images wk 2 Flashcards
What are angles of each of these?
lateral = 0
II = +60
aVF = +90
III = +120
aVL = -30
aVR = -150
What pathology is this? How can you tell?
SInus bradycardia
long space between QRS waves, but each waveform looks normal
indicates dysfunctional SA
What pathology is this?
First degree AV block
P-R interval should be 120-200 ms, these are 240 ms
means delayed but not blocked, all P waves still followed by QRS just takes longer
means you have delayed conduction through AV
What pathology is this?
second degree AV block
P waves not always followed by QRS
means some impulses fail to propagate to ventricles
What pathology is this?
Atrial fibrillation
P waves random and indistint
R-R intervals irregular
What pathology is this?
premature ventricular contractions [PVC]
normal QRS are very narrow
wide QRS are PVC = ventricles excite themselves
Two PVCs have different shapes = have multile ectopic foci
What pathology is this?
Monomorphic ventricular tachycardia [VT]
What is the pathology?
ventricular fibrillation
irregular, no identifiable pattern
asynchronous contractions, ineffective, can lead to death
What do each of these arrows point to on PV loop [x axis is LV volume, y axis is LV pressure]
left vertical arrow: end systolic volume [ESV]
right vertical arrow: end diastolic volume [EDV]
longer horizontal arrow: end diastolic volume [EDV]
shorter horizaontal arrow: stroke volume [SV]
What do these 3 arrows point to in PV loop?
top arrow: Pes = pressure at end systole [ESP]
middle arrow = EDP [end diastolic pressure]
bottom arrow = LAP [left atrial pressure]
What do these arrows point to in PV loop?
bottom arrow: DBP [diastolic blood pressure]
top arrow: SBP [systolic blood pressure]
What 2 things are constant in these circle, what is varied?
- contractility and afterload are constant
- preload is varied
What is varied here? What two things are constant?
TPR is varied
preload and contractility are constant
What is changing here? what is staying the same?
Contractility is changing [more contractility = bigger slope of ESPVR]
preload [EDP/EDV] and afterload [TPR] staying the same
What does this graph show [control on left]? What changed, what stayed the same?
- constriction of arterioles leading to: greater MAP, greater pressure drop across arterioles
same Pc