Otorrino ordinario Flashcards
Nasal epithelium
ciliated, pseudostratified columnar
a donde drena el nasolacrimal duct
al inferior meatus
a donde drenan los frontal, maxillary and anterior sinues
al ostheomeatal complex
a donde drena el posterior ethmoid sinus
al superior meatus
a donde drena el anterior ethmoid sinus
middle meatus
a donde drena el sphenoid sinus ostia
to the superior turbinate
the vasculatiry its given by..
Internal carotid –> ophthalmic–> anterior and posterior ethmoid arteries
External carotid –> sphenopalatine artery
the venois drainage its by…
pterygoid and ophtalmic plexuses
Principales symptoms of rhinits
nasal obstruction, hypeirrtibility and hypersecretion
persistent rhinits
more then 4 days a week AND more the 4 weeks
intermitent rhinits
symptoms for less then 4 days a week or less then 4 weeks
mild vx moderate/severe rhinits
normal or abnormal sleep (moderate and severe)
symptons of NON allergic rhinitis
nasal obstruction, clear rhinorrhea,
not tha common: sneezing, itchy and watery eyes
viral non allergic rhinits
associated with other manifestations of viral illness: headache, malaise, body aches, cough.
occupational non allergic rhinits
pollutants like dust, ozone, garden sprays, etc. (irritant agents). Nasal dryness.
vasomotos non allergyc rhinits
symptons associated with changes of temperature, eating, alcohol use, etc.
non allergyc rhitis with eosinophilia
they have more severe exacerbations (eosinophilia >25%)
medicamentosa non allergic rhinits
because of the over-the-corner topical vasoconstrictive nasal sprays (Afrin most common drug that gives this)
pregnancy non allergic rhinits
rise of estrogen –> rise of hyaluronic acid –> rise of nasal edema and congestion
basic tx of non allergic rhinits
-Irritant avoidance
-Saline irrigation (for a clean nose, improves ciliary function)
-Topical Intranasal steroids (reduce eosinophil and neutrophil chemotaxis and inflammation) (don’t give IV, IM)
-a-adrenergic drugs (2 main familys: phenylamines [contraindicated in px with hypertension, coronary artery disease, etc] and imidazolines)
-Anticholinergics (ipratropium, azelastine, cromolyn, etc)
for mild intermittent/persistent non allergic rhinits TX
- INAH
- INCS
for moderate/severe intermittent non allergic rhinitis tx
- INAH
- INCS
- IN (AH & CS) o INAH + INCS
for moderate/severe persisiten non alergic rhintis tx
- IN (AH & CS) o INAH+ INCS
- INAH
- INCS
surgical measures for non allergic rhinits
septoplasty and turbinate surgery (inferior turbinate)
*for anatomical problems
pathogenesis de allergic rhintis
IgE- mediated inflammatory nasal condition resulting from allergen introduction (sensitazion fase) and early inflammation by mast cell that liberates histamine and then late inflammation caused by chemotaxis (2 phase) (type 1 hypersensitivity)
typical symptoms of allergic rhinits
sneezing, itching, rhinorrea and nasal congestion
classification: season allergic rhinits
symptoms usually worst in the morning, aggravated by dry, windy conditions
classification: perennail allergic rhinits
only happens in specific moment or places, indoor inhalants like dust mites, animal dander, mold spores, etc. Most common sign its nasal congestion
FR de allergic rhinits
family history (atopias)
Male sex
Birth during the pollen season
1 born
Early use of antibiotics
Maternal smoking exposure
Exposure to allergens
Associated chronic conditions: asthma, otitis media with effusion
for intermittent mild allergic rhintis tx
- OAH o INAH
- OAH + PSE
- INCS
for intermittent moderate/severe allergic rhinits tx
- OAH o IANH
- INCS
- IN (AH & CS) o INAH + INCS
- OAH
for persisiten mild allergic rhintis tx
- INCS
- OAH o INAH
- OAH + PSE
- Intranasal cromolyn sodium
for persistent moderate/severe allergic rhinits tx
- IN ( AH & CS) or INAH+ INCS
- INCS
- INAH
dx for allergic rhinits
-Complete HC
-Physical examination
-Rhinoscopy: bluish, pale, boggy turbinates, wet and swollen mucosa, nasal congestion (predominant sign in perennial allergies), anatomic abnormalities
-Conjunctivitis, eczema, asthmatic wheezing, nasal salute, OME
-Allergy testing
-Skin testing (skin-prick test SPT most common, (gold standard)
-ID testing
-In vitro testing
Etiología rhinosinusitis
90% viral (sintomas < 10 dias y no empeoran)
sintomas que te orientan a bacteriano: purulent rhinorrhea, facial pain/pressure, and nasal obstruction., fever >38°, unilateral
bacterias mas comunes en ARS
Streptococcus pneumoniae, Haemophilus influenzae, M. catarrhalis
