👧🏾- Musculoskeletal, Vaccines & Neuro Test Flashcards
Spastic (pyramidal) CP
Hypertonicity with poor posture, balance, coordination, fine & gross motor functions, maintaining infant reflex’s
Cerebral palsy
Characteristics of, causes
Impaired muscular control resulting from non-progressive abnormalities in the nervous system
Characteristics of CP- abnormal muscle tone, impaired coordination
Causes- ⬇️ oxygen to the 🧠, prenatal brain abnormalities, prematurity (LBW), perinatal conditions, birth anoxia
Dyskinetic (extrapyramidal) CP
Abnormal involuntary movements, also known as athetoid movements, which are slow wormlike writhing movements with pharyngeal & oral muscle involvement causing drooling & speech problems
Ataxic CP
Characterized by a wide based gait, difficulty with rapid, repetitive movements & incoordination of movements of the upper extremities
Mixed type CP
A combination of spasticity & dyskinetic
Associated disabilities with CP
CI (30-50%)
Sensory impairments
Respiratory problems
Orthopedic complications
ADHD
Seizures
Dental problems (related to seizure meds)
Diagnosis of CP
Delayed gross motor development , abnormal motor performance , alteration in muscle tone (floppy baby) , abnormal posture , reflex abnormalities, associated disabilities
5 Drugs that are therapy for CP
Baclofen pump (⬇️ spasticity)
Ritalin (ADHD)
Tegretal (seizures)
Valium (anxiety & spasticity)
Botox (relaxes muscles)
Spina bifida occulta vs spina bifida cystica
Occulta- a defect that is not visible externally (sacral dimple, hair tufts, color difference)
Cystica- a visible defect with an external saclike protrusion (types meningocele & myleomeningocele)
Meningocele vs myelomeningocele
Meningocele- hernial protrusion of a saclike cyst of meninges filled with spinal fluid
Myelomeningocele- hernial protrusion of a saclike cyst containing meninges, spinal fluid & a portion of the spinal cord with its nerves
Myelo associated complications
Hydrocephalus
Bowel & bladder problems
Paralysis
Orthopedic problems
Meningitis
Latex allergies
Goal of treatment for CP
Promote child’s optimal development
Gross motor Communication Self-help skills Treat associated disabilities Provide education Promote socialization
Myelomeningocele goal of treatment
Protect sac
Prevent infection
Prevent rupture
Surgical closure 24-72hrs after birth
Nursing care for myelo (3 things)
1 prevent infection - antibiotics prophylactically
2 protect sac - keep sterile, keep moist (cover with sterile graze), change dressing q 2-4hrs, good hygiene (keep clean & dry)
3 proper positioning - prone, flat, hips flexed
Post op care for myelo
4 things
1 prone & flat
2 good skin care (urine & feces)
3 Monitor for complications - daily head circumference, CSF leakage (looks like a halo on dressing)
4 neuro assessment - movement of extremities, sphincter control, signs of increased ICP, supportive care
Latex allergy
Cause, allergies related to latex, foods
Cause- repeated exposure (multiple surgeries & urinary catheterizations)
Allergies related- balloons & balls, pacifiers, band-aids, catheters and gloves
Foods- bananas 🍌, avocados 🥑, kiwi 🥝, chestnuts 🌰
Symptoms of latex allergy
Urticaria (hives)
Wheezing
Watery eyes
Rashes
Anaphylactic shock
Muscular dystrophy
Progressive wasting of muscle groups
Progressive weakness of symmetrical skeletal muscles
Resulting in disability & deformity
Duchenne MD
Pseudohypertrophic MD
Increased muscle mass related to fatty infiltration (calves, thighs & upper arms)
Cause of duchenne MD
X-linked recessive disorder
Seen in males
Transmitted by females
Early onset (3-7 years)
S & S of duchenne MD
- waddling gait with frequent falls
- lordosis
- loss of ambulation
- enlarged muscles
- slow progressive weakness
- CI (mild to moderate)
- gower sign
Diagnosis of DMD
S&s of disease
Gene analysis
Increased CPK, AST, ALDOLASE
electromyography (emg)
Muscle biopsy
Care of child with DMD
- corticosteroids
- maintain function for as long as possible
- stay active (breathing exercises)
- ROM
- bracing
- release of contractures
- cough & ventilation assistance
- genetic counseling
Gullain barre’ syndrome (infectious polyneuritis)
Cause
Acute demyelinating polyneuropathy with a progressive, usually ascending flaccid paralysis
Cause- immune mediated disease, associated with viral or bacterial infection or vaccines
Patho of Guillain barre’
Inflammation & edema of the spinal and cranial nerves occur
Followed by impaired nerve conduction
The impaired nerve conduction results in symmetrical ascending paralysis
