Feb14 M2-Dental Caries Flashcards

1
Q

common example of systemic diseases linked to oral health

A

diabetes, obesity and caries are linked together by high sugar intake

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2
Q

oral manifestation of boulemia

A

erosion of teeth surface due to vomiting

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3
Q

oral manifestation of ulcerative colitis

A

pustules on oral mucosa (and in rest of gut too but easier to see in the mouth)

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4
Q

xerostomia def + in what pop

A

lack of saliva. frequent in aging population

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5
Q

consequences of xerostomia

A
  • drop in QoL
  • more prone to oral infection
  • more prone to caries lesions
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6
Q

most common cause of xerostomia + other causes

A

medication (asthma puffers for ex). other: chemo, radiation therapy, Shogern’s syndrome (affects sudation and salivary glands)

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7
Q

crystal meth effect on the mouth and why

A

teeth lesions bc causes xerostomia + has corrosive agents

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8
Q

HIV patients first thing seen (in the mouth)

A

hairy leukoplakia

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9
Q

HIV oral manifestations

A
  • hairy leukoplakia

- oropharyngeal candidiasis (on angle of lips = angular cheilitis, top of tongue which is the dorsal tongue, on palate)

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10
Q

oral manifestation of severe immunosuppression + what happens

A

necrotizing ulcerative periodontitis. rapid loss of bone around teeth: painful, teeth are mobile, smells bad

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11
Q

what’s the cause of the drop in dental decay incidence over the past decades

A

presence of fluoride in toothpaste and water

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12
Q

main risk factor for caries lesions

A

poverty (and lack of education)

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13
Q

3 age groups where prevalence of untreated caries peaks and where they have the worst burden

A

6, 25, 70

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14
Q

why real teeth not necessarily the best thing for elderly

A

if can’t clean them bc frail and limited in movements, dentures are better

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15
Q

dental caries disease process vs dental caries lesions

A
  • disease process = happening all the time to all teeth of everyone. can be controlled but not prevented
  • lesions = result of the disease process
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16
Q

chemical basis for caries disease process

A

bacteria metabolize fermentable sugars and produce acid INTO the dental plaque

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17
Q

how plaque pH (plaque is not a good thing) and amount of enamel (the good thing) change with time and why

A

-pH varies (up and down)
-enamel demineralization and remineralization all the time
don’t eat = higher pH and remineralization

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18
Q

pathological factors in caries disease process

A
  • acid producing bacteria
  • fermentable carbs (in the food)
  • insufficient saliva flow
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19
Q

protective factors in caries disease process

A
  • fluoride
  • saliva flow and buffering capacity
  • regular removal of dental plaque
20
Q

steps before get cavitated lesions

A
  • caries disease process
  • caries lesions
  • non-cavitated lesions
  • cavitated lesions
21
Q

TF dental caries = cavity in the tooth

A

false. can have caries lesions without cavities

22
Q

2 reasons to catch caries lesions as early as possible

A
  1. easier to arrest

2. prevents restoration (that’s a procedure) of a tooth which is a bad thing to have bc many complications

23
Q

visible part of the tooth and its 2 portions

A

crown. outside = enamel. layer inside that is dentin

24
Q

vital part of the tooth name + content

A

pulp. has nerves, blood vessels, cells, etc.

25
when do caries start to hurt
when reach the dentin (got through the enamel)
26
dentin components and why a single caries lesion only has to get there to cause pain
made of tubuli so sugar there goes to nerves in the pulp and it's sensed
27
4 clinical indicators of caries lesions
- lesion colour - lesion shape - lesion location and presence of dental plaque - tooth surface integrity
28
where caries develop and why
where plaque can stagnate for a long period of time so gum lines, pits, fissures
29
plaque shape when it stagnates
banana or arch shape. not circle or line
30
trick for checking if someone has plaque + test equivalent to that
-check if have red gum red gum = plaque -test = apply tooth biofilm
31
3 possible colours of dental caries lesions
- white opaque - yellow or brownish - dark brown or black
32
white spot lesions charact
-enamel with porosities, lost its translucency (glass appearance), looks matte and rough
33
2 causes for yellow or brownish caries lesion
1. if on enamel, food pigment | 2. if lesion reached dentin, normal colour bc dentin is naturally darker
34
black dental caries prognosis
best prognosis. are easy to arrest
35
explanation for caries lesion in area that is not a plaque stagnation area
caries lesion was there when the tooth was erupting (at some point, this was a plaque stagnation area)
36
tooth surface integrity indicates what
stage of dental caries lesions
37
5 stages of dental caries lesions
- sublinical initial lesions - D1: enamel non-cavitated lesions - D2: enamel cavitated lesions - D3: dentinal lesions - D4: pulp lesions
38
first sign of caries lesions
white spots
39
charact of an active dental caries lesions
- region of plaque stagnation - loss of lustre - rough, or soft on gentle probing - whitish yellowish enamel
40
charact of arrested dental caries lesions + treatment
-located at some distance from gingiva -no dental plaque -smooth (not rough) and hard (not soft) -shiny -brown-dark or brown-white colour NO need to treat
41
early childhood caries def
one or more decayed (cavit or non cavit lesions), missing or filled lesion on tooth surface
42
which population severely affected by early childhood caries
aboriginal children
43
nursing bottle caries
caries bc child would keep bottle of milk in their mouth while sleeping
44
main things to tell parents to avoid early childhood caries
- start brushing as soon as 1st tooth appears - sweet snacks between meals - FLUORIDE TOOTHPASTE - brush twice a day
45
how fluoride helps teeth
important for remineralization
46
where caries lesions develop if don't pass the floss + shape
between teeth, below point of contact. are kidney shaped lesions
47
when do you not need to pass the dental floss
when teeth don't touch