positive expl

Practical applications

  • principles, Sz caused by delusional thoughts led to treatment of CBT
  • affective in treating Sz, patients helped to identify, challenge del thoughts, help reduce + symptoms
  • therefore cog expl of Sz import part appl psych
18
Q

Decribe typical antipsychotics (chlorpromazine)

A
  • are dopamine antagonists, red lvls of dopamine activ in brain
  • inhibit D2 receptor on post synaptic neurone in brain red dopamine action
  • red positive symptoms of Sz (hallucinations)
19
Q

Describe atypical antipsychotics (clozapine)

A
  • act upon dopamine & serotonin
  • inhibit D2 rec on post syn neurone, red positive symptoms
  • act as agonists upon sero rec sites (2A&2C) increase sero lvls
  • red neg symptoms of Sz such as lack of emotions help improve mood, red depression and anxiety.