Physical and cognitive development in adulthood Flashcards
biological - 3
genetically predetermined path way - birth to death, mature, digress, multidirectional
asunchronous - never the same, each system move through diff patterns of aging and development
all the nurturing that were exposed to has an effect - food, PA, address
senescence -2
late adulthood
harmful events in terms of appearance and function like wrinkles and age spots
senility - 2
accumulation of senescence events to death - attached to disease processes
late adulthood - or earlier, some sort of disease
How to calculate life expectancy
go back to the year you were born
1900 life expectancy
50
2012 life expectancy M vs F
80 vs 84
life expectancy trend
went up and now slowing down
Do women outlive men?
gap thinning, originally thought to be x chromosome, industrialized (environmental
max life expectancy
under ideal conditions 120, oldest 122, but how long do you wanna live
2 theories of biological aging
levels of DNA and body cells
levels of organs and tissues
aging at the level of DNA and body cells - 2
programmed effects of genes - DNA make up
- aging genes called telomeres - caps that protect us from biological processes every time they replicate they shorten (supplementation?)
- disorders that support aging genes - closely linked to telomeres
cumulative effects of random events - phases where they want to then dont want to, body transitioning of what we do - to prevent aging
3 disorders supporting the aging genes
down’s syndrome - avg lifespan of 45, usually by CVD, accelerated aging process (decline of telomeres) like sensory deficits - spectrum disease
progeria - chr1, avg lifespan 13, CVD, 5-10 times their actual age, extremely small telomere and fast deterioation 0 most aggressive aging order thats rare and no way to predict - no genetic link - harmful senescense events but physiologically still the age they are
werners syndrome - chr8, recessive, onset 18-20, infertile, may reproduce before hand, accelerated process, 3-4 times regular, late diagnosis - 40-50
3 cumulative effects of random events
damage to DNA in cells - mutations
accumulations - less efficiency in repaire and replacement - aging
probable cause - free radicals - antioxidants to slow them down (good food gps)
levels of organs and tissues
cross linkage theory - fibres bend and crosslink to other fibres to make an x pattern which deterioate and speed up aging process quicker - predicable from biological parents
motor performance changes
decrease heart and lung function with gradual muscle loss - change in motor performance -reduced capacity/change sports/ maintain