heart sounds Flashcards

1
Q

when is S1 heard?

A
  • when the mitral and tricuspid valves close at the beginning of ventricular systole
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

where is S1 best heard?

A

in the apex

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

where do you listen for mitral stenosis?

A

in the apex area over the mitral valve

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

how do you enhance the intensity of S1 in mitral stenosis?

A

by rolling the patient onto their left hand side

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

what can cause a quiet S1?

A
  • low CO
  • poor left ventricular function
  • rheumatic mitral regurgitation
  • long P-R interval (first degree heart block)
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

what can cause a loud S1?

A
  • increased CO
  • large SV
  • mitral stenosis
  • short P-R interval
  • atrial myxoma (rare)
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

what can cause a variable S1?

A
  • AF
  • extrasystoles
  • complete heart block
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

when is S2 heard?

A
  • closure of aortic and pulmonary valves at the end of systole
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

where is S2 best heard?

A

at the left sternal edge

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

why does S2 splitting occur?

A
  • because left ventricular contraction slightly precedes that of the right ventricle so that the aortic valves close before the pulmonary valve
  • the splitting increases at end-inspiration because increased venous filling of the right ventricle further delays pulmonary valve closure
  • lub d-dub
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

when might the aortic component of S2 be quiet or absent?

A
  • in calcific aortic stenosis

- aortic regurgitation

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

when might the aortic component of S2 be loud?

A
  • systemic hypertension
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

when might the pulmonary component of S2 be loud?

A
  • pulmonary hypertension
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

when would wide splitting of S2 but normal resp variation occur?

A
  • right bundle branch block

- pulmonary hypertension

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

when would fixed splitting of S2 with no variation in resp occur?

A
  • atrial septal defect
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

when/where is S3 best heard?

A
  • low-pitched early distolic sound heard best at the apex
  • coincides with rapid ventricular filling immediately after the opening of the AV vales
  • lub-dub-dum
17
Q

is S3 normal?

A
  • yes, in young adults and during pregnancy

- pathological after the age of 40

18
Q

what are the most common pathological causes of S3?

A
  • left ventricular failure

- mitral regurgitation

19
Q

when/where is S4 heard?

A
  • soft and low-pitched
  • heard at apex
  • occurs just before S1 (da-lub-dub)
20
Q

is S4 pathological?

A
  • yes, always
21
Q

what causes S4?

A
  • forceful atrial contraction against a non-compliant or stiff ventricle
  • often heard with left ventricular hypertrophy
22
Q

when can S4 not occurs?

A
  • with AF
23
Q

when might a snap be heard and what is it?

A
  • mitral stenosis
  • results from a sudden opening of a stenosed valve and occurs in diastole, just after S2
  • best heard at the apex
24
Q

when are murmurs heard?

A
  • when there is turbulent blood flow across an abnormal valve, septal defect or outflow obstruction