Neuromuscular system Flashcards
The Frontal lobe is responsible for
Judgement, problem solving, creativity, logic, understanding consequences, Emotions,
Memory and learning
Brocas area - Non-fluent expressive aphasia
Motor cortex
Parietal lobe
Object recognition, sense of taste
Somatosensory cortex
Occipital lobe
Visual cortex
Temporal lobe
Hearing, smell, memory
(L) hemisphere - wernikes area, fluent, receptive aphsia
Hippocampus
Short to long term memory
Reticular activating system
Regulates alertness,
Deep sleep, drowsy, low alert, high alert
Anterior spinal artery
Supplies middle medulla including ant spinal cord
Posterior inferior cerebellar artery
Supplies cerebellum
Vertebral artery
supplies the post part of the circle of willis
Ascending pathways are also called and carry which information?
Afferent pathways - carry sensory information to the brain
Decending pathways are also called what and carry which information?
Efferent pathways - control motor function and muscle tone,
The Dorsal column medial lemniscal tract carries which information on which side?
Crosses in the medulla, travels ipsilaterally
Proprioception, vibration, tactile discrimination
Separated into fasiculus cunatus: upper extremity
Fasciculus gracilis: lower extremity
The spinothalamic tract carries which information on which side?
Pain and temperature (lateral) Crude touch (anterior) on the contralateral side. Decussation at the level.
The corticospinal tract is responsible for what?
Voluntary motor control
Vestibulospinal tract is responsible for what?
Muscle tone, antigravity muscles, Postural reflexes, head on trunk stabilisation, VOR
Rubrospinal tract
Proximal limb flexors - causes flexion in the upper extremities.
Reticulospinal tract
Excitatory to extensors and axial muscles (medial) inhibitory to extensors (lateral)
What is usually the cause of decreased DTR? and Increased?
Peripheral issue for decreased and a central issue for exaggerated
CN I
Olfactory - smell, sensory only
CNII
Optic - Vision, sensory only
CNIII
Oculomotor - Eye movements, depress, elevate, adduct
Motor only
CNIV
Trochlear - Eye movement - depress and adduct
Motor only
CN V
Trigeminal - sensation of face, scalp, eyeball and tongue
- masseter and temporalis jaw closing
Mixed
CNVI
Abducens - eye movement - abduction,
Motor only
CNVII
Facial - eye closing, facial expression, taste on ant 1/3 tongue
Mixed
CNVIII
Vestibulococlear - hearing and balance
Sensory only
CNIX
Glossopharyngeal - taste to post 2/3 tongue, Gag reflex
CNX
Vagus - sensation and motor to heart and lungs
Mixed
CNXI
Accessory - SCM and trap function. shoulder shrug
Motor oNly
CNXII
Hypoglossal - tongue movements Motor only
Dermatomes: C1
Top of skull
Dermatomes: C2
Temple, forehead, occuiput
Dermatomes: C3
Neck, post cheek, temporal area, under mandible
Dermatomes: C4
Tip of shoulder, upper scap, clavicle
Dermatomes: C5
Deltoid, ant arm to base of thumb
Dermatomes: C6
Ant arm - radial hand thumb and index finger
Dermatomes: C7
Medial arm and forearm to middle finger
Dermatomes: C8
Ring and pinky finger, ulnar forearm
Dermatomes: T1
Medial forearm
Dermatomes: T2
Axilla
Dermatomes: T3-6
Upper thoracic (T4 nipple)
Dermatomes: T5-7
Costal margin
Dermatomes: T7-12
Lower abdomen (T10 belly button, T12 ASIS)
Dermatomes: L1
Upper groin, lower back
Dermatomes: L2
Lower groin, ant thigh and knees
Dermatomes: L3
Sacral area, medial knee, ant lower leg
Dermatomes: L4
Med lower leg - medial calf and ankle
Dermatomes: L5
ant tibia, toes
Dermatomes: S1
Lat foot, plantar foot, Gastroc/soleus
Dermatomes: S2
Post thigh, lateral leg
Dermatomes: S3
Buttock
Dermatomes: S4
Rectum, anal area, saddle
Myotomes: C5
Deltoid, shoulder abduction
Myotomes: C5/6
Elbow flexion, wrist extension
Myotomes: C7
Elbow extension wrist flexion
Myotomes: C8
Ulnar deviation, FCU, ECU
Myotomes: T1
digit abd/add interossei
Myotomes: L2/3
Hip F
Myotomes: L3/4
Knee extension
Myotomes: L4/5
Ankle DF, toe ext
Myotomes: S1
Plantar flexion, Gastroc
ASIA impairment Scale - ASIA A
Complete, no sensory or motor function preserved
ASIA impairment Scale - ASIA B
