S&R: CLAP Flashcards

1
Q

What is articulation?

A

The act of producing the speech sound itself

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2
Q

What is phonology?

A

Concerned with the linguistic difference between the difference of sounds

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3
Q

What are the two main subtypes of articulation and phonological disorder?

A
  • Organically based disorder – speech sound errors have have an obvious etiology → cleft, hearing impairment, macroglossia, motor speech, etc.,
  • No obvious cause– functional articulation disorder, developmental disorder, speech sound disorder, speech delay
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4
Q

What is articulation disorder? What is intact? How will you know if it is an artic disorder?

A
  • Issues with the production level only
  • Phonological discrimination is intact.
  • SODA errors (substitution, omission, distortion, addition)
  • Aberrant speech sound form
  • Errors are consistent → word position (even if it’s initial, medial, final position–there will still be an error)
    - Regardless of the word position, the error will be there consistently, but the sound may be inconsistent especially if the sound is emerging.
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5
Q

What are the signals of articulation disorder?

A

Preservation of phonemic contrasts in errors (e.g., palatalized [s] for [θ]).

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6
Q

What is phonological disorder? Production is…? Issues with…?

A
  • Production capability could be intact → The child will be able to produce the sound in isolation, however, if using it in a phrase, the sound disappears.
  • Issue with phonological awareness –differentiating the sound with different sounds
    Discerning speech sounds for the purposes of meaning differentiation
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7
Q

What are the signals for a phonological disorder?

A
  • Widespread error patterns
  • Errors could be inconsistent
  • Severe limitations in produced sounds → phonemic inventory of the child is limited due to widespread error patterns
  • Syllable structure limitations
    Note: There is common co-occurrence with morphosyntactic issues
  • Intact audition and speech (there is no motor issue, planning issue, or audio issues)
  • Unusual errors not seen in typical development
  • Co occurrence of error patterns
  • Presence of idiosyncratic phonological processes
  • Phonological processes past of age of dissolution
  • Last hallmark for phonological disorders: vowel errors

Hallmarks
- inconsistent error
- idiosyncratic phonology processes
- Vowel errors
- limited phonemic inventory
- no motor & planning issue

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8
Q

What are idiosyncratic processes?

A

Not seen in typically developing children , this is unique for children with phonological disorders–Idiosyncratic processes are atypical or unusual speech sound patterns that are not commonly seen in typical speech development

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9
Q

Phonological delay

A

Phonological processes that continue past the age of dissolution

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10
Q

Consistent atypical phonological disorder

A

1 or more idiosyncratic processes consistently

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11
Q

Inconsistent atypical phonological disorder

A
  • 1 or more idiosyncratic processes inconsistently
  • Variation in output similar to CAS
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12
Q

What are velopharyngeal closures?

A

Movement of the soft palate and the pharyngeal walls, the soft palate will fully approximate with the pharyngeal wall

Variations of approximation of soft palate and pharyngeal walls to separate the oral and nasal cavity

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13
Q

VELOPHARYNGEAL CLOSURE – CORONAL

A
  • Most common
  • Posterior movement of the soft palate
  • Closing against the posterior pharyngeal wall

Soft palate ang umaatras sa pharyngeal wall

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14
Q

VELOPHARYNGEAL – CIRCULAR

A
  • Lateral pharyngeal wall and posterior pharyngeal wall and soft palate forms a “circular pattern”
  • Sphincter like
  • 23%; 5% circular with Passavant’s ridge
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15
Q

VELOPHARYNGEAL – SAGITTAL

A
  • Lateral posterior wall meets in the midline
  • Least common – 4%
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16
Q
  • What is a cleft?
  • Where does it occur?
  • Which structure often has cleft?
  • Are there missing structures?
A
  • An abnormal opening or a fissure in a structure that is usually closed–occurs in the embryological fusion lines
  • Usually occurs in the lips or either palates
  • Usually, structures are all present but not fused together normally
  • Congenital
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17
Q

Cleft lip and palate

  • The fusion starts where?
  • how does the cleft occur?
  • what is a complete cleft?
  • What is the pathway of a cleft lip?
A
  • Fusion starts at the incisive foramen
  • Any disruption will cause a cleft from that point to the periphery
    Complete cleft: from the periphery all the way to the foramen
    Cleft lip: Incisive foramen → alveolar ridge → nasal floor → Lips
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18
Q

What are the two main causes of cleft?

A
  • Endogenous → chromosomal, genetic, increased parental age
  • Exogenous → teratogens, mechanical factors of utero
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19
Q

What is the dividing point to identify the primary and secondary palate?

