Packet 10 - Fluid Balance (2) Flashcards
One of the major divalent cations in the body. Influences membrane potential and permeability, and is necessary for contraction of all muscle types.
a. ) Sodium
b. ) Calcium
c. ) Potassium
Calcium
One of the major divalent cations in the body. Influences membrane potential and permeability, and is necessary for contraction of all muscle types.
Helps regulate intracellular osmolality. Helps maintain resting membrane potential and generate action potentials in nerve and muscle tissue. Involved in regulating acid/base balance.
a. ) Sodium
b. ) Calcium
c. ) Potassium
Potassium
Helps regulate intracellular osmolality.
Helps maintain resting membrane potential and generate action potentials in nerve and muscle tissue.
Involved in regulating acid/base balance.
Regulates extracellular and vascular volume (r/t effect on osmolality). Helps maintain resting membrane potential and generate action potentials in nerve and muscle tissue. (Mostly extracellular)
a. ) Sodium
b. ) Calcium
c. ) Potassium
Sodium
Regulates extracellular and vascular volume (r/t effect on osmolality)
Helps maintain resting membrane potential and generate action potentials in nerve and muscle tissue.
Transcompartmental shifts (release of intracellular K related to burns, crush injuries, extreme exercise, or other types of cell damage).
a. ) Hyponatremia b.) Hypernatremia
c. ) Hypokalemia d.) Hyperkalemia
e. ) Hypocalcemia f.) Hypercalcemia
Hyperkalemia
increase in serum levels
P/C Factors:
- Decrease in elimination
- Renal failure
- Aldosterone deficiency
- K-sparing diuretics
- Excessive intake
- Oral or IV
- Transcompartmental shifts
- release of intracellular K related to burns, crush injuries, extreme exercise, other types of cell damage
Results in decreased membrane excitability (harder to generate action potentials).
a. ) Hyponatremia b.) Hypernatremia
c. ) Hypokalemia d.) Hyperkalemia
e. ) Hypocalcemia f.) Hypercalcemia
Hypokalemia
decrease in serum levels
P/C Factors:
- Inadequate intake
- Excessive loss
- Renal (i.e. diuretic therapy, excessive aldosterone or glucocorticoids)
- GI (i.e. vomiting, diarrhea)
- Skin (i.e. heavy sweating, burns)
- Transcompartmental shifts
- from extracellular to intracellular, r/t pH changes
Acute form causes decreased membrane excitability (muscle weakness, flaccid muscles, cardiac dysrhythmias, constipation, and stoopor. Chronic form causes kidney stones.
a. ) Hyponatremia b.) Hypernatremia
c. ) Hypokalemia d.) Hyperkalemia
e. ) Hypocalcemia f.) Hypercalcemia
Hypercalcemia
increase in serum levels
Acute form:
- Decreased membrane excitability (too much of the membrane stabilizer
- muscle weakness
- flaccid muscles (no reflexes, no muscle tone, flabby muscles)
- cardiac dysrhythmias
- constipation (no peristalsis)
- stoopor (extreme lethargy)
Chronic form:
- kidney stones
P/C Factors:
- increased bone resorption
- excess parathyroid hormone
- prolonged immobility
- some cancers
- increase in intestinal absorption
- excess vitamin D3
- excess dietary calcium
Excessive water intake in relation to output.
a. ) Hyponatremia b.) Hypernatremia
c. ) Hypokalemia d.) Hyperkalemia
e. ) Hypocalcemia f.) Hypercalcemia
Hyponatremia
Decrease in serum sodium
P/C factors:
- increase in sodium/water loss, accompanied by replacement with sodium-free fluid.
- Skin (i.e. excessive sweating, burns)
- GI loss (i.e. vomiting, diarrhea, tap water enemas)
- Renal (i.e. aggressive diuretic therapy)
- Excessive water intake in relation to output (dilutional hyponatremia)
- Psychogenic polydipsia
- increased intake + impaired elimination
- kidney disease and increase in ADH levels
S/S include alkalosis, muscle weakness, decreased peristalsis (constipation → paralytic ileus), cardiac disrhythmias, and polyuria.
a. ) Hyponatremia b.) Hypernatremia
c. ) Hypokalemia d.) Hyperkalemia
e. ) Hypocalcemia f.) Hypercalcemia
Hypokalemia
decrease in serum levels
P/C Factors:
- Inadequate intake
- Excessive loss
- Renal (i.e. diuretic therapy, excessive aldosterone or glucocorticoids)
- GI (i.e. vomiting, diarrhea)
- Skin (i.e. heavy sweating, burns)
- Transcompartmental shifts
- from extracellular to intracellular, r/t pH changes
If severe, cells can only depolarize and cannot repolarize (cardiac arrest).