bacterias más comunes en CRS
S. auerus. P. auriginos, H. influenzae, Anaerobios
acute rhinosinusits ARS generalidades
incia como un resfriado, normalmente es vrial ( adenovirus, rhinovirus, respiratory virus)
acute rhinosinusitis ARS dx
2 o mas sintomas, 1 a fuerzas nasal congestion o nasal discharge y el otro facial pain, reduction or loss of smell de > 4 semanas
subacute rhinosinusitis
4-12 weeks
recurretn acute rhinosinusists
4 or more episodes in 1 year, with complete resolution between episodes
CRS
2 or more symptoms, one of which should be either nasal blockage/congestion or nasal discharge, and facial pan/pressure, reduction or loss of smell for > 12 weeks
classification of CRS
-primary CSR: unilateral o bilateral
-secundary CSR: localize o diffuse
dx rhinosinusists
2 o mas major factors + 1 minos
mayor factors RS
facial pain/pressure
nasal obstruction/blocakege
nasal dishcarge/ postnasal drainage
hyposmia
purulance
fever
minor factors RS
headache
fever
halitosis
fatgiue
dental pain
cough
ear pain
caldweel RX
for ethomoidal and frontal
water rx
maxillary
lateral rx
Sphenoid, Frontal, ethmoidsandmaxillary
lund mackey system
del 0 al 2(obstruccion total) y en ostiomeatal complex es 0 o 2 , se califica cada lado, de 0-6 es leve, 6-10 moderado y >10 severo
que sinues califica lund mackey system
-frontal
-anterior ethmoidal
-posterior ethmaidal
-maxillary
-esphenoid
-osteomeatal complex
pathogenesis the RS
Mucosal swelling (allergy, infection, enviorment, etc)–> obstruction of sinus ostia –> mucus stasis –> infecion
TX ARS
Saline irrigation
Nasal steroids
Antibiotics (for bacterial): amoxicillin with clavunate
Antihistamine
Systemic steroids
Decongestant (like Afrin: oxymetazoline)
tx CRS
Antibiotics
Nasal steroids
Saline irrigation
Leukotriene antagonist
Oral steroids
Antihistamine
Monoclonal bodies (omalizumab [ige] or mepolizumab [IL5]
Antifungal
fuctional endsocpic sinus surgery
tx for CRS with polips
dupilamab
tx fungal infectio RS (common in no control DM)
Anfortemince B
alarma symptoms in RS (inmediate referral)
-periorbital edema/erythema
-displaced globe
-dobule vision
-ophtalmoplejia
-reduced visual acuity
-severe headche
-frontal swelling
-signs of sepsis
-signs of meningitis
-neurologicla signs
chandler complicatiosn of SR
1 inflammatory edema: no visual lost ni ophtalmoplejia
2 Orbital cellulitis: pain, proptosis, chemosis, little ophthalmoplejia, edema of extra vascular muscles and mild disminución of agudeza visual tx: intravenous antibiotic
3 Subperiosteal abscess: operative drainage
4 Orbital abscess: proptosis, chemosis, ophtalmoplejia, visual lost operative drainage
5 Cavernous sinus thrombosis: tromboflebitis, 3,4,5 NC affected, life threatening, IV antibiotic + OR drainage
most common emergency 60% in otorrino
epistaxis
causes epistaxis
1° idiopathic
2° traumatic
3 iatrogenic
leucemia (common in children)
irrigation of nasal cavity
EXTERNAL CAROTID
-facil artery (anterior nasal septum)
-internal maxillary artery
-sphenopalatine artery
(septal y conchal brand)
-descendign palatine
artery
irrigation of nasal cavitiy
INTERNAL CAROTIDE
-ophtalmic artery
-anterior ethomoid artery
-posterior ethomoid artery
el 90% de anterior epistaxis viene de
Kieselbach plexus or littles area
la anterior y posterior (dificl de controlar la bleeding) epistaxisi se divide por el
ostium of the maxillary sinus
cuantos ml es lo habitual de epistaxisis
700-900 ml
manegement epistaxis
-initial assesment (HC, EF, BH, future crossmatching)
-headlamp examination (local anestesia)
-nasal endoscopy (mostly in posterior)
- identify site de sangrado
- buscar patolgoia desecadenante
- digital pressure on alar cartilages fot 20 min
most common source of bleedign in children
from a vessel in the mucocutaneous junction
5-10% of epistaxisis in children its cause
of an undiagnosed von willbrand disease
tx epistaxis
-topical silver nitrate
-petroleum jelly (Best method)
-cautery in OR (NEVER bilateral)
-nasal packign with antibiotic (gauze, bilateral,) (never in children)
*if after packing sigue sangrado, suele ser posterior, y se manda al otrorrino para poner foley catheter
every posterior epistaxis belongs in the hospital
true
tx posterior epistaxis
-nasal packing
-gold standar: cauterization (endoscopic sphenopalatin artery ligation)
-maxillary a., external carotid a. ligation
-embolization
donde es mas comun una neoplasia paranasal de sinus
en el maxilary sinus
sintomas que sugieren proceso neoplasico
unilateral swelling, pain, and epistaxis.
