[Exam 2/Final] Chapter 27: Management of Patients with Coronary Vascular Disorders Flashcards
Atherosclerosis: What is this?
The buildup of fat inside of the coronary arteries.
Atherosclerosis: What two main arteries come off of aorta?
One feeds right side of heart, and other feeds left side.
Atherosclerosis: What is the main clinical manifestations of this?
Angina Pectoris
Atherosclerosis: What is Angina PEctoris??
Chest pain result of ischemia due to not enough blood flow getting through.
Atherosclerosis - Risk Factors: What Non-Modifiable Risk Factors are included?/
Age, Gender, Race, Family History
Atherosclerosis - Risk Factors: Whaht are some Modifiable Risk Factors?
High blood pressure, obesity, high cholesterol, inactivity, smoking, diabetes
Atherosclerosis - Prevention: What dietary measures can be done?
Eating heart healthy diet, low cholesterol, low fat, low salt.
Atherosclerosis - Prevention: What physical activity changes can be done?
Making sure patients are exercising, getting cardio in.
Atherosclerosis - Prevention: How can meds be preventive?
Can control blood pressure, monitoring diabetes.
Atherosclerosis - Prevention: What to do about tobacco?
Smoking causes plaque buildup, so its best to get them to stop smoking.
Angina Pectoris: What is stable angina?
Very predictable, persistent. .Pain occurs on exertion. Relieved by rest and nitro. Patients are aware of when this chest pain will develop.
Angina Pectoris: What is unstable angina?
Start to develop chest pain more often, higher in frequency. Not relieved by nitro or rest.
Angina Pectoris: What is MI Angina?
Vessel is completely blocked now.
Angina Pectoris: What is a Intractable or Regractory Angina?
Patient is in severe pain and we cannot get the chest pain under control.
Angina Pectoris: what is Variant Angina?
Vasospasms, which can be induced by cocaine, or other stimulants like smoking. Even hypertension. Since its spasming, may be some ischemia.
Angina Pectoris: What is Silent Ischemia?
Patients having ischemia, but experience no chest pain. May be caught on patients taking stress test.
Angina: What are the CMs?
The main one is Chest Pain
Angina: What tests can be performed for Chest Pain?
- EKG, will look for T Wave Inversion or ST Depression.
- Cardiac Markers - Troponin
- Stress test, Echocardiogram
Angina: How will ST look if they were having an MI?
ST Elevation
Angina: Why is a Troponin level drawn?
To rule out that acute coronary syndrome is occuring. Normal Troponin is less than 0.3 and shows if hearts being damaged.
Angina: What will you first do when assessing the patient?
Assess them to see if its stable, unstable, or if they’re experiencing acute coronary syndrome
Angina Medication: If they don’t need surgery or cath lab visit, what medications can be given?
Nitroglycerin (Vasodilates)
Beta Blockers (decrease myocardiac O2 consumption)
CCB (Slow AV/SA node conduction and vasospasm
Antiplatelet/Anticoagulant
Angina Medication: How do the beta blockers work here?
Decrease myocardiac o2 consumption by blocking beta androgenic sitmulation to heart which decreases heart rate which decreases bp.
Angina Medication: Why are antiplatelet and anticoagulants given?
Plavix, Aspirin. Given to prevent a thrombus from forming when plaque ruptures, and forms clotting cascade.
Angina Medication: Why is Oxygen given?
To help perfuse the heart. Not given if patients have O2 sat in low 90s.
Angina - Assessment: What will we assess?
Assess for pain, what caused pain, what relieves it, is it substernal/left arm/ radiating?
Angina - Assessment: What are some nursing diagnoses?
RF Decreased CO
Anxious
Deficient Knowledge
Non-Compliance with Meds
Angina - Potential Problems: What could happen
Acute coronary syndrome, that leads to MI which leads to HF and cardiac arrest, leading to shock.
Angina - Goals: This will be aimed at what?
Preventing the angina and making sure patient understands tx plan.
Reducing anxiety.
Acute Coronary Syndrome (ACS) and MI: What is ACS?
Emergent situation characterized by an acute onset of myocardial ischemia
Acute Coronary Syndrome (ACS) and MI: MI may be referred to as STEMI, which is what?
ST Elevated MI
Acute Coronary Syndrome (ACS) and MI: What kind of Pain will a MI patient refer to?
Sudden onset, substernal, will complain of crushing feeling, tightness, unrelieved by nitro
Acute Coronary Syndrome (ACS) and MI: Pain may radiate where?
Back, neck, jaw/tooth, shoulder, arm
Acute Coronary Syndrome (ACS) and MI: What CMs may be seen?
Dyspnea, Syncope (DEcreased BP), Nausea, Vomiing, Extreme Weakness, Increased Pulse
Acute Coronary Syndrome (ACS) and MI: What broad treatments may occur?
O2, IV, Meds, Monitor
Acute Coronary Syndrome (ACS) and MI: What dietary restrictions may occur
Decreased Na, Cholesterol, and Caffeine
Acute Coronary Syndrome (ACS) and MI - Areas of Damage after MI: What happens in the zone of infarction and necrosis?
O2 Deprieved, Damage Irreversible, Changes “Q” wave on ECG (Elevated about everything else)
Acute Coronary Syndrome (ACS) and MI - Areas of Damage after MI: What happens in the Zone of Injury?
Next to infarction zone. Tissue viable if cirrculation remains adequate. Increasing O2 may save area. ST segment change, and elevation on ECG