S&S of guillain barre’
- usually begins with a viral or bacterial infection
- 10 days later neurological signs begin
- ascending bilateral paralysis from lower extremities occurs
- prognosis for these children is good
- most children recover in 2-4 weeks
Diagnosis of guillain barre’
History
S&S
Increased protein in CSF
EMG (shows evidence of acute muscle denervation)
Nursing care for GBS
- assessment of respiratory function
- pharyngeal assessment
- skin care
- CA Monitor
- good body alignment
- suction
- position changes/rom
Nursing care for GBS
Severe paralysis
- IV immunoglobulin
- temporary trach
- ventilator assistance
- IV steroids
- NG feeds
- bowel & bladder care
- prevention of DVT’s
- plasmapheresis (removal, treatment and return of blood plasma)
Contusion
Damage to soft tissue, subcutaneous structures & muscles causing injury to blood vessels and inflammation
Dislocation
Stress on ligament causing displacement of two bones or bone to its socket
Sprain
Joint trauma so severe, ligaments partially or completely tear or stretch causing damage to blood vessels, muscles, tendons & nerves
Strain
Microscopic tear to the musculotendinous unit
Treatment of soft tissue injuries
R - rest
I - ice
C - compression
E - elevation
First 12 to 24hrs is critical
Why is ice used for soft tissue injuries
6 things
1 decreases edema 2 decreases tissue oxygen needs 3 decreases peripheral vasoconstriction 4 increases deep tissue vasodilation 5 reduces pain
** apply for only 30 mins (effects last up to 7hrs)**
Why are rest , compression and elevation used for soft tissue injuries
Rest- prevents further damage to tissue
Compression - stops active bleeding or fluid loss into tissue, decreases edema, reduces pain
Elevation- increases venous return
Fracture
Goal of treatment
A break in the bone structure
Goal- regain & maintain alignment, restore function, prevent further injury
S&S of fractures
Generalized swelling
Pain or tenderness
Deformity
Decreased use
Ecchymosis
Muscle rigidity
Crepitus
Compartment syndrome / 6 P’s
Compression of nerves, blood vessels and muscle inside a closed space
Pain Pulselessness Pallor Paresthesia Paralysis Pressure
Emergency treatment of fracture
- assess 6 P’s
- determine mechanism of injury
- move injured part as little as possible
- cover open wounds
- immobilize injured limb
- apply traction if circulatory compromise (no pulse, blue, cold)
- elevate injured limb
- apply cold to injured area
- call ems
Fracture complications
8 things
1 circulatory impairment 2 Nerve compression syndrome 3 compartment syndrome 4 epiphyseal damage (effect leg length) 5 non or malunion 6 osteomyelitis 7 kidney stones 8 pulmonary embolus
Cast care
8 things
1 elevate extremity 2 handle w/ palms of hand 3 perform frequent neurovascular checks 4 don’t put anything in cast 5 petal cast edges 6 keep clean of urine & feces 7 assess for “hot spots” = infection 8 assess for compartment syndrome
3 essential components of traction
Done to align bone
1 traction (weights)
2 counter traction (body weight)
3 friction (bed)
Bryant traction and something to note about it
1 Bryant traction - pull is in only one direction. Skin traction is applied to the legs and child’s trunk (with butt raised slightly off the bed) provides counter traction
** only used in patients less than 2 years old or <30 lbs cuz of postural hypertension **
Acyanotic heart defect vs cyanotic heart defect
Acyanotic- left to right shunting of blood
Cyanotic- right to left shunting of blood
Atrial septal defect
Abnormal opening between the atria, allowing blood from the higher-pressure left atrium to flow to the lower-pressure right atrium
Coarctation of the aorta
High blood pressure and bounding pulses in arms, weak or absent femoral pulses, and cool lower extremities with lower blood pressure
Ventricular septal defect
Abnormal opening between the right and left ventricles
Small defects surgically repaired with a purse-strung approach; large defects repaired by a Dacron patch sewn over the opening; both procedures done via cardiopulmonary bypass
Patent ductus arteriosus
Failure of the fetal ductus arteriosus (artery connecting the aorta and pulmonary artery) to close within the first weeks of life - allows blood to flow from higher pressure aorta to lower pressure pulmonary artery which causes a left-to-right shunt
Has a characteristic machine-like murmur
Tetralogy of fallot
The classic form includes four defects: 1 ventricular septal defect 2 pulmonic stenosis 3 overriding aorta 4 right ventricular hypertrophy
