other types of injuries Flashcards

1
Q

Pregnancy and trauma
Mechanism of Injury
think what sort

A

Possible causes of trauma:
Domestic violence
RTC’s
Falls from height

Possible trauma to enlarged internal organs
Organs are displaced especially in the third trimester
Anatomy and physiology somewhat altered

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2
Q

Pregnancy and trauma
Mechanism of Injury
Consider…..

A

Signs of shock appear very late
Hypotension is an extremely late sign
Any signs of hypovolaemia = stage 3 shock
Compression of the inferior vena cave >20 weeks is a serious potential complication

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3
Q

Pregnancy and trauma

Assessment and Treatment

A
Consider MOI (could have been 3-4/7 ago)
Ensure AcBC are assessed and managed
O2 @ 15L per minute
Stage of pregnancy
Specifically assess for abdominal pain
Vaginal blood loss
Analgesia
15-30 degree tilt to the left if supine
Anything else…….
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4
Q

Suspension injuries

What is it….

A
Often no physical injury
Form of syncope
Causes venous pooling in the legs
Reduced cardiac output due to reduced                              venous return
Reduced cardiac output = fall in BP
Known as orthostasis
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5
Q

Suspension injuries

Treatment

A

No evidence that toxins build up as suspected by several authors
No need to change first aid procedures
As soon as possible, place in horizontal position even if still suspended
High flow O2 if unconscious
Consider they may have injuries that have caused the unconsciousness, might not be a syncope

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6
Q

Blast injuries

Mechanism of Injury

A

Terrorism - CBRNe
Industrial accidents
Potential for mass casualties
Blast waves can travel at 5000m per second – https://youtu.be/2yWwAsABIlM
Multi system injury
Initial blast wave increases pressure in the body causes stress and shear in gas filled organs
Immediate death (barrow trauma) occurs more often in enclosed space than open air bombings
Injuries generally categorised into five groups:
Primary
Secondary
Tertiary
Quaternary
Quinary

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7
Q

Blast injuries

Signs and Symptoms

A

Primary – tympanic membrane rupture, blast lung, eye injuries, concussion
Secondary – penetrating injuries, traumatic amputations, lacerations
Tertiary – blunt injuries, crush syndrome
Quaternary – burns, inhalation injury
Quinary – injuries from ‘dirty bombs’ such as bacteria and radiation (NAEMT, 2011)

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8
Q

Ballistic injuries

Mechanism

A
Type of GSW
Medium energy (handguns and some rifles)
High energy
Caliber
Missile size
Bullet construction
Tumbling/yaw
Distance traveled – increased distance will decrease the energy at impact resulting in less injury
Internal wound
Tissue contact damage
High-velocity transfer of energy
Shock waves
Temporary cavity up to 25 x greater than caliber of bullet
Pulsation of temporary cavity
Highly dense tissue sustains more damage
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9
Q

Gunshot Wounds: Cavitation Signs and Symptoms

A

Pain
Wounds – entry and exit
If shotgun potential for up to 2000 pellets (birdshot shell)
Consider the anatomy the GSW has potentially effected
What was the bullet trajectory?

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10
Q

Gunshot Wounds:Assessment and Treatment

A

Safety – ballistic trained staff to respond with Police if still active shooting
Catastrophic haemorrhage management
Seal open chest wounds
Analgesia
IV access and consider fluids (paramedic)
TXA
Emergency Department with pre-alert

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11
Q

Drowning

A

Conscious – hold breath <60 seconds
Victim will then take a breath inhaling fluid
Laryngospasm will occur
This is rapidly terminated by hypoxia and aspiration will resume
To drown requires active ventilation while submerged hypoxia, hypercarbia and acidotic = unconsciousness and apnoea

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12
Q

Diving Response

Drowning

A

Diving Response
Cooling of cold thermoreceptors on the face
Profound bradycardia
Parasympathetic stimulation
Sympathetic nervous vasoconstriction to trunk and limbs
Stimulation of both parasympathetic and sympathetic nerves can result in life threatening arrhythmias

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13
Q

Drowning

Signs and Symptoms

A

Submersed or immersed in fluid
Respiratory impairment
Absence of breathing
Cardiac arrest

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14
Q

Drowning

Management

A

Do not go within three metre’s of the water’s edge unless you are water rescue trained and have the appropriate PPE
If conscious, encourage them to self rescue – buoyancy aids
If unconscious, rescue as soon as safely possible
How long have they been submerged if <90 minutes resuscitate
Remove horizontally from water

Increased pressure on the legs from the water increase venous return and cardiac output. Baroreceptors mistake this for hypervolaemia and increase diuresis. Alomgside this there is peripheral vasoconstriction which exacerbates this further. When removed from the water, this pressure is lost and the sudden reduction in venous return can result in cardiac arrest so remove from water horizontally to aid venous return.

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15
Q

Drowning

Management Cardiac Arrest

A

If still in water, ventilations only, 10-15 per minute
Incidence of spinal injuries is low (0.5%) and immobilising can cause delay and inadequate resuscitation
Spinal immobilisation is not indicated unless signs of severe injury are apparent or MOI is consistent with possibility of severe injury
Must be removed from water as quickly as possible even if back board not available

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16
Q

Drowning

Management Cardiac Arrest

A

Initial five rescue breaths as main cause of cardiac arrest is hypoxaemia
Regurgitation is likely, have suction ready
Consider early intubation
If hypothermic <30C limit defibrillation to three attempts and withhold IV drugs until >30C
IV fluid to correct hypovolaemia
‘not dead until they are warm and dead’

17
Q

Crush injuries What is it….?

A

Also known as traumatic rhabdomyolysis
Arises from a crushing type injury to large muscle mass commonly thigh or calf
Destroyed muscle releases myoglobin and potassium which causes damage to the kidneys and life threatening cardiac arrhythmias
When released the affected limb is perfused but the old blood with elevated chemicals floods the rest of the body
Onset after 30 minutes, can be less so assess your patient

18
Q

Crush syndrome is characterised by:

A
Crush syndrome is characterised by:
Hypovolaemic shock (due to sequestration of water in the injured muscle cells)
Hyperkalaemia (release of cellular potassium by the injured muscle cells)