A

Incisive foramen

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20
Q

Clefts can be of the ___________, ____________, _____________.

A

Clefts can be of the primary palate, secondary palate, or both

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21
Q

What is the primary palate?
What are these structures?
When should it fuse?

A
  • Structures anterior to the incisive foramen
  • Fuses at around 7 weeks of gestation
  • Alveolus + lip (&nose)
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22
Q

What is the secondary palate?
What are these structures?
When do they usually fuse?

A
  • Structures posterior to the incisive foramen
  • Fuses around 9 weeks of gestation
  • Hard palate (except the alveolus) + velum
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23
Q

Unilateral cleft palate?
Where is it more common?
What is the appearance of the lips?

A
  • On either side
  • Most common on the left side
  • The lips is short in the affected side
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24
Q

What is the occurrence of Primary palate clefts?

A

May occur in isolation but is more often found to be associated with cleft on the palate

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25
Q

Complete primary palate clefts

A

Extends through the lip, nostril and alveolus to the incisive foramen

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26
Q

Incomplete primary palate cleft

A
  • If the cleft does not extend to the incisive foramen
  • Can be as minor as subcutaneous notch
27
Q

What are the presentations of primary palate clefts?

A
  • Unilateral
  • Bilateral
28
Q

Bilateral cleft palate? It is indicated by the presence of which structure?

A
  • Both sides are affected
  • Complete separation of the tissue
  • Presence of a prolabium
29
Q

What is prolabium?

A

Refers to the central part of the upper lip, specifically the portion that lies between the two philtral ridges

30
Q

What is the form fruste?

A
  • Intact skin
  • Muscle affected
31
Q

What is simonarts band?

A

Soft tissue and muscle in the area of the cleft but the skin is not there

32
Q

What are secondary palates can be?

A
  • It can be complete or incomplete
    - Incomplete: can be as mild as a line in the midline of the uvula
    - Complete: follows the median palatine suture all the way to the incisive foramen
33
Q

What are fistulas? When does it occur?

A

Small holes that are usually along the embryological line → Post surgery

34
Q

What are the two types of fistulas?

A
  • Palatal fistula
  • Nasolabial fistula
35
Q

What is palatal fistula?

A
  • A hole in the palate after surgical intervention
  • Due to a partial breakdown after cleft repair
  • Unintentional fistula
36
Q

What is nasolabial fistula?

A
  • Located in the alveolus ONLY , not due to the breakdowns or dehiscence
  • “Intentional fistula” to allow growth of the premaxilla
37
Q

What are resonance disorders? It is not classified as? What happens in a resonance disorder?

A
  • Abnormal transmission of sound energy through the oral, nasal, or pharyngeal cavity
  • Causes the perception that people call “nasality”
  • Are NOT classified as voice disorders
  • Resonated in a cavity that shouldn’t be resonated in
38
Q

What are the types of resonance disorders?

A
  • Hyponasality
  • Hypernasality
  • Cul de sac Resonance
  • Mixed resonance
39
Q

What are the causes of resonance disorders?

A
  • Velopharyngeal dysfunction
  • Fistula
  • Obstructions
  • Misarticulations (motor planning)
40
Q

What is hypernasality?
What is it caused by?
It is always associated with?
Which sounds is it more perceptible?

A
  • Abnormal nasal resonance during oral sound production
  • Caused by abnormal coupling of the cavities
  • Always associated with oral speech sounds
  • Even more perceptible on high vowel sounds → higher the tongue position is, the lower the velum tends to be. So high vowel sounds like /i/ it tends to be more nasal in comparison as /a/
  • /m/ for /p/, /n/ for /t/, /ng/ for /k/

It gets resonated in the oral and nasal cavities.

41
Q

What are the causes of hypernasality?

A
  • A large velopharyngeal opening → cleft of the secondary palate
  • A thin velum (submucous cleft)
  • Large oronasal fistulas
  • Velopharyngeal Mislearning
42
Q

What is hyponasality? If severe occurs with which sounds? What is its cause?

A
  • Reduction in normal nasal resonance during speech; “stuffy” or “runny nose” vocal quality
  • Occurs primarily for /m, n, ŋ/ → /b, d, g/
  • Also occurs for vowel sounds if severe
    Cause: obstruction somewhere in the nasal cavity
43
Q

What is cul de sac? Sound is absorbed by the?

A
  • The acoustic energy is blocked from the exiting at the outlet
  • Sound is absorbed by soft tissues
44
Q

What is mixed resonance?