Transposition of great arteries
The pulmonary artery leaves the left ventricle, and the aorta exits from the right ventricle with no communicable between the systemic and pulmonary circulations
Blood flow pattern groups of classifying heart disease
Increased pulmonary, obstruction, decreased pulmonary, mixed
Increased pulmonary blood flow- atrial or ventricular septal defects , patent ductus arteriosus
Obstruction blood flow- coarctation of aorta, aortic or pulmonic stenosis
Decreased pulmonary blood flow- tetralogy of fallot
Mixed blood flow- transposition of the great arteries
Congestive heart failure
Inability of the heart to pump adequate amounts of blood to meet the body’s metabolic demands
The most common cause of congestive heart failure in children is
Increased blood volume and increased pressure within the heart secondary to structural abnormalities
List the four goals of the therapeutic management of congestive heart failure
1 improve cardiac function (⬆️ contractility ⬇️ afterload)
2 remove accumulated fluid & Na (⬇️ preload)
3 ⬇️ cardiac demands
4 improve tissue oxygenation and ⬇️ O2 consumption
What are the two meds primarily used in kids with CHF
Digoxin and ace inhibitors
Identify the signs of digoxin toxicity in children
Nausea, vomiting 🤮, anorexia, ⬇️ ❤️ rate, dysrhythmias
Clinical manifestations of systemic congestion , a consequence of right-sided heart failure
Systemic venous congestion
Weight gain
Peripheral edema
Ascites
Neck vein distention
⬆️❤️ rate
Sweating
⬇️ urine output
Fatigued
Hypoxia vs cyanosis
Hypoxia- ⬇️ tissue oxygenation
Cyanosis- blue discoloration with ⬇️ o2 sat
Children with tetralogy of fallot May have hypercyanotic spells , how are they treated
Place infant in knee/chest position
Administer 100% “blow-by” O2
Give morphine SQ or IV
Begin fluid replacement
The most common causative agent of bacterial endocarditis is
Treatment
Staph aureus and streptococcus viridans
Treatment- high dose antibiotics IV for 2-8wks
Rheumatic Fever
Sequela, Cause, prevention, treatment
Sequela- heart damage to mitral valve
Cause- abnormal immune response to a group a strep infection
Prevention- prompt diagnosis & treatment of strep infections
Treatment- penicillin, salicylates, bed rest, quiet activities, good nutrition, prophylactic treatment against recurrence
Kawasaki disease
Treatment
Acute systemic vasculitis which primarily involves the cardiovascular system
Treatment- high doses of IV immunoglobulin along with salicylate therapy
True or false. In fetal circulation, the pressure on the left side of the heart exceeds the pressure on the right side
False
Buck extension traction
Type of traction with the legs in an extended position
Primarily used for short-term immobilization , such as preoperative management of a child with a dislocated hip or for correction of contractures or bone deformities
Russell traction
Uses skin traction on the lower leg and a padded sling under the knee
90 degree-90 degree traction
The lower leg is supported by a boot cast or a calf sling and a skeletal steinmann pin or kirschner wire is placed in the in the distal fragment of the femur, resulting in a 90-degree angle at both the hip and the knee
Traction nursing care
Maintain traction at all times
Maintain correct body alignment
Assess skin for breakdown
Provide pin care (skeletal traction)
Assess 6 P’s
Overuse syndrome
S&S, treatment, nursing interventions
Repetitive microtrama due to the same movements over a long period of time
S&S- inflammation, pain, swelling & disability
Treatment- rest & alteration in activities, PT (whirlpools, taping, bracing, splinting), medications (NSAIDS)
Nursing interventions - prevention & treatment of injuries, rehabilitation
Distraction
Process of separating opposing bone to encourage regeneration of new bone in the created space
Ilozarov external fixators
System of wires, rings and telescoping rods that permits limb lengthening to occur by manual distraction
Developmental dysplasia of hip
3 degree of dysplasia
Head of femur not well placed in acetabulum
3 degrees:
•acetabular dysplasia- shallow acetabulum
- subluxation- incomplete dislocation (femoral Head remains in contact w/ acetabulum)
- dislocation- femoral Head loses contact w/ acetabulum
S&S of developmental dysplasia of hip
Limited abduction
Asymmetrical skin folds
+ galeazzi or allis test
+ ortolani & barlow signs
+ trendelenburg sign
Galeazzi or allis test vs ortolani & Barlow
Shortened femur w/ flexion of knees
Ortolani- femoral Head moves out & into the acetabulum