Sensory incomplete: sensory preserved but not motor
ASIA impairment scale ASIA C
Motor incomplete, motor preserved but muscle grade <3/5
ASIA impairment scale ASIA D
Motor incomplete, motor preserved, muscle grade >3/5
ASIA impairment scale ASIA E
Normal - sensory and motor function normal
Rancho Los Amigos level of cognitive function
I: No response II: Generalised response III: Localised response IV: Confused agitated V: Confused inappropriate VI: Confused appropriate VII: Automatic VIII: Purposeful
Glasgow Coma Scale stages of severity
8 or less; severe
9-12 moderate
13-15 mild
Deep Tendon Reflexes: Biceps: Brachioradialis: Triceps: Patellar: Achilles:
Biceps: C5,C6 Brachioradialis: C5/6 Triceps: C7 Patellar: L3/4 Achilles: S1/2
Grades of DTR
0 - no response 1+ - diminished response 2+ normal response 3+ brisk/exaggerated response 4+ Hyperactive - always abnormal
Superficial reflexes include;
Abdominal T8-L1
Corneal Blink CN V and VII
Gag reflex CN IX and X
Cremasteric Reflex L1-2
Wheelchair measurements should be
Seat height +2
Seat depth -2
Seat width +2
Back height -4
Functional independence measure (FIM)
Score 18-126 with each item scored 1-7
1 total assistance, 2 max assist, 3 mod assist, 4 min assist, 5 supervision. 6 modified independent (device), 7 complete independence.
Self care, bladder bowel, transfers, locomotion, communication, social cognition
Tinetti Balance test
Sit, sit to stand, attempt to rise, first 5 sec standing, standing nudged, eyes closed, 360 degree turn, sit
Dynamic Gait index
Level surface change in speed horizontal head turns Vertical head movement gait pivot and turn Step over obstacle Step around obstacles Stiars. <19/24 fall risk >22 safe ambulator
Brunnstroms stages
- No spasticity - flaccid, no synergy no function
- Spasticity onset + weak associated movements
- Peak spasticity, voluntarily perform synergy
- Spasticity decreasing, partial limb synergy
- Spasticity decreasing, independent of synergy, almost clear
- Minimal spasticity, free of synergy, isolated joint movements with co-ordination
- Normal
Alzheimers
progressive mental deterioration, deterioration of cerebral cortex and subcortical areas. Low neurotransmitters and high aluminum.
- loss of orientation, word finding difficulty, depression, poor judgment, rigidity, slow movement, shuffling gait
ALS
Chronic degenerative UMN and LMN impairment
Ant cord syndrome
Hyperflexion injury.
Loss of spinothalamic tracts with bilateral loss of pain and temperature.
DCML pathway preserved.
Bilateral loss of motor function, spastic paralysis below level of the lesion
Motor function, pain and temp.
Arthrogryposis multiplex congenita
Occurs during first trimester, fibrosis of muscles and joint structures. Non-progressive.
Multiple contractures, joint dislocation, atrophy, cylindershaped limbs with no definition/tone
Autonomic dysreflexia
Occurs with SCI above T6
Reaction of the ANS to overstimulation
- Pounding headache
- flushed skin
- red blotches on head and neck
- sweating above injury level
- nausea
- slow heat rate
- goosebumps below level
- blurred vision
Bells Palsy
Temporary unilateral facial paralysis due to trauma with demyelination of the facial nerve.
CNVII. - facial paralysis unilateraly
Inability to taste
Inability to smile, puff cheeks, close eye
Drooping eye, drooping mouth, tearing eye, loss of saliva control
Exacerbated by use of muscles
Brown sequard syndrome
Loss of contralateral pain and temperature and ipsilateral, motor, proprioception, vibration, deep pressure and discrimination.
Contralateral spinothalamic tract
Ipsilateral: DCML tract and corticospinal tract
Bulbar palsy
Weakness/paralysis of muscles innervated by motor nuclei of the lower brainstem
Dysphageia, nasal regurgitations, slurred speech, choking, dysphonia, dysarthria, dysphasia
Cauda equina
LMN lesion caused by injury below L1
Flaccidity, areflexia, incontinence bladder/bowel
Central cord syndrome
Compression to central SC due to hyperextension injury
Motor more than sensory, upper limb more than lower
Loss of spinothalamic tract bilateral, preservation of DCML
Bilateral loss of motor function.