A

Combination of hypernasality, hyponasality, and cul-de-sac resonance

45
Q

Can hypernasality and hyponasality occur at the same time?

A

In the same utterance, yes, but for the same sound, no.

46
Q

What is the potential cause of mixed resonance disorder?

A

Apraxia or VPI + blockage

47
Q

What is velopharyngeal dysfunction? It is a major cause of?

A

Major cause of hypernasality
VP does not close consistently or completely during oral sound production

48
Q

What are the two types of VPD?

A

Velopharyngeal insufficiency
Velopharyngeal incompetence

49
Q

What is velopharyngeal insufficiency? In terms of speech therapy?

A
  • Anatomical defect
  • Prevents VP closure
  • SHORT velum
    This is not corrected through speech therapy.
50
Q

What is velopharyngeal incompetence? In terms of speech therapy?

A
  • Poor VP movement
    Neurophysiological
  • Velum may be long enough
  • The problem is the movement not the length of the velum
  • This is not corrected through speech therapy.
51
Q

What is velopharyngeal mislearning? What are the errors? It is secondary to? In terms of speech therapy?

A
  • Articulation disorder in response to abnormal structure
  • May be secondary to Velopharyngeal Insufficiency and may persist post surgery
  • Substitutes nasal/pharyngeal sounds for oral sounds
  • Corrected through speech therapy
52
Q

What is nasal emission? Which sounds is it most perceptible?

A
  • “Leakage” of airflow while attempting to build air pressure → plosives, fricative and affricate. These sounds require air pressure to be produced correctly. So in the production of these sounds, if airflow goes out of the nose, this is nasal air emission.
  • Voiceless sounds have higher pressure.
53
Q

What is the difference between NAE and hypernasality? Can they co-occur?

A

Difference of hypernasality and NAE:
- NAE is air escaping while
- hypernasality is sound escaping

NAE: Airflow (NOT SOUND) is released through the nose; more perceptible on voiceless sounds. Often occurs with hypernasality, but can occur with normal resonance.

54
Q

What are the types of nasal air emission?

A
  • Inaudible nasal air emission
  • Audible nasal air emission
  • Nasal rustle
  • Phoneme specific NAE
55
Q

What is inaudible nasal air emission?

A
  • Large opening, leakage is often imperceptible
  • Masked by hypernasality
  • The smaller the opening the more perceptible airflow is.
56
Q

What is audible nasal air emission? In terms of hypernasality?

A
  • Medium opening, more audible
  • Less hypernasality
  • The more hypernasal the less NAE and the more NAE the less hypernasal
57
Q

What is nasal rustle? In terms of hypernasality?

A
  • Small opening, causes the most distortions
  • Almost no hypernasality
58
Q

What is phoneme specific NAE? What is its cause?

A
  • Occurs only on selected sounds (/s/ or /z/) (not all pressured sensitive sounds are affected)
  • Due to faulty articulation (mislearning)
59
Q

What are obligatory errors?

A
  • Normal articulation placement
  • Errors caused by abnormal structure or physiology
60
Q

What are compensatory errors?

A
  • Misarticulations
  • Placement of the sound is wrong or incorrect in an attempt to produce the right sound
  • In response to abnormal structures
61
Q

What are the commonly exhibited obligatory errors?

A
  • Weak or omitted consonants - reduced airflow in the oral cavity
  • Short utterance length - nasal airflow reduces oral airflow for connected speech
  • Nasalization of oral consonants - nasal cognate replacement for oral stops (/n/ for /p/)
  • Nasal grimace - muscle contractions just above the nasal bridge
62
Q

What are the commonly exhibited compensatory errors?

A
  • Mid dorsum palatal stop - dorsum of the tongue against the middle of the hard palate ( For example, the sounds /t/ and /k/ are common stop consonants in English. However, in a mid-dorsum palatal stop, the tongue placement is between the usual positions for a /t/ (alveolar ridge) and /k/ (soft palate).
  • Velar plosive - Substitution for /k, g/ for anterior oral sounds (e.g., /k/ for /t/)
  • Velar fricatives/affricates - fricatives/affricates that are produced with the back of the tongue slightly elevated
  • Pharyngeal plosive - back of tongue to the pharyngeal wall, typically substituted for velar plosives
  • Pharyngeal fricatives
  • Posterior nasal fricative
  • Glottal stop - Voiced plosive adducting the glottis; usually substituted for glottal stops
  • Glottal fricative - usually substituted for oral fricatives /s/ for /h/
63
Q

Can obligatory errors be corrected through speech therapy?

A

No. It cannot be corrected solely through speech therapy because they stem from a physical or anatomical cause.