Module 8: Diseases of the Biliary Tract and Exocrine Pancreas Flashcards

1
Q

Cholelithiasis AKA

A

gallstones

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2
Q

Gallstones are associated with obesity, rapid wt loss, type 2 diabetes, and even __________, or sometimes they’re just idiopathic.

A

pregnancy

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3
Q

Biliary Dyskinesia is a spasm of Sphincter of Oddi that prevents ______ release.

A

bile

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4
Q

Cholecystitis is the total obstruction of ________ with gall bladder inflammation or blockage by _______________.

A

bile, gallstones

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5
Q

Cholestasis is sludge build-up in the _______ _____________.

A

gall bladder

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6
Q

Gallstones are made from what?

A

cholesterol + bilirubin + calcium salts

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7
Q

Choledocholithiasis is bile duct obstruction by a ___________ that causes abdominal pain and cramping.

A

stone

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8
Q

T/F: Choledocholithiasis may cause fat malabsorption due to no emulsifying fat and steatorrhea.

A

T

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9
Q

The main pigment in bile is _______, which gives feces the dark color.

A

bilirubin

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10
Q

T/F: Backup of bile from choledocholithiasis could lead to jaundice and liver damage.

A

T

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11
Q

MNT for acute cholecystitis may need NPO and a diet in ________ or MCT to stop gallbladder contractions.

A

low-fat

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12
Q

Cholecystectomy is the surgical removal of the ___________.

A

gallbladder

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13
Q

T/F: It’s okay to have excessive amounts of fat after having a cholecystectomy.

A

F

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14
Q

T/F: You DON’T NEED to have a gallbladder.

A

T

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15
Q

The pancreas is a ___________ organ.

A

glandular

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16
Q

The pancreas has endocrine and ________ glands.

A

exocrine

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17
Q

The endocrine glands in the pancreas are ductless and secrete hormones into the ___________.

A

bloodstream

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18
Q

The exocrine glands in the pancreas secretes ________ enzymes and ________ rich fluid through _______ (i.e. intestinal lumen) that lead to the primary pancreatic duct.

A

digestive, bicarbonate, ducts

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19
Q

Cephalic stimulation is mediated by the ________ nerve that signals the pancreas to release digestive juices.

A

vagus

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20
Q

Through cephalic stimulation, sight, smell, taste or the thought of ______ can trigger the secretion of __________ and pancreatic enzymes.

A

food, bicarbonate

21
Q

Gastric distention (food in the stomach) can stimulate _________ secretion from the pancreas.

A

enzyme

22
Q

The major stimulus for pancreatic secretion is when _______ reaches the _______.

A

food, duodenum

23
Q

When food reaches the duodenum, the SI release _____ and __________ which travel through the blood to the pancreas.

A

CCK, secretin

24
Q

Secretin stimulates the release of __________ rich fluid to neutralize the acidic chyme.

A

bicarbonate

25
Q

CCK stimulates the release of _________ enzymes.

A

pancreatic

26
Q

Pancreatitis is inflammation of the _________.

A

pancreas

27
Q

T/F: Pancreatitis can lead to premature activation of _______ enzymes in the pancreas instead of the duodenum.

A

zymogen

28
Q

Pancreatic proteases are secreted as _________ into the SI.

A

zymogens

29
Q

T/F: Pancreatitis can cause severe abdominal pain.

A

T

30
Q

You can see elevated levels of pancreatic enzymes (of CHO and fat digestion) of serum ________ and ________in someone with pancreatitis.

A

amylase, lipase

31
Q

T/F: About 25% of acute pancreatitis will go on to chronic pancreatitis

A

T

32
Q

T/F: Low levels of triglycerides can lead to pancreatitis.

A

F (high levels)

33
Q

T/F: Excessive alcohol intake can lead to pancreatitis.

A

T

34
Q

T/F: Gallstones can lead to pancreatitis.

A

T

35
Q

MNT for acute pancreatitis may be _____ on IV fluids for a couple of days and _______ feeding (parenteral nutrition) may be issued within a few days.

A

NPO, tube

36
Q

Those with pancreatitis may need _____PRO needs in their diet.

A

higher

37
Q

T/F: It is important to NOT OVERFEED patients with pancreatitis.

A

T

38
Q

T/F: Those with pancreatitis may have low blood glucose.

A

F (high blood glucose)

39
Q

T/F: Somatostatin can be added in MNT for pancreatitis to inhibit pancreatic secretion.

A

T

40
Q

__________ nutrition is always preferable to parenteral nutrition for MNT of pancreatitis.

A

Enteral

41
Q

For severe cases of pancreatitis, MNT may be using _______ tube feedings to bypass the gastric phase of pancreatic secretion.

A

jejunal

42
Q

______ is the most potent stimulus for the release of pancreatic enzymes.

A

CCK

43
Q

______ is the most potent stimulus for CCK release.

A

Fat

44
Q

Chronic pancreatitis is usually presented as current flare-ups of ________ ____________.

A

acute pancreatitis

45
Q

T/F: Chronic pancreatitis could be idopathic.

A

T

46
Q

Chronic pancreatitis could lead to secondary _________ because it could lead to damage of the _______ cells that secrete insulin.

A

diabetes, beta

47
Q

The most common cause chronic pancreatitis is what?

A

Heavy alcohol consumption

48
Q

MNT for chronic pancreatitis includes ________ diet, small & frequent meals, supplemental pancreatic enzymes taken before meals, ______-chain triglycerides, antacids, H2 blockers or PPIs to reduce stomach acid,

A

low-fat, medium

49
Q

Vitamin deficiencies in B12, A, D, E, and K must be monitored in patients with ______ pancreatitis.

A

chronic