Cerebral palsy
Non progressive brain injury sustained during birth, in utero or in infancy.
Abnormal tone, reflexes and mobility
Dysautonomia
Any disease or malfunction of the ANS
Abnormal HR, breathing, sweating, pupil size, bladder function, fatigue
Guillian Barre
Polyneuritis with progressive muscle weakness that develops rapidly. Often lasts 4 weeks. Most patients heal completely within a year.
Acute inflammatory demyelinating polyneuropathy
Peripheral nerves
Lower extremity weakness progressing to upper extrem
Sensory loss less than motor
Huntingtons disease
Inherited - causes degeneration and atrophy of basal ganglia and cerebral cortex (chromosome 4)
Choreic movement, dystonia, atheoid movement, ataxia, impaired gait, posture and balance. Difficulty with speech and swallowing, incontinence, tongue protrusion
Polio myelitis
Viral disease inflames the grey matter of the spinal cord. Infection.
Post cord syndrome -
Posterior SCI - Compression due to hyperflexion of csp. Rare - only affects DCML tract (prop, vib, deep pressure, sensory discrim)
Post polio syndrome
LMN pathology affects ant horn cells of people who have had poliomyelitis. Progressive weakness, fatiuge, atrophy, pain, swallowing issues.
Trigeminal Neuralgia
Degeneration or compression of the Trigeminal nerve. Harder to reverse if left a long time.
Decreased sensation to forehead, cheek, jaw. Jaw and corneal reflex absent.
Facial pain or numbness from mouth, nostrils eye, ear One side or bilateral
Exacerbated by stress/cold
Upper motor neuron lesion: Location: Structures: Disorders: Tone: Reflexes: Involuntary Movements: Strength: Muscle bulk: Voluntary movements:
Location: CNS
Structures: Brainstem, cortex, corticospinal tracts, SC
Disorders: CVA, TBI, SCI
Tone: Increased
Reflexes: Increased
Involuntary Movements: muscle spasm
Strength: SCI: bilat loss below, CVA: contralateral loss
Muscle bulk: Variable, atrophy from disuse
Voluntary movements: Impaired or absent
Lower motor neuron lesion: Location: Structures: Disorders: Tone: Reflexes: Involuntary Movements: Strength: Muscle bulk: Voluntary movements:
Location: PNS
Structures: Ant horn cell, spinal roots, peripheral nerves, cranial nerves
Disorders: Polio, GBS, PNI, peripheral neuropathy, radiculopathy
Tone: Decreased/absent
Reflexes: Decreased / absent
Involuntary Movements: Fasciculation (with denervation)
Strength: Severe atrophy, limited distribution
Muscle bulk: atrophy from neurogenic causes, rapid and focal wasting
Voluntary movements: weak or absent
Archicerebellum
Vestibular: ocular dysmetria, poor eye pursuit, dysfunctional VOR, Impaired hand eye co-ordingation
Gait and trunk ataxia, with BOS gait
Paleocerebellum
Hypotonia
Truncal ataxia - disequilibrium, posture worse with eyes closed, static postural tremor, increased sway.
High guard arm position, wide BOS
Ataxic gait: unsteady increased falls, uneven/decreased step length, increased step width
Neocerebellum
Intention tremor
Dysdiadochokinesia
Dysmetria: overshooting, errors of direction, amplitude, rebound phenomenon
Dyssynergia - abnormal timing, impairment of multijoint co-ordination, movement sequences and complex motor tasks
Types of MS
Relapsing- remitting: relapses with full recovery, some residual deficit between relapses
Primary progressive - no plateaus or remissions, occasional plateaus.
Secondary progressive - Initial relapsing remitting then progression at different rates
Progressive relapsing - some remission or recovery but progressive disease from onset.
Nonfluent aphasia
Brocas motor aphasia
Expressive
speech is awkward, restricted, interrupted and produced with effort. (L) hemisphere frontal lobe.
Fluent aphasia
Wernikes aphasia Receptive Spontaneous speech preserved and flows smoothly Auditory comprehension is impaired Temporal lobe - (L) hemisphere
Ideomotor apraxia
Cannot perform task on command but can do if left on own
Ideational Apraxia
Cannot perform task at all
Spastic Cerebral palsy
High muscle tone in AG muscles
mass patterns of F/E
Imbalance of tone across joints - contractures
hip F/adductors/ internal rotators / knee flexors/ ankle PF
Crouched gait hip F/knee F
Athetoid Cerebral Palsy
Tonic reflexes ATNR, STNR, TLR block functional postures and movement.
