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Virology Michelle > Herpes Viridae Part 2 > Flashcards

Herpes Viridae Part 2 Flashcards

1
Q

What are the 4 pathotypes of Marek’s Dz?

A

Mild
Virulent
Very Virulent
Very Virulent +

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2
Q

mMDV is mostly associated with ____ MD and prevntable with ______

A

neural MD

turkey herpesvirus vx

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3
Q 
Marek's Dz
Etiology
Hosts
Distribution
Transmission
A

Gallid herpesvirus 2
Chickens mostly but other poultry too
Worldwide
Feather follicles and dander (less infectious)

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4
Q

vMDV is associated with high incidence of ______ and _______ lymphomas. Disease is preventable with ________

A

neural and visceral

HTV, turkey herpes virus vx

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5
Q

vvMDV is associated with high incidence of ______ and _______ lymphomas. Viruses are oncogenic in ______ _________ chickens. Dz is preventable with _______

A

neural and visceral lymphomas
HVT vx chickens
bivalent vx

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6
Q

vv+MDV is associated with high incidence of neural and visceral lymphomas. Viruses are oncogenic in chickens ________

A

vx with bivalent vx

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7
Q

What are the genetically susceptible and resistant haplotypes of MHC II of chickens in regards to Marek’s Dz?

A

B19- susceptable

B21- resistant

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8
Q

What are the clinical signs in Marek’s Dz

A

Neurolymphamatosis
Visceral lymphomatosis
Ocular lymphomatosis
Cutaneous lymphomatosis

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9
Q

In Marek’s Dz, which is reportable, what is the most protective commercial vx available?

A

CVI988/Rispens

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10
Q 
Inclusion Body Rhinitis 
Etiology
Host
Distribution
Transmission
A

Porcine Herpesvirus2/Porcine cytomeglovirus
Pigs, 2-10 wks old. Severe if less than 3 wks old.
Worldwide
Inhalation and Transplacental

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11
Q

Pathogenesis of Inclusion Body Rhinitis

A

Widespread petechiae and edema

Most common in thoracic cavity and sq tissues

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12
Q

Clinical signs associated with Inclusion Body Rhinitis

A

Pigs less than 3 wks- mucopurulent rhinitis, violent sneezing and resp distress
Neonatal pigs appear weak, anemic, stunted
Can cause mummification or still births
Subclinical in older adults

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13
Q

Malignant Catarrhal Fever
Host
Etiology

A

Cattle and Wild Ruminants
Alcephaline herpesvirus-1 (Wildebeast associated MCF)
Ovine herpesvirus-2 (Sheep associated MCF)

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14
Q

Wildebeast associated MCF

A

transmitted from cattle to wildebeast
occurs in most african countries
doesn’t cause dz in Wildebeast/principal host
is epizootic and seasonal

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15
Q

Sheep associated MCF

A

world wide
transmitted from sheep (or goats) to cattle
occurs year round in cattle
sporadic, occasional outbreaks

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16
Q

How is Wildebeast associated MCF transferred to cattle?

A

It’s present in the nasal and occular secretions of young wildebeast, cows eat it from contaminated pasture or direct/close contact, inhalation of aerosol, contact with calving wildebeast. Rarely transmitted from adults.

17
Q

How is MCF transmitted from Sheep to Cattle?

A

Unknown, maybe inhalation or ingestion

18
Q

Clinical Signs in MCF

A

Peracute- sudden death
Head and eye- majority
Alimentary/Intestinal- diarrhea, more common in sheep MCF
Mild form- innoculated animals

19
Q

In what virus would you find Zebra striping of the bovine colon?

A

MCF

20
Q

Hemorrhagic Dz of Puppies
Etiology
Host
Transmission neonates and older dogs

A

Canine herpes virus 1
Dogs and wild canidae
neonates- contact w/ oral/nasal/vaginal secretions, in utero, from passage through birth canal
older dogs- venereal transmission, contact w/ infected puppies

21
Q

At what temp does CHV-1 replicate at?

A

33C

Found in outer genital and upper respiratory tracts

22
Q

Clinical signs of Hemorrhagic Dz of puppies

A

Hemorrhages in multiple organs
painful crying, abdominal pain, anorexia, dsypnea, no elevation in body temperature
If survive, develop persistant neruo signs like ataxia and blindness

23
Q

Hemorrhagic Dz of Puppies
Adult genital infection
Adult Respiratory Infection
Ocular Infection

A

Genital- generally asymptomatic, in utero can cause abortion/mummies
Respiratory- older dogs, rhinitis and pharyngitis
Ocular- conjunctivitis

24
Q

Hemorrhagic Dz of Puppies

A

Focal areas of necrosis
Intranuclear inclusion bodies
FAT serology

25
Q

Control of Hemorrhagic Dz of Puppies

A

Heated whleping box

Isolation of infected bitch and litter

26
Q 
Feline Rhinotracheitis 
Etiology 
Distribution
Host
Transmission
A

Feline herpesvirus 1
worldwide
all felidae
direct contact

27
Q

Pathogenesis of FHV-1

A

Replicates in mucosae of nasal septum, etc
Viremia rate
multifocal epithelial necrosis, inflammation and fibrinous exudation

28
Q

Clinical signs of FHV-1 in Kittens up to 4 wks and Cats older than 6 mo

A

Kittens- URI, rhinitracheitis, fatal bronchopneumonia

Cats- mild or subclinical dz

29
Q

Clinical signs of pregnant queen with Feline Rhinotracheitis

A

Abortion around 6th wk of pregnancy

virus doesn’t cross placenta

30
Q

How can you differentiate between Feline Calici virus and FHV-1 infections?

A

Tongue ulcers and lameness is associated with Feline Calici virus

31
Q

What 3 types of vx are available for FHV-1?

A

MLV parenterally
MLV intranasally
Inactivated parenterally

32
Q

Infectious Laryngotracheitis
Etiology
Host
Distribution

A

Gallid herpes virus 1
chickens 4-18 mo and other fowl
worldwide

33
Q

ILT transmission

A

Inhalation mostly

Can also be droplets to conjunctiva, ingestion, carriers, fomites, mechanical (vultures and crows)

34
Q

ILT virus can persist in infected birds. What is the target for latency?

A

Trigeminal ganglion

35
Q

Clinical Signs of ILT

A

Incubation 6-12 Days, mild coughing/sneezing, nasal and ocular dc, depression
Severe form- respiratory distress, pump handle respiration, coughs up bloody mucus
Mild form most common

36
Q

Dx of ILT

A

History and clinical finding
necropsy- tracheal plug
grow on CAM

37
Q

What types of vx are there for ILT and which has better immunity

A

CEO has better immunity but can revert to virulence

TCO has limited immunity but doesn’t revert or spread

Virology Michelle (11 decks)

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