a. ) Hyponatremia b.) Hypernatremia
c. ) Hypokalemia d.) Hyperkalemia
e. ) Hypocalcemia f.) Hypercalcemia
Hyperkalemia
increase in serum levels
P/C Factors:
- Decrease in elimination
- Renal failure
- Aldosterone deficiency
- K-sparing diuretics
- Excessive intake
- Oral or IV
- Transcompartmental shifts
- release of intracellular K related to burns, crush injuries, extreme exercise, other types of cell damage
Transcompartmental shifts (from extracellular to intracellular related to pH changes).
a. ) Hyponatremia b.) Hypernatremia
c. ) Hypokalemia d.) Hyperkalemia
e. ) Hypocalcemia f.) Hypercalcemia
Hypokalemia
decrease in serum levels
P/C Factors:
- Inadequate intake
- Excessive loss
- Renal (i.e. diuretic therapy, excessive aldosterone or glucocorticoids)
- GI (i.e. vomiting, diarrhea)
- Skin (i.e. heavy sweating, burns)
- Transcompartmental shifts
- from extracellular to intracellular, r/t pH changes
Results in increased membrane excitability (too easy to generate action potentials).
a. ) Hyponatremia b.) Hypernatremia
c. ) Hypokalemia d.) Hyperkalemia
e. ) Hypocalcemia f.) Hypercalcemia
Hyperkalemia
increase in serum levels
P/C Factors:
- Decrease in elimination
- Renal failure
- Aldosterone deficiency
- K-sparing diuretics
- Excessive intake
- Oral or IV
- Transcompartmental shifts
- release of intracellular K related to burns, crush injuries, extreme exercise, other types of cell damage
S/S include intercellular dehydration (dry skin, dry mucous membranes, and no tears).
a. ) Hyponatremia b.) Hypernatremia
c. ) Hypokalemia d.) Hyperkalemia
e. ) Hypocalcemia f.) Hypercalcemia
Hypernatremia
increase in serum sodium
P/C Factors:
- Excess water loss
- Renal (i.e. polyuria)
- GI (i.e. watery diarrhea)
- Skin (i.e. increase in sweating)
- Decreased water intake
- Thirst defect or inability to drink
- Excessive sodium intake
- Oral or IV administration, near-drowning in salt water
Acute form causes increased membrane excitability (paresthesia, seizures, cardiac dysrhythmias, tetany [muscle spasms/twitching]), and Laryngospasms (airway obstruction). Chronic form results in osteoporosis.
a. ) Hyponatremia b.) Hypernatremia
c. ) Hypokalemia d.) Hyperkalemia
e. ) Hypocalcemia f.) Hypercalcemia
Hypocalcemia
decrease in serum levels
Acute form:
- increased membrane excitability (too little of the membrane stabilizer).
- paresthesia
- seizures
- cardiac dysrhythmias
- tetany (muscle spasms/twitching)
- Laryngospasms (airway obstruction).
Chronic form:
- osteoporosis.
P/C Factors:
- Decrease in intestinal absorption
- decrease in vitamin D3 malabsorption
- Decrease in ability to mobilize from bone
- related to altered parathyroid function
- Abnormal renal loss
- renal failure
- increase in protein binding
- alkaline pH
If mild, the person experiences tingling (paresthesia) and increased peristalsis (diarrhea).
a. ) Hyponatremia b.) Hypernatremia
c. ) Hypokalemia d.) Hyperkalemia
e. ) Hypocalcemia f.) Hypercalcemia
Hyperkalemia
increase in serum levels
P/C Factors:
- Decrease in elimination
- Renal failure
- Aldosterone deficiency
- K-sparing diuretics
- Excessive intake
- Oral or IV
- Transcompartmental shifts
- release of intracellular K related to burns, crush injuries, extreme exercise, other types of cell damage
ECF is hypotonic causing fluid to move into the cells (swelling of body cells). Notice first when brain cells start to swell (neurological symptoms: lethargy, headache, nausea and vomiting, motor weakness, confusion, seizures, or coma).
a. ) Hyponatremia b.) Hypernatremia
c. ) Hypokalemia d.) Hyperkalemia
e. ) Hypocalcemia f.) Hypercalcemia
Hyponatremia
Decrease in serum sodium
P/C factors:
- increase in sodium/water loss, accompanied by replacement with sodium-free fluid.
- Skin (i.e. excessive sweating, burns)
- GI loss (i.e. vomiting, diarrhea, tap water enemas)
- Renal (i.e. aggressive diuretic therapy)
- Excessive water intake in relation to output (dilutional hyponatremia)
- Psychogenic polydipsia
- increased intake + impaired elimination
- kidney disease and increase in ADH levels