Poor proximal stability
Ataxia and inco-ordination
Poor visual tracking, speech delay, oral motor problems
Ataxic Cerebral Palsy
Low tone Poor balance Unco-ordinated movements hand tremor Poor visual tracking, nystagmus Wide stance gait
Myasthenia Gravis
Progressive muscular disorder
Autoimmune
Strength worse with continuing contraction
Traumatic Brain injury - frontal lobe
Non-fluent Aphasia (brocas) Apraxia Motor planning difficulties difficulty concentrating unpredictable, unstable emotions Difficulty discriminating odours
Traumatic Brain injury - Parietal lobe
Asterognosis (tactile agnosia)
Loss of two point discrim
(R) side - apraxia, impaired body scheme, visual spatial, autitory perceptual disorders
Taste impaired in tongue on contalat side.
Traumatic Brain injury - Temporal lobe
Fluent Aphasia
Decrease in hearing, localise sounds
impaired learning and memory
Difficulty learning new tasks
Traumatic Brain injury - occipital lobe
visual agnosia (can see but cannot recognise or interpret visual information
Homonymous hemianopsia
perceptual impairment
Primary brain damage -
Diffuse axonal injury - disruption or tearing caused by shear strain on small blood vessels
Focal injury: Contusion, laceration, mass effect from hemorrhage and edema
Coup-countercoup: point of impact and opposite point of impact
Closed or open injury with fractured skull
Secondary brain damage:
Hypoxic-ischemia injury from systemic problems, reps or cardiovascular that compromise cerebral circulation
Swelling/edema - increased cranial pressures, brain herniation. Elecrolyte imbalance and release of damaging neurotransmitters.
Anterior Cerebral Artery Strokes affect which areas of the brain and cause which deficits?
Ant and med frontal lobe, medial parietal lobe
Agraphia, aphasia. apraxia, akinetic mutism
Lower limb more affected than UL
Bladder and bowel problems
Medial Cerebral Artery Strokes affect which areas of the brain and cause which deficits?
Cerebrum and basal ganglia UL more affected than LL Facial paralysis Wernikes aphasia Homonymous hemianopia Asognosia
Posterior Cerebral Artery Strokes affect which areas of the brain and cause which deficits?
Occipital lobe. thalamus, midbrain
prosopagnosia
LL and UL contralat hemiplegia
CL pain and temp loss
Lacunar strokes
internal capsule lesion
pure motor loss
contralateral hemi
no aphasia, no visual field loss
Mid brain lesion
CNIII - ptosis, strabismus
C/L Hemi
Medial inferior pontine lesion
paramedian branch of the basilar artery
Ipsilateral - diplopia, ataxia, nystagmus, paralysis of conjugate gaze
Contralateral - hemiparesis UL/LL, impaired sensation
Lateral inferior pontine lesion
Ipsilateral - tinitus, deafness, ataxia, nystagmus, vertigo, paralysis of conjugate gaze, facial paralysis, impaired facial sensation
Contralateral - pain and temp sensation impaired
Locked in syndrome
Anterior inferior cerebellar artery
Lower bulbar paralysis CN V - XII
Can move eyes vertically and blink, cannot talk
Medial medullary Syndrome
Ipsilateral tongue paralysis 1/2 tongue
Contalateral hemiplegia UL and LL
Impaired sensation
Lateral medullary syndrome
PICA - Wallenbergs
Ipsilateral - cerebellar ataxia, vertigo, nystagmus
Loss of pain and temp to face
Sensory loss in Upper or Lower extrem or trunk
Contralat pain and temp to body and face
Left brain CVA
Speech and language Slow cautious behaviour difficulty expressing positive emotion difficulty with verbal cues/commands Apraxia - movement planning
Right brain CVA
Visual perceptual impairment quick impulsive behaviour poor judgement, can't self correct difficulty with perception of emotions difficulty with visual cues difficulty sustaining movement
Neuro-developmental treatment
Controls initiation and sequencing
Rood method
Reflex stimulus model - treatment based on sensorimotor learning consider movement as autonomic and non-cognitive - result of sensory input
Hierarchial reflex theory
higher centers control or inhibit lower centres
Bobath Approach
Neurodevelopmental treatment - patients learn to control movement through normal activities that promote normal movement patterns.
Kabat Knott Voss
Stronger parts stimulate and strengthen weaker parts
Horners syndrome is characterised by
Anhydrosis
Ptosis
Myosis
Fugl-meyer Sensorimotor Assessment of Balance
Performance Battery
Specific balance ax for hemiplegia
Fregley-Graybreil Ataxia Test Battery
High level motor skill only