Janes' Wiggs Chapter 15 and 16 - Endodontics Flashcards
According to Wiggs Chapter 15 (endodontics)
What is contained within a normal dentinal tubule?
- Tubules contain tomes fibre, nerve ending, and fluid
- Tomes fibres are the odontoblast processes which perforate the dentin
N.B. Tomes process is something different - involved in enamel secretion by ameloblast
According to Wiggs Chapter 15 (endodontics)
What is the difference between reversible and irreversible pulpitis?
With reversible pulpitis, pulp can return to a non-inflamed state.
With irreversible pulpitis, tooth will progress to pulp necrosis.
Reversible pulpitis is generally a symptom of
pulpal inflammation caused by some form of short-term
irritation such as caries, dental treatment, or trauma.
According to Wiggs Chapter 15 (endodontics)
Describe anachoresis:
Anachoresis – infection of a tooth via bloodstream or regional lymphatics,
According to Wiggs Chapter 15 (endodontics)
Describe how trauma without pulp contamination may result in non vital tooth?
Trauma causes inflammation within the pulp.
Since it is in a confined space, as inflammation progresses, rather than classical swelling (enlargement of tissue) pressure within the pulp chamber develops
Leads to collapse of venules.
This prevents blood from draining away, and leads to pulp strangulation,
According to Wiggs Chapter 15 (endodontics)
How often does sterile pulp necrosis cause periapical periodontitis
in the absence of bacterial contamination this necrosis does not cause periapical periodontitis
According to Wiggs Chapter 15 (endodontics)
Describe this pathology:

CCF 108, hyperplastic pulpitis
According to Wiggs Chapter 15 (endodontics)
How is periapaical pathology definitively diagosed?
Histopathology
According to Wiggs Chapter 15 (endodontics)
What are the 5 categories of periapical pathology?
- acute apical periodontitis of pulpal origin,
- chronic apical periodontitis,
- periapical abscess with sinus,
- periapical abscess without sinus,
- radicular cysts
According to Wiggs Chapter 15 (endodontics)
What are the radiographic and clinical signs of acute apical periodontitis?
Rads may be normal, or slight widening of PDL at apex
Clinical signs are tenderness, pain on palpation/percussion (tough to see in our pts)
According to Wiggs Chapter 15 (endodontics)
What is the classic radiographic sign of Chronic Apical Periodontitis?
produces the classical periapical lucency seen on radiographs
According to Wiggs Chapter 15 (endodontics)
What are the radiographic signs of a periapical abscess?
May be no signs if acute
may be chronic signs of endodontic disease if an acute flare of a chronic process
According to Wiggs Chapter 15 (endodontics)
What is a phoenix abscess?
A periapical abscess which has arisen from a chronic apical periodontitis.
According to Wiggs Chapter 15 (endodontics)
What is a periapical abscess with sinus?
endodontic disease which has progressed and made a draining tract (sinus)
sinus tracts may be seen facially or suborbital, ventral‐mandibular, or within the oral mucosa, either gingival or at the mucogingival line
According to Wiggs Chapter 15 (endodontics)
What is a radicular cyst?
What are the two types?
What are radiographic features?
proliferation of cell rests of mallasez, which create an apical epithelium lined cyst cavity at the apex.
Pocket cyst is if it communicates with the pulp
true cyst is when cavity lining is complete and does not communicate with the pulp
appears as a classic radiolucency.
According to Wiggs Chapter 15 (endodontics)
How is a radicular cyst differentiated from a periapical granuloma?
On the basis of histopathology.
cysts are sack-like structures with a complete epithelial lining whereas granulomas only contain a mixed cell population including epithelial tissue
will both appear as a lucency on rads
According to Wiggs Chapter 15 (endodontics)
How does condensing osteitis appear radiographically and what is it’s cause?
Reactive bone formation and increased radiodensity of periradicular alveolar bone.
Pulp inflammation stimulates osteoblast activity
According to Wiggs Chapter 15 (endodontics)
How can condensing osteitis (pathological) be differentiated on radiographs from periapical osteosclerosis (non-pathological) seen in hard chewing dogs?
Can’t be differentiated on rads
According to Wiggs Chapter 15 (endodontics)
What are 4 signs of endodontic disease on radiographs?
Condensing osteitis
Radicular ankylosis
external root resorption occurs in some endodontic disease
Periradicular osteomyelitis (alveolar bone expansion) appears as osteopenia and expansion of the alveolus
According to Wiggs Chapter 15 (endodontics)
what are the most common radiographic findings in endodontic disease?
- Condensing osteitis
- Chronic focal sclerosing osteomyelitis
According to Wiggs Chapter 15 (endodontics)
What are classic features to differentiate perio-endo disease from endo-perio disease?
- Primary endo usually have a crown fracture. If it continues to a wide PDL can be called a J lesion
- Primary perio usually have deep pockets and wedge shaped periodontal lesions
According to Wiggs Chapter 15 (endodontics)
Describe class I, II and III Perio/endo lesions
Class I - Primary endo
Class II - Primary Perio
Class III - separate endo/perio component
Memory trick: alphabetical order - Endo, Perio, Separate
According to Wiggs Chapter 15 (endodontics)
What are group I Endodontic instruments?
Hand instruments like barbed broaches, K files and H files
According to Wiggs Chapter 15 (endodontics)
What are group II endodontic instruments?
Low speed attachments, with a latch grip
like gates glidden drills and peeso reamers
According to Wiggs Chapter 15 (endodontics)
What are group III endodontic instruments?
Engine driven Ni-Ti rotary instruments which adapt to curved canals
lightspeed etc.















