Packet 3 - Blood (part 2) Flashcards
Cancer of WBCs and their precursors (abnormal proliferation and differentiation).
a. ) Anemia b.) Aplastic anemia
c. ) Hemolytic anemia d.) Leukemia
e. ) Neutropenia f.) Polycythemia
g. ) Thrombocytopenia
d.) Leukemia
Bone marrow failure causes drop in all three types blood cells.
a. ) Anemia b.) Aplastic anemia
c. ) Hemolytic anemia d.) Leukemia
e. ) Neutropenia f.) Polycythemia
g. ) Thrombocytopenia
b.) Aplastic anemia
Body destroys RBCs faster than normal. An example of this is sickle cell anemia.
a. ) Anemia b.) Aplastic anemia
c. ) Hemolytic anemia d.) Leukemia
e. ) Neutropenia f.) Polycythemia
g. ) Thrombocytopenia
c.) Hemolytic anemia
Increase during allergic reactions and parasitic infections.
a. ) neutrophils
b. ) eosinophils
c. ) basophils
d. ) B lymphocytes
e. ) T lymphocytes
f. ) natural killer (NK) cells
b.) eosinophils
What are the 5 stages of hemostasis?
- ) Vessel spasm / vasoconstriction
- ) Formation of platelet plug
- ) Blood coagulation
- ) Clot retraction
- ) Clot dissolution
Involved in cell-mediated immunity.
a. ) neutrophils
b. ) eosinophils
c. ) basophils
d. ) B lymphocytes
e. ) T lymphocytes
f. ) natural killer (NK) cells
e.) T lymphocytes
This stage of hemostasis triggered by contact with injured vessel wall, and by thromboxane A2 and other mediators. (platelet aggregation)
a. ) Formation of platelet plug
b. ) Clot retraction
c. ) Vessel spasm / vasoconstriction
d. ) Clot dissolution
e. ) Blood coagulation
a.) Formation of platelet plug
A decrease/deficiency in the number of platelets. S/S include bleeding, GI bleeding, hematemesis, bleeding heavily in between periods, bleeding gums. P/C include: bone marrow depression, splenomegaly, and autoimmune destruction.
a. ) Anemia b.) Aplastic anemia
c. ) Hemolytic anemia d.) Leukemia
e. ) Neutropenia f.) Polycythemia
g. ) Thrombocytopenia
g.) Thrombocytopenia
Involved in innate immunity.
a. ) neutrophils
b. ) eosinophils
c. ) basophils
d. ) B lymphocytes
e. ) T lymphocytes
f. ) natural killer (NK) cells
f.) natural killer (NK) cells
Lymphocytes are involved in adaptive/acquired immunity
Defend against pathogens and foreign substances. Function as phagocytes.
a. ) neutrophils
b. ) eosinophils
c. ) basophils
d. ) B lymphocytes
e. ) T lymphocytes
f. ) natural killer (NK) cells
a.) neutrophils
Mature forms called segs
Immature forms called bands
Increase in the number of RBCs. Increases viscosity/thickness of blood. Major organs to not get enough blood → increases blood volume → increase in BP → hypertension.
a. ) Anemia b.) Aplastic anemia
c. ) Hemolytic anemia d.) Leukemia
e. ) Neutropenia f.) Polycythemia
g. ) Thrombocytopenia
f.) Polycythemia
This stage of hemostasis is the development of a fibrin clot to stabilize platelet plug, related to activation of series of procoagulation factors that sequentially activate each other in a cascade effect.
a. ) Formation of platelet plug
b. ) Clot retraction
c. ) Vessel spasm / vasoconstriction
d. ) Clot dissolution
e. ) Blood coagulation
e.) Blood coagulation
Decrease in the number of WBCs.
a. ) Anemia b.) Aplastic anemia
c. ) Hemolytic anemia d.) Leukemia
e. ) Neutropenia f.) Polycythemia
g. ) Thrombocytopenia
e.) Neutropenia
Also called granulocytopenia.
This stage of hemostasis is when serum is squeezed from the clot, causing the edges of the broken vessel to be pulled together/joined.
a. ) Formation of platelet plug
b. ) Clot retraction
c. ) Vessel spasm / vasoconstriction
d. ) Clot dissolution
e. ) Blood coagulation
b.) Clot retraction
This stage of hemostasis allows blood flow to be reestablished, resulting in tissue healing. (fibrinolysis)
a. ) Formation of platelet plug
b. ) Clot retraction
c. ) Vessel spasm / vasoconstriction
d. ) Clot dissolution
e. ) Blood coagulation
d.) Clot dissolution
Become antibody producing plasma cells ⇒ humoral immunity.
a. ) neutrophils
b. ) eosinophils
c. ) basophils
d. ) B lymphocytes
e. ) T lymphocytes
f. ) natural killer (NK) cells
d.) B lymphocytes
During the blood coagulation stage of hemostasis, there is a development of a fibrin clot to stabilize platelet plug, related to activation of series of procoagulation factors that sequentially activate each other in a cascade effect: the ________ is activated by injury to blood vessels.
a. ) intrinsic pathway
b. ) extrinsic pathway
a.) intrinsic pathway
activated by injury to blood vessels
Mature forms of neutrophils are called ______.
Mature forms called segs
Immature forms called bands
Involved in allergic responses.
a. ) neutrophils
b. ) eosinophils
c. ) basophils
d. ) B lymphocytes
e. ) T lymphocytes
f. ) natural killer (NK) cells
c.) basophils
A decrease in the number of platelets is called _______.
thrombocytopenia
This stage of hemostasis is triggered by injury to a vessel, and thromboxane A2 (a prostaglandin released from platelets and cells).
a. ) Formation of platelet plug
b. ) Clot retraction
c. ) Vessel spasm / vasoconstriction
d. ) Clot dissolution
e. ) Blood coagulation
c.) Vessel spasm / vasoconstriction
During the blood coagulation stage of hemostasis, there is a development of a fibrin clot to stabilize platelet plug, related to activation of series of procoagulation factors that sequentially activate each other in a cascade effect: the ________ is activated by tissue injury.
a. ) intrinsic pathway
b. ) extrinsic pathway
b.) extrinsic pathway
activated by tissue injury
Low numbers of RBCs and/or hemoglobin levels. P/C factors include: nutritional deficiencies, blood loss, destruction/hemolysis.
a. ) Anemia b.) Aplastic anemia
c. ) Hemolytic anemia d.) Leukemia
e. ) Neutropenia f.) Polycythemia
g. ) Thrombocytopenia
a.) Anemia
Immature forms of neutrophils are called ______.
Immature forms called bands
Mature forms called segs