que es lo primero que pido ante un tumor
CT scan WITH constras and RM (T1 liquod negro, T2 liquido blanco)
inverted papilloma “scheinderian tumor”
Most common benignal tumor, HPV has 75% grade of malignization, usually men, usually on maxilary sinus, pale, multiboluted, dx CT with contrast and MR
-TX: surgical resection +adyuvant RT
osteoma
slow growth, benignal, 2-5° decade of life, males, msot common place: anterior ethomidal, the frontal then maxillary then sphenoidal
-tx: watchfull waiting, open approach
juvenile angiofibroma
vascular tumor, adolscent boys, usually in the pterigomaxilar fosse. has holamn miller sign (tumor psuhes the posterior bone of maxillary sinus)
juvenile angiofibroma manifestations, and tx
-common manifestation: recurrent epistaxis, nasal voice, eustachian tube dysfunction
-tx: surgical resection after embolization preop
AVOID BIOPSY
Lobular capillary hemangioma
caused by Nasal trauma
Microscopic AVM
Veryyy small and can grow sooo much
beingn
fibrous dysplasia
-tumor on the bone medula
-loose teeth, numbness, facr assymetry
-tx: observation, embolization 1°, cx (quitan todo el hueso, queda el hueco) 2°,
squamosu cell carcioma
-most common mallignat tumor
-cx + rt
-bado rpognosis
olfatory neuroblastoma
-olfacory epithelium
-malignat
-extension to the orbit and anterior fossa
-kadish classification
ESTHESIONEUROBLASTOMA
from the olfatory epitheluim
-anosmia, iposmia
larynx roles
-protection (epiglotis, vocal cords, cough
-respiration
-phonation
we divide the larynge in:
-supraglottic (suprahyoid and infrahyod epiglotis, ariepiglotic folds, aritenoids, vocal fols)
-glottis (vocal cords)
-subglottic
cartilages of the larynx
-Epiglottis
-Thyroid (biggest, protection of vocal cord)
-Cricoid (like an incomplete ring, attachment with the traquea)
-Cuneiform
-Arytenoid
muscles in the larynx are inerveted by the
-recurretn laryngeal nerve (branch of vagus)
-other ones: superior laryngeal nerve (external [moves cricothyroid msucle] and internal [gives the sensibility])
vocal cords
-anterior view es donde se forma el pico de la V
-es un squamous stratified epitheluim
-most common cancer in cords: suqamosu cell carcinoma
-has tyroarithenoid muslce
que divide larynge de hipofarigne?
the aryepiglothic
4 types of cords paralisis
-vagal bialteral
-vagal unialteral
vocal cords in intermediate position (cadaveric)
-recurrent laryngeal bilateral
-recurrent laryngeal unilateral
parameida position
*recurrent larygneal are more common
*leer its more common
vagal bilaterla cord paralysis
idiopathic or neurological causes, CANT generate voice, hisotry of choking
vagal unilateral cord paralysis
iatrogenic, neoplasia, brainstem infarction
-weak, breathy hoarseness
-history of aspiration
recurrent laryngeal cord paralysisi bilateral
-usually after a cx
-Stridor, problems breathing
-no dysphonia
-its an emergency
tx of recurrent laryngeal cord paralysis
-lateralization of vocal cord (but wont be able to speak)
-or cordectomy
recurrent laryngeal cord paralysisi unilateral
-causes: neoplasia, iatrogenic, trauma, anerurysm (left)
-hay disphonia
-bovine cough
-tx: vocal and speech therapy, hyalurnic acid or fat to the cord, or thyroplasty (impkan tpushes the cord to middle line)
bening laryngeal lesions
vocal cord nodules, polyps, intubation granuloma, reinke edema, laryngeal cyst, laryngocele, papillomatosis
vocal cord nodules
-comunes en gente que usa mucho la voz
-nodulos no permiten que se toquen las cuerdas: dysphonia
-bilateral
-in the junction of the 1/3 anterior and 2/3 posterior
-tx: speehc therapy 3 meses, no se extirpan porque crecen back
vocal cord polyps
-unilateral
-red and pednculated
-por reflujo o UN esfuerzo vocal grande
-tx: speech therapy 3 months, omprazol and cx (take out polyp)
intubation granuloma
-usually unilateral
-neer to the arythenoid process
-in posterior 1/3
-px with intubation hisotory
-tx: NO smoking or drinking, dietic control, speech therapy or cx (quitas tejido para adelgazar la cord)
reinke edema
-Bilateral in the lamina propia
- Diffuse polyposis of the vocal cord (like they have water inside)
- Risk factor: smoking
- Clinic: raspy voice (like alejandra guzman)
- Treatment
o Avoid smoking, food irritants and voice overuse
o Speech therapy
o If that doesn’t work: surgery
laryngeal cyst
-usually unilateral
-INSIDE the mucosa (full of mucosa and epithelial cells)
-big
-dysphonia
-tx: surgery 1°
papillomatosis
-infection by VPH 6 and 11
-children
-lesiones verrugosas en larynx
-dysphonia
-tx: surgery (but they grow again)
malignant laryngela lesions donde es mas comun de las vocal cros
1 glotis 59%
2 supraglotis 40%
3 suglotis 1% (bad prognosis)