heart part II Flashcards
occlusion
blockage
ischemia
blood deficiency in a breach of coronary artery often due to constriction/blockage from clot
angina pectoris
chest pain that accompanies ischemia
myocardial infarction
heart attack; death to portion of heart due to ischemia
cardioacceleratory center (CAC)
medulla oblongata; sympathetic- INCREASE heart rate
sympathetic nerve fibers
leave CAC region in medulla oblongata; INCREASE rate/strength of heartbeat
parasympathetic nerve fibers
leave CIC region to HEART via vagus nerve (cranial X); DECREASE heart beat
cardioinhibitory center (CIC)
medulla oblongata to heart via vegas nerve (cranial X)- parasympathetic DECREASE heart rate
What is responsible for increase heart rate
sympathetic nerve fibers in CAC (medulla oblongata)
What is responsible for decrease heart rate
parasympathetic nerve fibers in CIC (medulla oblongata)
vagus nerve
cranial X; parasympathetic nerve fibers leave CIC via this route to slow heart rate
intrinsic regulation
nervous system WITHIN heart itself
where is sino-atrial node (SA node)
posterior of right atrium beneath epicardium below superior vena cava
SA node aka
pacemaker of heart
why is SA node called pacemaker of heart
starts action potentials (intrinsic regulation)
where do action potentials from SA node spread to
both atria (contract together)
where is atrioventricular node (AV node)
floor of right atrium
where does action potential spread to from AV node
AV bundle (bundle of His)
action potential location after bundle of His
through inter ventricular septum to left/right bundle branches
where do bundle branches carry action potential to
purkinje fibers
purkinje fibers
carry action potential of heart into cells of myocardium and cause ventricles to contract